Expression of TLR-2, TLR-4, NOD2 and pNF-κB in a Neonatal Rat Model of Necrotizing Enterocolitis

Background The etiology of necrotizing enterocolitis (NEC) results from a combination of several risk factors that act synergistically and occurs in the same circumstances as those which lead to innate immunity activation. Pattern recognition molecules could be an important player in the initiation...

Full description

Saved in:

Bibliographic Details
Published in:	PloS one Vol. 2; no. 10; p. e1102
Main Authors:	Aurelie Le Mandat Schultz, Bonnard, Arnaud, Barreau, Frédérick, Aigrain, Yves, Pierre-Louis, Coralie, Berrebi, Dominique, Peuchmaur, Michel
Format:	Journal Article
Language:	English
Published:	San Francisco Public Library of Science 31-10-2007
Subjects:	Antibodies Apoptosis Breastfeeding & lactation Caspase Caspase-3 Enterocolitis Epithelial cells Etiology Gangrene Gastroenterology and Hepatology/Small Intestine Gastrointestinal diseases Gene expression Gestation Growth factors Hypoxia Immune system Immunity Immunohistochemistry Immunology Immunology/Innate Immunity Inflammation Inflammatory response Injuries Innate immunity Milk Morphology Mucosa Necrosis Necrotizing enterocolitis Neonates Newborn babies NF-κB protein NOD2 protein Paraffin Pathogenesis Pathogens Pathology/Histopathology Pathology/Immunology Pattern recognition Pediatrics and Child Health/Neonatology Polymerase chain reaction Proteins Rats Real time Risk analysis Risk factors Rodents Small intestine Toll-like receptors France
Online Access:	Get full text
Tags:	Add Tag No Tags, Be the first to tag this record!

Be the first to leave a comment!