Search Results - "van Weeren, Pascale"

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    Binding of protein kinase B to the plakin family member periplakin by van den Heuvel, A Pieter J, de Vries-Smits, Alida M M, van Weeren, Pascale C, Dijkers, Pascale F, de Bruyn, Kim M T, Riedl, Jürgen A, Burgering, Boudewijn M T

    Published in Journal of cell science (15-10-2002)
    “…The serine/threonine kinase protein kinase B (PKB/c-Akt) acts downstream of the lipid kinase phosphoinositide 3-kinase (PI3K) and functions as an essential…”
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    Journal Article
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    Involvement of E-cadherin and β-catenin in germ cell tumours and in normal male fetal germ cell development by Honecker, Friedemann, Kersemaekers, Anne-Marie F, Molier, Michel, van Weeren, Pascale C, Stoop, Hans, de Krijger, Ronald R, Wolffenbuttel, Katja P, Oosterhuis, Wolter, Bokemeyer, Carsten, Looijenga, Leendert HJ

    Published in The Journal of pathology (01-10-2004)
    “…Intercellular contacts, mediated by E‐cadherin, are essential for germ cell migration and maturation. Furthermore, it has been suggested that decrease or loss…”
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    Involvement of the Fas/FasL pathway in the pathogenesis of germ cell tumours of the adult testis by Kersemaekers, Anne-Marie F., van Weeren, Pascale C., Wolter Oosterhuis, J., Looijenga, Leendert H. J.

    Published in The Journal of pathology (01-04-2002)
    “…Induction of apoptosis by Fas ligand (FasL) of Fas‐containing cells is a known mechanism involved in the eradication of inappropriate cells during normal…”
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    Role of P53 and MDM2 in treatment response of human germ cell tumors by KERSEMAEKERS, Anne-Marie F, MAYER, Frank, MOLIER, Michel, VAN WEEREN, Pascale C, OOSTERHUIS, J. Wolter, BOKEMEYER, Carsten, LOOIJENGA, Leendert H. J

    Published in Journal of clinical oncology (15-03-2002)
    “…Testicular germ cell tumors (TGCTs) of adolescents and adults are very sensitive to systemic treatment. The exquisite chemosensitivity of these cancers has…”
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    Essential role for protein kinase B (PKB) in insulin-induced glycogen synthase kinase 3 inactivation. Characterization of dominant-negative mutant of PKB by van Weeren, P C, de Bruyn, K M, de Vries-Smits, A M, van Lint, J, Burgering, B M

    Published in The Journal of biological chemistry (22-05-1998)
    “…Activation of phosphatidylinositide 3'-OH kinase (PI 3-kinase) is implicated in mediating a variety of growth factor-induced responses, among which are the…”
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    Essential Role for Protein Kinase B (PKB) in Insulin-induced Glycogen Synthase Kinase 3 Inactivation by van Weeren, Pascale C., de Bruyn, Kim M.T., de Vries-Smits, Alida M.M., van Lint, Johan, Burgering, Boudewijn M. Th

    Published in The Journal of biological chemistry (22-05-1998)
    “…Activation of phosphatidylinositide 3′-OH kinase (PI 3-kinase) is implicated in mediating a variety of growth factor-induced responses, among which are the…”
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    Epidermal growth factor induces phosphorylation of extracellular signal-regulated kinase 2 via multiple pathways by BURGERING, B. M. T, DE VRIES-SMITS, A. M. M, MEDEMA, R. H, VAN WEEREN, P. C, TERTOOLEN, L. G. J, BOS, J. L

    Published in Molecular and Cellular Biology (01-12-1993)
    “…Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley…”
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    Mhc-DQB repertoire variation in hominoid and Old World primate species by Otting, N, Kenter, M, van Weeren, P, Jonker, M, Bontrop, RE

    Published in The Journal of immunology (1950) (15-07-1992)
    “…Comparison of 87 distinct Mhc-DQB sequences, obtained from 13 primate species, demonstrates that five out of eight trans-species Mhc-DQB allele lineages are at…”
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    Epidermal Growth Factor Induces Phosphorylation of Extracellular Signal-Regulated Kinase 2 via Multiple Pathways by Burgering, Boudewijn M. T., de Vries-Smits, Alida M. M., Medema, René H., van Weeren, Pascale C., Tertoolen, Leon G.J., Bos, Johannes L.

    Published in Molecular and cellular biology (01-12-1993)
    “…Expression of p21 rasAsn-17 , a dominant negative mutant of p21 ras that blocks p21 ras activation by growth factors, inhibits activation of extracellular…”
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