A systematic review of dietary acids and habits on dental erosion in adolescents

Background Dental erosion is the dissolution of dental hard tissues caused by acids of a non‐bacterial origin. Dietary acids are considered the predominant and most controllable factor. Aim To synthesise the literature on the effects of dietary acids and habits on dental erosion in the permanent den...

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Published in:International journal of paediatric dentistry Vol. 30; no. 6; pp. 713 - 733
Main Authors: Chan, Alex Simon, Tran, Theresa Thien Kim, Hsu, Yi Hsuan, Liu, Stephanie Yue Sum, Kroon, Jeroen
Format: Journal Article
Language:English
Published: England Blackwell Publishing Ltd 01-11-2020
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Summary:Background Dental erosion is the dissolution of dental hard tissues caused by acids of a non‐bacterial origin. Dietary acids are considered the predominant and most controllable factor. Aim To synthesise the literature on the effects of dietary acids and habits on dental erosion in the permanent dentition of 10‐ to 19‐year‐old adolescents. Materials and Methods An electronic literature search was undertaken in Cochrane, MEDLINE, CINAHL, Dentistry & Oral Sciences Source via EBSCOhost, and Embase with no restriction on the date of publication. Results The initial search identified 449 articles, and 338 remained after removal of duplicates. Seventy‐seven articles remained after screening of titles and s, and 52 were eligible for the full‐text review. A considerable variety of beverages, food, and dietary habits were reported as risk factors for dental erosion. The most consistent findings implicated the erosive potential of carbonated beverages and the consumption of acidic drinks at bedtime. Conclusions Although results were not consistent between cohort and cross‐sectional studies, this review suggests certain dietary risk factors may contribute to dental erosion in adolescents. There is a need for more high‐quality cohort studies to establish more conclusive evidence on the role of dietary acids and habits on dental erosion.
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ISSN:0960-7439
1365-263X
DOI:10.1111/ipd.12643