No evidence for altered muscle mitochondrial function in Parkinson's disease

Recent reports indicate that reductions in mitochondrial respiratory chain function occur in substantia nigra, platelets, and muscle from patients with Parkinson's disease. To confirm and further characterise the presence of a generally distributed mitochondrial defect, mitochondrial metabolism...

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Bibliographic Details
Published in:	Journal of neurology, neurosurgery and psychiatry Vol. 56; no. 5; pp. 477 - 480
Main Authors:	Anderson, J J, Bravi, D, Ferrari, R, Davis, T L, Baronti, F, Chase, T N, Dagani, F
Format:	Journal Article
Language:	English
Published:	London BMJ Publishing Group Ltd 01-05-1993 BMJ BMJ Publishing Group LTD
Subjects:	Adult Aged Antiparkinson Agents - therapeutic use Biological and medical sciences Brain Diseases - physiopathology Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases Female Humans Levodopa - therapeutic use Male Medical sciences Middle Aged Mitochondria, Muscle - enzymology Mitochondria, Muscle - metabolism Multienzyme Complexes NADH Dehydrogenase - metabolism Neurology Oxygen Consumption Parkinson Disease - drug therapy Parkinson Disease - metabolism Parkinson Disease - physiopathology Respiratory Function Tests Substantia Nigra - enzymology Substantia Nigra - metabolism Substantia Nigra - physiopathology Human Nervous system diseases Respiratory chain Mitochondria Oxygen consumption Exploration Degenerative disease Striated muscle
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