Oligomeric and fibrillar species of β-amyloid (Aβ42) both impair mitochondrial function in P301L tau transgenic mice

• Published in: Journal of molecular medicine (Berlin, Germany) Vol. 86; no. 11; pp. 1253 - 1265
• Main Authors: ECKERT, Anne, HAUPTMANN, Susanne, SCHERPING, Isabel, MEINHARDT, Jessica, RHEIN, Virginie, DRÖSE, Stefan, BRANDT, Ulrich, FÄNDRICH, Marcus, MÜLLER, Walter E, GÖTZ, Jürgen
• Format: Journal Article
• Language: English
• Published: Berlin Springer 2008
• Subjects: Biological and medical sciences; Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases; General aspects; Medical sciences; Neurology; Protein aggregation; Peptides; Alzheimer disease; Toxicity; Transgenic animal; Tau; Oligomer; Aggregation; Mitochondria; Degenerative disease; Amyloid; Species; Alzheimer's disease; Nervous system diseases; Fibrils; Rodentia; Transgenic mice; Cerebral disorder; Medicine; Vertebrata; Mammalia; Mouse; β Amyloid protein; Globulomer; Amyloid toxicity; Central nervous system disease; Amyloid aggregates; Aggregate; Frontotemporal dementia; Respiration; Fibril; Amyloid β-peptide
• ISSN: 0946-2716; 1432-1440