Neurocognitive and Synaptic Potentiation Deficits Are Mitigated by Inhibition of HIF1a Signaling following Intermittent Hypoxia in Rodents

Neurocognitive and Synaptic Potentiation Deficits Are Mitigated by Inhibition of HIF1a Signaling following Intermittent Hypoxia in Rodents

A HIF1a-Dependent Pro-Oxidant State Disrupts Synaptic Plasticity and Impairs Spatial Memory in Response to Intermittent Hypoxia. Alejandra Arias-Cavieres, Maggie A. Khuu, Chinwendu U. Nwakudu, Jasmine E. Barnard, Gokhan Dalgin and Alfredo J. Garcia III.

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Bibliographic Details
Published in:eNeuro Vol. 7; no. 6; p. ENEURO.0449-20.2020
Main Author: Carney, DPhil, Rosalind S E
Format: Journal Article
Language:English
Published: United States Society for Neuroscience 01-11-2020
Subjects:
Animals
Hippocampus
Hypoxia
Neuronal Plasticity
Reactive Oxygen Species
Rodentia
Spatial Memory
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Description
Summary:A HIF1a-Dependent Pro-Oxidant State Disrupts Synaptic Plasticity and Impairs Spatial Memory in Response to Intermittent Hypoxia. Alejandra Arias-Cavieres, Maggie A. Khuu, Chinwendu U. Nwakudu, Jasmine E. Barnard, Gokhan Dalgin and Alfredo J. Garcia III.
Bibliography:ObjectType-Article-2
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ISSN:2373-2822
2373-2822
DOI:10.1523/ENEURO.0449-20.2020