Mitochondria Permeability Transition versus Necroptosis in Oxalate-Induced AKI

Serum oxalate levels suddenly increase with certain dietary exposures or ethylene glycol poisoning and are a well known cause of AKI. Established contributors to oxalate crystal-induced renal necroinflammation include the NACHT, LRR and PYD domains-containing protein-3 (NLRP3) inflammasome and mixed...

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Bibliographic Details
Published in:	Journal of the American Society of Nephrology Vol. 30; no. 10; pp. 1857 - 1869
Main Authors:	Mulay, Shrikant Ramesh, Honarpisheh, Mohsen M, Foresto-Neto, Orestes, Shi, Chongxu, Desai, Jyaysi, Zhao, Zhi Bo, Marschner, Julian A, Popper, Bastian, Buhl, Ewa Miriam, Boor, Peter, Linkermann, Andreas, Liapis, Helen, Bilyy, Rostyslav, Herrmann, Martin, Romagnani, Paola, Belevich, Ilya, Jokitalo, Eija, Becker, Jan U, Anders, Hans-Joachim
Format:	Journal Article
Language:	English
Published:	United States American Society of Nephrology 01-10-2019
Subjects:	Acute Kidney Injury - chemically induced Acute Kidney Injury - pathology Animals Basic Research Cells, Cultured Humans Male Mice Mitochondrial Transmembrane Permeability-Driven Necrosis Necroptosis Oxalates - administration & dosage cyclosporine kidney tubule acute renal failure cell biology and structure mitochondria
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