Search Results - "Zhu, Xinzhou"
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RBM3 promotes neurogenesis in a niche-dependent manner via IMP2-IGF2 signaling pathway after hypoxic-ischemic brain injury
Published in Nature communications (04-09-2019)“…Hypoxic ischemia (HI) is an acute brain threat across all age groups. Therapeutic hypothermia ameliorates resulting injury in neonates but its side effects…”
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The immunomodulatory mechanism of acupuncture treatment for ischemic stroke: research progress, prospects, and future direction
Published in Frontiers in immunology (02-05-2024)“…Ischemic stroke (IS) is one of the leading causes of death and disability. Complicated mechanisms are involved in the pathogenesis of IS. Immunomodulatory…”
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Versatile strategies for adult neurogenesis: avenues to repair the injured brain
Published in Neural regeneration research (01-04-2024)“…Brain injuries due to trauma or stroke are major causes of adult death and disability. Unfortunately, few interventions are effective for post-injury repair of…”
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Corrigendum: The immunomodulatory mechanism of acupuncture treatment for ischemic stroke: research progress, prospects, and future direction
Published in Frontiers in immunology (2024)“…[This corrects the article DOI: 10.3389/fimmu.2024.1319863.]…”
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The Trithorax group protein ASH1 requires a combination of BAH domain and AT hooks, but not the SET domain, for mitotic chromatin binding and survival
Published in Chromosoma (01-09-2021)“…The Drosophila Trithorax group (TrxG) protein ASH1 remains associated with mitotic chromatin through mechanisms that are poorly understood. ASH1 dimethylates…”
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The Impact of Gut Microbiota on Post-Stroke Management
Published in Frontiers in cellular and infection microbiology (12-10-2021)Get full text
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The RNA-Binding Protein RBM3 Promotes Neural Stem Cell (NSC) Proliferation Under Hypoxia
Published in Frontiers in cell and developmental biology (21-11-2019)“…Neural stem cells (NSCs) reside physiologically in a hypoxic niche to maintain self-renewal and multipotency. Whereas mild hypoxia is known to promote NSC…”
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Cold-inducible proteins CIRP and RBM3, a unique couple with activities far beyond the cold
Published in Cellular and molecular life sciences : CMLS (01-10-2016)“…Cold-inducible RNA-binding protein (CIRP) and RNA-binding motif protein 3 (RBM3) are two evolutionarily conserved RNA-binding proteins that are…”
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TREM2 promotes glioma progression and angiogenesis mediated by microglia/brain macrophages
Published in Glia (01-11-2023)“…Triggering receptor expressed on myeloid cell 2 (TREM2), a myeloid cell‐specific signaling molecule, controls essential functions of microglia and impacts on…”
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RBM3 Promotes Anti-inflammatory Responses in Microglia and Serves as a Neuroprotective Target of Ischemic Stroke
Published in Molecular neurobiology (01-10-2024)“…Ischemic stroke is a major cause of death and disability in adults. Hypothermic treatment is successful in treating neonatal cerebral ischemia, but its…”
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Cold‐inducible RBM3 inhibits PERK phosphorylation through cooperation with NF90 to protect cells from endoplasmic reticulum stress
Published in The FASEB journal (01-02-2016)“…The cold‐inducible RNA‐binding motif protein 3 (RBM3) is involved in the protection of neurons in hypoxic‐ischemic and neurodegenerative disorders. RBM3…”
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Visualization of Protein-protein Interaction in Nuclear and Cytoplasmic Fractions by Co-immunoprecipitation and In Situ Proximity Ligation Assay
Published in Journal of visualized experiments (16-01-2017)“…Protein-protein interactions are involved in thousands of cellular processes and occur in distinct spatial context. Traditionally, co-immunoprecipitation is a…”
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The Analysis of Neurovascular Remodeling in Entorhino-hippocampal Organotypic Slice Cultures
Published in Journal of visualized experiments (23-10-2014)Get full text
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The analysis of neurovascular remodeling in entorhino-hippocampal organotypic slice cultures
Published in Journal of visualized experiments (23-10-2014)“…Ischemic brain injury is among the most common and devastating conditions compromising proper brain function and often leads to persisting functional deficits…”
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