A Serine12Stop mutation in PB1-F2 of the 2009 pandemic (H1N1) influenza A: a possible reason for its enhanced transmission and pathogenicity to humans

As the scientific community scrambles to define the ancestry and lineages of the eight segments of new pandemic H1N1 strain, we looked for unique genetic events in this virus's genome to explain the newly found enhanced virulence and transmissibility among humans. Genome annotations of this vir...

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Bibliographic Details
Published in:	Journal of veterinary science (Suwŏn-si, Korea) Vol. 10; no. 4; pp. 349 - 351
Main Authors:	Ramakrishnan, Muthannan A., University of Minnesota, St. Paul, MN, USA, Gramer, Marie R., University of Minnesota, St. Paul, MN, USA, Goyal, Sagar M., University of Minnesota, St. Paul, MN, USA, Sreevatsan, Srinand, University of Minnesota, St. Paul, MN, USA
Format:	Journal Article
Language:	English
Published:	Korea (South) 대한수의학회 01-12-2009 The Korean Society of Veterinary Science
Subjects:	Amino Acid Sequence Gene Expression Regulation, Viral - physiology Host-Pathogen Interactions Humans Influenza A Virus, H1N1 Subtype - genetics Influenza A Virus, H1N1 Subtype - pathogenicity Influenza, Human - virology INFLUENZAVIRUS Molecular Sequence Data Mutation pandemic influenza A PB1-F2 protein S12Stop Short Communication Viral Proteins - chemistry Viral Proteins - genetics Viral Proteins - metabolism Virulence virulence marker 수의학 PB1-F2 protein pandemic influenza A virulence marker S12Stop
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