Search Results - "del Campo, Jacob J"

The EFF-1A Cytoplasmic Domain Influences Hypodermal Cell Fusions in C. elegans But Is Not Dependent on 14-3-3 Proteins by Shinn-Thomas, Jessica H, del Campo, Jacob J, Wang, Jianjun, Mohler, William A

“…Regulatory and biophysical mechanisms of cell-cell fusion are largely unknown despite the fundamental requirement for fused cells in eukaryotic development…”
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Minimotif Miner: a tool for investigating protein function by Schiller, Martin R, Balla, Sudha, Thapar, Vishal, Verma, Snigdha, Luong, ThaiBinh, Faghri, Tanaz, Huang, Chun-Hsi, Rajasekaran, Sanguthevar, del Campo, Jacob J, Shinn, Jessica H, Mohler, William A, Maciejewski, Mark W, Gryk, Michael R, Piccirillo, Bryan, Schiller, Stanley R

“…In addition to large domains, many short motifs mediate functional post-translational modification of proteins as well as protein-protein interactions and…”
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Fusogenic Activity of EFF-1 Is Regulated via Dynamic Localization in Fusing Somatic Cells of C. elegans by del Campo, Jacob J., Opoku-Serebuoh, Eugene, Isaacson, Ariel B., Scranton, Victoria L., Tucker, Morgan, Han, Min, Mohler, William A.

“…Background: Many animal tissues form via fusion of cells. Yet in all instances of developmental cell fusion, the mechanism underlying fusion of plasma…”
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The Type I Membrane Protein EFF-1 Is Essential for Developmental Cell Fusion by Mohler, William A., Shemer, Gidi, del Campo, Jacob J., Valansi, Clari, Opoku-Serebuoh, Eugene, Scranton, Victoria, Assaf, Nirit, White, John G., Podbilewicz, Benjamin

“…Multinucleate cells are widespread in nature, yet the mechanism by which cells fuse their plasma membranes is poorly understood. To identify animal fusogens,…”
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EVI1 Blocks Apoptosis in DA-1 Myeloid Leukemia Cells Via Enhanced Transcription of the Prosurvival Gene Bcl2a1 (A1) by Perkins, Archibald S., del Campo, Jacob J., Xiao, Ying-Yi, Zhang, Yi, Lin, Sharon J., Dudley, Jonathan, Tuck, David, Yatsula, Bogdan

“…Retroviral insertion at the Evi1 locus causes high level of expression of the gene in myeloid neoplasms in mice and in nonmalignant expansions of myeloid cells…”
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