The neuroprotective potential of endoplasmic reticulum chaperones

Diseases of the nervous system are characterized by axon dysfunction,axon degeneration,and neuronal cell death.The roster of neurodegenerative diseases is striking in the selective vulnerability of the affected neuronal populations and the timing of the onset of disease manifestation and functional...

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Published in:	Neural regeneration research Vol. 10; no. 8; pp. 1211 - 1213
Main Authors:	McLaughlin, Todd, Zhang, Sarah X
Format:	Journal Article
Language:	English
Published:	India Medknow Publications and Media Pvt. Ltd 01-08-2015 Medknow Publications & Media Pvt. Ltd Departments of 0phthalmology and Biochemistry, Ross Eye Institute, University at Buffalo, State University of New York, Buffalo, NY, USA%SUNY Eye Institute, State University of New York, Buffalo, NY, USA Medknow Publications & Media Pvt Ltd Wolters Kluwer Medknow Publications
Subjects:	Apoptosis Development and progression Endoplasmic reticulum Genetic aspects Kinases Macular degeneration Molecular chaperones Nervous system diseases Neurodegeneration Physiological aspects Retina 伴侣保护电位内质网功能障碍神经元群神经系统神经细胞死亡神经退行性疾病 United States
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Summary:	Diseases of the nervous system are characterized by axon dysfunction,axon degeneration,and neuronal cell death.The roster of neurodegenerative diseases is striking in the selective vulnerability of the affected neuronal populations and the timing of the onset of disease manifestation and functional deficits.
Bibliography:	Diseases of the nervous system are characterized by axon dysfunction,axon degeneration,and neuronal cell death.The roster of neurodegenerative diseases is striking in the selective vulnerability of the affected neuronal populations and the timing of the onset of disease manifestation and functional deficits. endoplasmic reticulum neuronal degeneration manifestation striking populations retina extreme elusive 11-5422/R ObjectType-Article-1 SourceType-Scholarly Journals-1 ObjectType-Feature-2 content type line 23
ISSN:	1673-5374 1876-7958
DOI:	10.4103/1673-5374.162696