Supraventricular tachycardia promotes release of troponin I in patients with normal coronary arteries

• Published in: International journal of cardiology Vol. 102; no. 3; pp. 521 - 522
• Main Authors: Redfearn, D.P., Ratib, K., Marshall, H.J., Griffith, M.J.
• Format: Journal Article
• Language: English
• Published: Shannon Elsevier Ireland Ltd 20-07-2005; Elsevier Science
• Subjects: Adult; Aged; Biological and medical sciences; Cardiac dysrhythmias; Cardiology. Vascular system; Cohort Studies; Coronary Vessels - physiology; Female; Heart; Humans; Male; Medical sciences; Middle Aged; Normal coronary arteries; Supraventricular tachycardia; Tachycardia, Supraventricular - metabolism; Troponin I; Troponin I - biosynthesis; Troponin I - blood; Troponin I; Normal coronary arteries; Supraventricular tachycardia; Human; Arrhythmia; Coronary artery; Cardiovascular disease; Paroxysmal supraventricular tachycardia; Normal; Phlebology; Excitability disorder; Heart disease; Circulatory system; Cardiology; Release
• ISSN: 0167-5273; 1874-1754
• DOI: 10.1016/j.ijcard.2004.05.076

Troponin I and T are sensitive markers of myocardial injury. The presence of elevated troponin often prompts further investigation with coronary angiography.
We present a series of patients presenting to our unit with raised troponin I and supraventricular tachycardia (SVT) that prompted coronary angiography.
All patients had normal epicardial vessels at angiogram, no evidence of muscle bridging or pulmonary embolism.
The presence of tachycardia sufficient to warrant hospital admission can raise troponin, and this should be viewed in context when a decision on angiography is to be taken.