Search Results - "Zang, Ming‐Xi"
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1
miR‐29a attenuates cardiac hypertrophy through inhibition of PPARδ expression
Published in Journal of cellular physiology (01-08-2019)“…Although cardiac hypertrophy is widely recognized as a risk factor that leads to cardiac dysfunction and, ultimately, heart failure, the complex mechanisms…”
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2
Induction of the CLOCK gene by E2-ERα signaling promotes the proliferation of breast cancer cells
Published in PloS one (02-05-2014)“…Growing genetic and epidemiological evidence suggests a direct connection between the disruption of circadian rhythm and breast cancer. Moreover, the…”
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3
SUMOylation of AhR modulates its activity and stability through inhibiting its ubiquitination
Published in Journal of cellular physiology (01-12-2012)“…Aryl hydrocarbon receptor (AhR) is a transcription factor that belongs to the basic helix–loop–helix (bHLH) Per‐Arnt‐Sim homology domain (PAS) family. AhR can…”
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4
EGF Protects Cells Against Dox-Induced Growth Arrest Through Activating Cyclin D1 Expression
Published in Journal of cellular biochemistry (01-08-2015)“…ABSTRACT It has been reported that the antitumor drug doxorubicin (Dox) exerts its toxic effects via GATA‐4 depletion and that over‐expression of GATA‐4…”
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5
Insulin induces C2C12 cell proliferation and apoptosis through regulation of cyclin D1 and BAD expression
Published in Journal of cellular biochemistry (01-12-2013)“…ABSTRACT Insulin is a secreted peptide hormone identified in human pancreas to promote glucose utilization. Insulin has been observed to induce cell…”
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6
Effects of Doxorubicin and Fenofibrate on the Activities of NADH Oxidase and Citrate Synthase in Mice
Published in Basic & clinical pharmacology & toxicology (01-12-2011)“…: Doxorubicin (Dox) has widely been used as an anticancer drug, but its use is limited by serious toxicity to the heart, kidney and liver. Mitochondrial…”
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7
Insulin induces proliferation and cardiac differentiation of P19CL6 cells in a dose‐dependent manner
Published in Development, growth & differentiation (01-09-2013)“…Insulin is a peptide hormone produced by beta cells of the pancreas. The roles of insulin in energy metabolism have been well studied, with most of the…”
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8
GSK-3β-mediated activation of NLRP3 inflammasome leads to pyroptosis and apoptosis of rat cardiomyocytes and fibroblasts
Published in European journal of pharmacology (05-04-2022)“…We previously demonstrated that GSK-3β mediates NLRP3 inflammasome activation and IL-1β production in cardiac fibroblasts (CFs) after myocardial infarction…”
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9
Circ-sh3rf3/GATA-4/miR-29a regulatory axis in fibroblast–myofibroblast differentiation and myocardial fibrosis
Published in Cellular and molecular life sciences : CMLS (01-02-2023)“…The transdifferentiation from cardiac fibroblasts to myofibroblasts is an important event in the initiation of cardiac fibrosis. However, the underlying…”
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10
Cooperative activation of atrial naturetic peptide promoter by dHAND and MEF2C
Published in Journal of cellular biochemistry (15-12-2004)“…An intricate array of cell‐specific multiprotein complexes participate in programs of cell‐specific gene expression through combinatorial interaction with…”
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11
A GRIP-1–EZH2 switch binding to GATA-4 is linked to the genesis of rhabdomyosarcoma through miR-29a
Published in Oncogene (02-12-2022)“…Terminal differentiation failure is an important cause of rhabdomyosarcoma genesis, however, little is known about the epigenetic regulation of aberrant…”
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12
Circular RNA expression in isoproterenol hydrochloride-induced cardiac hypertrophy
Published in Aging (Albany, NY.) (05-02-2020)“…Circular RNA (circRNA) is a novel class of noncoding RNAs, and the roles of circRNAs in the development of cardiac hypertrophy remain to be explored. Here, we…”
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13
Cooperative Interaction between the Basic Helix-loop-helix Transcription Factor dHAND and Myocyte Enhancer Factor 2C Regulates Myocardial Gene Expression
Published in The Journal of biological chemistry (24-12-2004)“…Cardiac-restricted transcription factors dHAND and myocyte enhancer factor 2C are expressed in the developing heart and activate several cardiac promoters…”
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14
miR-200b targets GATA-4 during cell growth and differentiation
Published in RNA biology (01-04-2013)“…GATA-4 is an important transcription factor involved in several developmental processes of the heart, such as cardiac myocyte proliferation, differentiation…”
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15
EGF is required for cardiac differentiation of P19CL6 cells through interaction with GATA-4 in a time- and dose-dependent manner
Published in Cellular and molecular life sciences : CMLS (01-05-2015)“…The regulation of cardiac differentiation is critical for maintaining normal cardiac development and function. The precise mechanisms whereby cardiac…”
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16
Transcription Factor GATA-6 Recruits PPARα to Cooperatively Activate Glut4 Gene Expression
Published in Journal of molecular biology (06-01-2012)“…Peroxisome proliferator-activated receptor α (PPARα) is a nuclear hormone receptor that regulates energy metabolism, but its precise mechanisms remain unknown…”
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17
Improvement of myocardial lipid accumulation and prevention of PGC-1α induction by fenofibrate
Published in Molecular medicine reports (01-06-2012)“…We recently demonstrated that fenofibrate induces the activities of citrate synthase and NADH oxidase in cardiac mitochondria. To further determine the…”
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18
Induction of the CLOCK Gene by E2-ER[alpha] Signaling Promotes the Proliferation of Breast Cancer Cells
Published in PloS one (02-05-2014)“…Growing genetic and epidemiological evidence suggests a direct connection between the disruption of circadian rhythm and breast cancer. Moreover, the…”
Get full text
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19
Insulin induces C2 C 12 cell proliferation and apoptosis through regulation of cyclin D1 and BAD expression
Published in Journal of cellular biochemistry (01-12-2013)Get full text
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20
Transcription Factor GATA-6 Recruits PPARI+/- to Cooperatively Activate Glut4 Gene Expression
Published in Journal of molecular biology (06-01-2012)“…Peroxisome proliferator-activated receptor alpha (PPAR alpha ) is a nuclear hormone receptor that regulates energy metabolism, but its precise mechanisms…”
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