Histone H3-K9 Methyltransferase ESET Is Essential for Early Development

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Published in:	Molecular and Cellular Biology Vol. 24; no. 6; pp. 2478 - 2486
Main Authors:	Dodge, Jonathan E., Kang, Yong-Kook, Beppu, Hideyuki, Lei, Hong, Li, En
Format:	Journal Article
Language:	English
Published:	United States American Society for Microbiology 01-03-2004 Taylor & Francis
Subjects:	AG9a gene Animals Base Sequence Blastocyst - cytology Blastocyst - enzymology Cell Line Cell Survival DNA Methylation DNA, Complementary - genetics Embryonic and Fetal Development - genetics Embryonic and Fetal Development - physiology Eset gene Female Fetal Death - enzymology Fetal Death - genetics G9a gene Gene Expression Regulation, Developmental Gene Targeting histone methyltransferase Histone Methyltransferases Histone-Lysine N-Methyltransferase - deficiency Histone-Lysine N-Methyltransferase - genetics Histone-Lysine N-Methyltransferase - physiology Histones - chemistry Histones - metabolism Lysine - chemistry Mage gene Mammalian Genetic Models with Minimal or Complex Phenotypes Methyltransferases - deficiency Methyltransferases - genetics Methyltransferases - physiology Mice Mice, Knockout Phenotype Pregnancy Protein Methyltransferases Suv39h gene
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Summary:	Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit StumbleUpon Twitter current issue Spotlights in the Current Issue MCB About MCB Subscribers Authors Reviewers Advertisers Inquiries from the Press Permissions & Commercial Reprints ASM Journals Public Access Policy MCB RSS Feeds 1752 N Street N.W. • Washington DC 20036 202.737.3600 • 202.942.9355 fax • journals@asmusa.org Print ISSN: 0270-7306 Online ISSN: 1098-5549 Copyright © 2014 by the American Society for Microbiology. For an alternate route to MCB .asm.org, visit: MCB
Bibliography:	ObjectType-Article-2 SourceType-Scholarly Journals-1 ObjectType-Feature-1 content type line 23 ObjectType-Article-1 ObjectType-Feature-2 Corresponding author. Present address: Novartis Institute for Biomedical Research, 100 Technology Sq., Rm. 6654, Cambridge, MA 02139. Phone: (617) 871-7072. Fax: (617) 871-7263. E-mail: en.li@pharma.novartis.com. J.E.D. and Y.-K.K. contributed equally to the present work. Present address: Novartis Institute for Biomedical Research, Cambridge, MA 02139.
ISSN:	0270-7306 1098-5549 1098-5549
DOI:	10.1128/MCB.24.6.2478-2486.2004