Phylogeny and Immunoreactivity of Norovirus GII.P16-GII.2, Japan, Winter 2016-17

• Published in: Emerging infectious diseases Vol. 24; no. 1; pp. 144 - 148
• Main Authors: Nagasawa, Koo, Matsushima, Yuki, Motoya, Takumi, Mizukoshi, Fuminori, Ueki, Yo, Sakon, Naomi, Murakami, Koichi, Shimizu, Tomomi, Okabe, Nobuhiko, Nagata, Noriko, Shirabe, Komei, Shinomiya, Hiroto, Suzuki, Wataru, Kuroda, Makoto, Sekizuka, Tsuyoshi, Ryo, Akihide, Fujita, Kiyotaka, Oishi, Kazunori, Katayama, Kazuhiko, Kimura, Hirokazu
• Format: Journal Article
• Language: English
• Published: United States U.S. National Center for Infectious Diseases 01-01-2018; Centers for Disease Control and Prevention
• Subjects: Adolescent; Analysis; Caliciviridae Infections - epidemiology; Caliciviridae Infections - virology; capsid; Child; Child, Preschool; Communicable diseases; Disease Outbreaks; Dispatch; Gastroenteritis; Gastroenteritis - epidemiology; Gastroenteritis - virology; Humans; immunoreactivity; Infant; Infant, Newborn; Japan - epidemiology; norovirus; Norovirus - genetics; Norovirus - immunology; Phylogeny; RNA-dependent RNA polymerase; Seasons; winter; Young Adult; Japan; immunoreactivity; viruses; norovirus; Japan; RNA-dependent RNA polymerase; capsid; phylogeny; winter
• ISSN: 1080-6040; 1080-6059
• DOI: 10.3201/eid2401.170284

During the 2016-17 winter season in Japan, human norovirus GII.P16-GII.2 strains (2016 strains) caused large outbreaks of acute gastroenteritis. Phylogenetic analyses suggested that the 2016 strains derived from the GII.2 strains detected during 2010-12. Immunochromatography between 2016 strains and the pre-2016 GII.2 strains showed similar reactivity.