Search Results - "van Meyel, D"

Disruption of an EAAT-Mediated Chloride Channel in a Drosophila Model of Ataxia by Parinejad, Neda, Peco, Emilie, Ferreira, Tiago, Stacey, Stephanie M, van Meyel, Donald J

“…Patients with Type 6 episodic ataxia (EA6) have mutations of the excitatory amino acid transporter EAAT1 (also known as GLAST), but the underlying…”
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p53 mutation, expression, and DNA ploidy in evolving gliomas: evidence for two pathways of progression by van Meyel, D J, Ramsay, D A, Casson, A G, Keeney, M, Chambers, A F, Cairncross, J G

“…Two lines of evidence indirectly implicate the tumor suppressor p53 (also known as TP53) gene in glioma development. First, germline mutations of the p53 gene…”
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Absence of hereditary mutations in exons 5 through 9 of the p53 gene and exon 24 of the neurofibromin gene in families with glioma by van Meyel, D J, Ramsay, D A, Chambers, A F, Macdonald, D R, Cairncross, J G

“…Inherited mutations of the p53 and neurofibromin genes are thought to cause two distinct neoplastic disorders in which gliomas occur, the Li-Fraumeni syndrome…”
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Genetic background influences timing, morphology and dissemination of lymphomas in p53-deficient mice by van Meyel, D J, Sanchez-Sweatman, O H, Kerkvliet, N, Stitt, L, Ramsay, D A, Khokha, R, Chambers, A F, Cairncross, J G

“…To examine the influence of genetic background on tumorigenesis in p53-deficient mice, we used selective breeding to produce congenic mice with a null p53 gene…”
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Chip and Apterous Physically Interact to Form a Functional Complex during Drosophila Development by van Meyel, Donald J, O'Keefe, David D, Jurata, Linda W, Thor, Stefan, Gill, Gordon N, Thomas, John B

“…LIM homeodomain (LIM-HD) proteins play key roles in a variety of developmental processes throughout the animal kingdom. Here we show that the LIM-binding…”
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Gliomas in families by Ikizler, Y, van Meyel, D J, Ramsay, D A, Abdallah, G L, Allaster, R M, Macdonald, D R, Cavenee, W K, Cairncross, J G

“…This is a descriptive study of 19 families with glial tumors. Twelve were identified prospectively from 178 consecutive, unrelated adults and children with…”
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Chip is an essential cofactor for apterous in the regulation of axon guidance in Drosophila by van Meyel, D J, O'Keefe, D D, Thor, S, Jurata, L W, Gill, G N, Thomas, J B

“…LIM-homeodomain transcription factors are expressed in subsets of neurons and are required for correct axon guidance and neurotransmitter identity. The…”
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Lack of germ-line mutations of CDK4, p16(INK4A), and p15(INK4B) in families with glioma by Gao, L, Liu, L, van Meyel, D, Cairncross, G, Forsyth, P, Kimmel, D, Jenkins, R B, Lassam, N J, Hogg, D

“…Gliomas are tumors of the central nervous system that may be inherited in some patients. The gene(s) responsible for the clustering of gliomas in families have…”
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Loss of heterozygosity analysis of chromosomes 9, 10 and 17 in gliomas in families by Watling, C J, van Meyel, D J, Ramsay, D A, Macdonald, D R, Cairncross, J G

“…Studies of sporadic malignant gliomas have identified structural abnormalities in a number of chromosomal regions, especially losses of DNA on 9p, 10 and 17p…”
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p53 and ras gene expression in human esophageal cancer and Barrett's epithelium: a prospective study by Sorsdahl, K, Casson, A G, Troster, M, Van Meyel, D, Inculet, R, Chambers, A F

“…To assess potential clinical applications for molecular genetic markers associated with human esophageal tumorigenesis, ten patients with primary esophageal…”
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Gliomas in families: Chromosomal analysis by comparative genomic hybridization by Patel, Atul, van Meyel, Donald J., Mohapatra, Gayatry, Bollen, Andrew, Wrensch, Margaret, Gregory Cairncross, J., Feuerstein, Burt G.

“…Gliomas that aggregate in otherwise unremarkable families may have a heritable genetic basis. To determine the spectrum of genetic alterations in…”
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Ssdp proteins bind to LIM-interacting co-factors and regulate the activity of LIM-homeodomain protein complexes in vivo by van Meyel, Donald J, Thomas, John B, Agulnick, Alan D

“…LIM-homeodomain transcription factors control a variety of developmental processes, and are assembled into functional complexes with the LIM-binding co-factor…”
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Lack of germ-line mutations of CDK4, p16INK4A, and p15INK4B in families with glioma by GAO, L, LIU, L, VAN MEYEL, D, CAIRNCROSS, G, FORSYTH, P, KIMMEL, D, JENKINS, R. B, LASSAM, N. J, HOGG, D

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Abstracts of the 6th Canadian Neuro-Oncology Meeting May 18–21, 1994 Lake Louise, Alberta by Fukuyama, Kozo, Matsuzawa, Kazuhito, Hubbard, Sherri Lynn, Dirks, Peter, Rulka, James T., Maisuzawa, K., Hubbard, S. L., Rutka, J. T., Del Maestro, R. F., Vaithilingam, I. S., McDonald, W., Weiss, J. B., Mikkelsen, T., Kohn, E., Nclson, K., Rosenblum, M. L., Guha, Abhijit, Shamah, Steve, Stiles, Charles, Dooley, N. P., Baltuch, G. H., Roslworowski, M., Villemure, J. G., Yong, V. W., Baltuch, G., Rostworowski, M., Couldwell, W. T., Hinton, D. R., Weiss, M. H., Law, R., Couldwell, William T., Hinton, David R., Law, Ron, Weiss, Martin H., Piepmeier, J. M., Pedersen, P. E., Greer, C. A., Dirks, PB, Hubbard, SL, Taghian, A., Budach, W., Freeman, J., Gioioso, D., Suit, H. D., Turner, J., Barron, G., Zia, P., Wong, C. S., Van Dyk, J., Milosevic, M., Laperriere, N. J., Myles, S. T., Lauryssen, C., Shaw, E. G., Scheithauer, B. W., Suman, V., Katzmann, J., Preul, M., Shenouda, G., Langleben, A., Arnold, D., Watling, C., van Meyel, D., Ramsay, D., Cairncross, G., Bahary, J. P., Wainer, I., Pollak, M., Leyland-Jones, B., Tsatoumas, A., Choi, A., Rosenfeld, S. S., Gillespie, G. Y., Gladson, C. L., Drake, J. M., Hoffman, H. J., Humphreys, R. P., Holowka, S., Fullon, D. S., Urtasun, R. C., Hamilton, Mark G., Beals, S., Joganic, E., Spetzler, R., Buckner, J. C., Schaefer, P. L., Dinapolit, R. P., O'Fallon, J. R., Burch, P. A., Chandler, C. L., Hopkins, K., Coakham, H. B., Bullimore, J., Kemshead, J. T., Bernstein, Mark, Laperriere, Normand, MeKenzie, Stephen, Glen, Jennifer, Lee, D., Macdonald, D.

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