Hepatic artery pseudoaneurysm caused by acute idiopathic pancreatitis

Hepatic artery pseudoaneurysm(HAP) is a very rare disease but in cases of complication,there is a very high mortality.The most common cause of HAP is iatrogenic trauma such as liver biopsy,transhepatic biliary drainage,cholecystectomy and hepatectomy.HAP may also occur with complications such as inf...

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Published in:World journal of gastroenterology : WJG Vol. 18; no. 18; pp. 2291 - 2294
Main Authors: Yu, Yeon Hwa, Sohn, Joo Hyun, Kim, Tae Yeob, Jeong, Jae Yoon, Han, Dong Soo, Jeon, Yong Cheol, Kim, Min Young
Format: Journal Article
Language:English
Published: United States Baishideng Publishing Group Co., Limited 14-05-2012
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Summary:Hepatic artery pseudoaneurysm(HAP) is a very rare disease but in cases of complication,there is a very high mortality.The most common cause of HAP is iatrogenic trauma such as liver biopsy,transhepatic biliary drainage,cholecystectomy and hepatectomy.HAP may also occur with complications such as infections or inflammation associated with septic emboli.HAP has been reported rarely in patients with acute pancreatitis.As far as we are aware,there is no report of a case caused by acute idiopathic pancreatitis,particularly.We report a case of HAP caused by acute idiopathic pancreatitis which developed in a 61-year-old woman.The woman initially presented with acute pancreatitis due to unknown cause.After conservative management,her symptoms seemed to have improved.But eight days after admission,abdominal pain abruptly became worse again.Abdominal computed tomography(CT) was rechecked and it detected a new HAP that was not seen in a previous abdominal CT.Endoscopic retrograde cholangiopancreatography(ERCP) was performed because of a suspicion of hemobilia as a cause of aggravated abdominal pain.ERCP confirmed hemobilia by observing fresh blood clots at the opening of the ampulla and several filling defects in the distal common bile duct on cholangiogram.Without any particular treatment such as embolization or surgical ligation,HAP thrombosed spontaneously.Three months after discharge,abdominal CT demonstrated that HAP in the left lateral segment had disappeared.
Bibliography:14-1219/R
Hepatic artery Pseudoaneurysm Pancreatitis Acute Hemobilia
Hepatic artery pseudoaneurysm(HAP) is a very rare disease but in cases of complication,there is a very high mortality.The most common cause of HAP is iatrogenic trauma such as liver biopsy,transhepatic biliary drainage,cholecystectomy and hepatectomy.HAP may also occur with complications such as infections or inflammation associated with septic emboli.HAP has been reported rarely in patients with acute pancreatitis.As far as we are aware,there is no report of a case caused by acute idiopathic pancreatitis,particularly.We report a case of HAP caused by acute idiopathic pancreatitis which developed in a 61-year-old woman.The woman initially presented with acute pancreatitis due to unknown cause.After conservative management,her symptoms seemed to have improved.But eight days after admission,abdominal pain abruptly became worse again.Abdominal computed tomography(CT) was rechecked and it detected a new HAP that was not seen in a previous abdominal CT.Endoscopic retrograde cholangiopancreatography(ERCP) was performed because of a suspicion of hemobilia as a cause of aggravated abdominal pain.ERCP confirmed hemobilia by observing fresh blood clots at the opening of the ampulla and several filling defects in the distal common bile duct on cholangiogram.Without any particular treatment such as embolization or surgical ligation,HAP thrombosed spontaneously.Three months after discharge,abdominal CT demonstrated that HAP in the left lateral segment had disappeared.
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Author contributions: Yu YH, Sohn JH and Kim TY made substantial contributions to conception and design; Jeong JY, Han DS and Jeon YC contributed to endoscopic procedures; Kim MY provided discussion of the radiology; Yu YH wrote the paper; and all authors approved the version to be published.
Telephone: +82-31-5602225 Fax: +82-31-5552998
Correspondence to: Joo Hyun Sohn, MD, Department of Internal Medicine, Hanyang University Guri Hospital, 249-1, Gyomun-Dong, Guri, Gyunggi-Do 471-701, South Korea. sonjh@hanyang.ac.kr
ISSN:1007-9327
2219-2840
DOI:10.3748/wjg.v18.i18.2291