Tissue protection by ischemic preconditioning depends mainly on expression of heat shock protein 32

Using the hairless mouse ear skin flap model we demonstrate that HSP-32 expression has to be considered as a significant mechanism of ischemic preconditioning-induced protection of critically perfused tissue.

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Bibliographic Details
Published in:	Langenbecks Archiv fur Chirurgie. Supplement. Kongressband. Deutsche Gesellschaft fur Chirurgie. Kongress Vol. 115; no. Suppl I; p. 251
Main Authors:	Rösken, F, Kubulus, D, Amon, M, Rücker, M, Bauer, I, Menger, M D
Format:	Journal Article
Language:	German
Published:	Germany 1998
Subjects:	Animals Blood Flow Velocity - physiology Graft Survival - physiology Heme Oxygenase (Decyclizing) - physiology Heme Oxygenase-1 Ischemic Preconditioning - methods Laser-Doppler Flowmetry Membrane Proteins Mice Mice, Hairless Microcirculation - physiopathology Microsurgery Surgical Flaps - blood supply
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Summary:	Using the hairless mouse ear skin flap model we demonstrate that HSP-32 expression has to be considered as a significant mechanism of ischemic preconditioning-induced protection of critically perfused tissue.
ISSN:	0942-2854