Glucose- and Interleukin-1β-Induced β-Cell Apoptosis Requires Ca2+ Influx and Extracellular Signal-Regulated Kinase (ERK) 1/2 Activation and Is Prevented by a Sulfonylurea Receptor 1/Inwardly Rectifying K+ Channel 6.2 (SUR/Kir6.2) Selective Potassium Channel Opener in Human Islets

Glucose- and Interleukin-1β-Induced β-Cell Apoptosis Requires Ca 2+ Influx and Extracellular Signal-Regulated Kinase (ERK) 1/2 Activation and Is Prevented by a Sulfonylurea Receptor 1/Inwardly Rectifying K + Channel 6.2 (SUR/Kir6.2) Selective Potassium Channel Opener in Human Islets Kathrin Maedler...

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Bibliographic Details
Published in:	Diabetes (New York, N.Y.) Vol. 53; no. 7; pp. 1706 - 1713
Main Authors:	MAEDLER, Kathrin, STØRLING, Joachim, STURIS, Jeppe, ZUELLIG, Richard A, SPINAS, Giatgen A, ARKHAMMAR, Per O. G, MANDRUP-POULSEN, Thomas, DONATH, Marc Y
Format:	Journal Article
Language:	English
Published:	Alexandria, VA American Diabetes Association 01-07-2004
Subjects:	Biological and medical sciences Diabetes. Impaired glucose tolerance Endocrine pancreas Endocrine pancreas. Apud cells (diseases) Endocrinopathies Fundamental and applied biological sciences. Psychology Hormones. Régulation Medical sciences Vertebrates: endocrinology Endocrinopathy Human Calcium Extracellular signal-regulated protein kinase Enzyme Diabetes mellitus Cytokine Interleukin 1β Ionic channel Glucose Inorganic element Micronutrient Cell death Sulfonylureas β Cell Endocrine pancreas Apoptosis Biological receptor
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