Risk factors for rapid kidney function decline in diabetes patients

Diabetic nephropathy, as a severe microvascular complication of diabetes, manifests in four clinical types: classic, albuminuria regression, a rapid decline in kidney function (RDKF), and non-proteinuric or non-albuminuric DKD. Rapidly progressive diabetic nephropathy advances to end-stage renal dis...

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Bibliographic Details
Published in:	Renal failure Vol. 46; no. 2; p. 2398188
Main Authors:	Xing, Jixin, Huang, Linxi, Ren, Weifu, Mei, Xiaobin
Format:	Journal Article
Language:	English
Published:	England Taylor & Francis 01-12-2024 Taylor & Francis Group
Subjects:	Albuminuria - etiology Chronic Kidney Disease and Progression clinical risk factors Diabetic Nephropathies - diagnosis Diabetic Nephropathies - etiology Diabetic Nephropathies - physiopathology Diabetic nephropathy Disease Progression genetic factors Glomerular Filtration Rate Humans Kidney - physiopathology Kidney Failure, Chronic - complications Kidney Failure, Chronic - etiology Kidney Failure, Chronic - physiopathology rapid decline in kidney function Review Risk Factors Diabetic nephropathy clinical risk factors rapid decline in kidney function genetic factors
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Summary:	Diabetic nephropathy, as a severe microvascular complication of diabetes, manifests in four clinical types: classic, albuminuria regression, a rapid decline in kidney function (RDKF), and non-proteinuric or non-albuminuric DKD. Rapidly progressive diabetic nephropathy advances to end-stage renal disease more swiftly than the typical form, posing significant risks. However, a comprehensive understanding of rapidly progressive diabetic nephropathy is currently lacking. This article reviewed latest developments in genetic and clinical risk factors associated with rapidly progressive diabetic nephropathy, aiming to broad perspectives concerning the diagnosis and interventions of this condition.
Bibliography:	ObjectType-Article-1 SourceType-Scholarly Journals-1 ObjectType-Feature-2 ObjectType-Review-3 content type line 23 These authors contributed equally to this work.
ISSN:	0886-022X 1525-6049 1525-6049
DOI:	10.1080/0886022X.2024.2398188