Search Results - "Yu, Kunwu"
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Ablation of CXCR4 expression in cardiomyocytes exacerbates isoproterenol‑induced cell death and heart failure
Published in International journal of molecular medicine (01-02-2023)“…CXCR4 is a seven‑transmembrane‑spanning Gi‑coupled receptor for the SDF‑1 chemokine and plays a critical role in cardiovascular development and post‑injury…”
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Exogenous interleukin 37 ameliorates atherosclerosis via inducing the Treg response in ApoE-deficient mice
Published in Scientific reports (12-06-2017)“…Our previous study indicated that interleukin (IL)-37 is involved in atherosclerosis. In the present study, Anterior tibial arteries were collected from…”
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Primary Aldosteronism Influences Cardiac Structure, Function, and Disease Risk: Evidence From Mendelian Randomization Analysis
Published in The journal of clinical hypertension (Greenwich, Conn.) (01-11-2024)“…ABSTRACT Although observational studies have linked primary aldosteronism (PA) with cardiovascular diseases (CVDs), the causality remains uncertain. In this…”
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Editorial: Inflammatory factors in coronary heart disease: mechanism, diagnosis, and therapy
Published in Frontiers in cardiovascular medicine (25-09-2023)Get full text
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Interleukin-27 Ameliorates Atherosclerosis in ApoE-/- Mice through Regulatory T Cell Augmentation and Dendritic Cell Tolerance
Published in Mediators of inflammation (11-11-2022)“…Atherosclerosis, which is characterized by chronic inflammation in the arterial wall, is driven by immune cells and cytokines. Recent evidence indicated that…”
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The IL-2/Anti-IL-2 Complex Attenuates Cardiac Ischaemia-Reperfusion Injury Through Expansion of Regulatory T Cells
Published in Cellular physiology and biochemistry (01-01-2017)“…Background/Aims: Regulatory T cells (Tregs) can suppress immunologic damage in myocardial ischaemia/reperfusion injury (MIRI), however, the isolation and ex…”
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Galectin-9: A Suppressor of Atherosclerosis?
Published in Frontiers in immunology (04-11-2020)“…It is no longer controversial that atherosclerosis is a vascular wall chronic inflammatory disease mediated by cells of innate and adaptive immunity…”
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Digoxin reduces atherosclerosis in apolipoprotein E‐deficient mice
Published in British journal of pharmacology (01-05-2016)“…Background and Purpose Numerous in vitro studies have suggested that digoxin suppresses inflammation and alters lipid metabolism. However, the effect of dioxin…”
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Interleukin-2/Anti-Interleukin-2 Immune Complex Attenuates Cardiac Remodeling after Myocardial Infarction through Expansion of Regulatory T Cells
Published in Journal of immunology research (01-01-2016)“…CD4+CD25+Foxp3+ regulatory T cells (Treg cells) have protective effects in wound healing and adverse ventricular remodeling after myocardial infarction (MI)…”
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10
Case report: Malignant vasovagal reflex syndrome during percutaneous transcatheter closure of patent foramen ovale
Published in Frontiers in cardiovascular medicine (04-07-2023)“…Malignant vasovagal reflex syndrome can be induced by pulling of cardiac tissue during percutaneous transcatheter closure of patent foramen ovale. In this…”
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Corrigendum: Galectin-9: A Suppressor of Atherosclerosis?
Published in Frontiers in immunology (13-04-2021)“…[This corrects the article DOI: 10.3389/fimmu.2020.604265.]…”
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IL-4-Induced Gene 1: A Potential Player in Myocardial Infarction
Published in Reviews in cardiovascular medicine (01-09-2024)“…Myocardial infarction (MI), a severe outcome of cardiovascular disease, poses a serious threat to human health. Uncontrolled inflammation and excessive…”
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Effects of IL-38 on Macrophages and Myocardial Ischemic Injury
Published in Frontiers in immunology (13-05-2022)“…Macrophages play an important role in clearing necrotic myocardial tissues, myocardial ischemia-reperfusion injury, and ventricular remodeling after myocardial…”
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Elevated Plasma IL-37, IL-18, and IL-18BP Concentrations in Patients with Acute Coronary Syndrome
Published in Mediators of Inflammation (01-01-2014)“…Objective. More recently, evidence showed that the novel anti-inflammatory cytokine interleukin- (IL-) 37 was expressed in the foam-like cells of…”
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IL-37 Plays a Beneficial Role in Patients with Acute Coronary Syndrome
Published in Mediators of inflammation (2019)“…Background. Interleukin-37 (IL-37) acts as an inhibitor of innate and adaptive immunity. However, the exact role of IL-37 in the patients with acute coronary…”
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MicroRNA-208a Dysregulates Apoptosis Genes Expression and Promotes Cardiomyocyte Apoptosis during Ischemia and Its Silencing Improves Cardiac Function after Myocardial Infarction
Published in Mediators of Inflammation (01-01-2015)“…Aims. miR-208a is associated with adverse outcomes in several cardiac pathologies known to have increased apoptosis, including myocardial infarction (MI). We…”
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Interleukin‐37 and Dendritic Cells Treated With Interleukin‐37 Plus Troponin I Ameliorate Cardiac Remodeling After Myocardial Infarction
Published in Journal of the American Heart Association (01-12-2016)“…Background Excessive immune‐mediated inflammatory reactions play a deleterious role in postinfarction ventricular remodeling. Interleukin‐37 (IL‐37) emerges as…”
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Elevated Plasma IL-38 Concentrations in Patients with Acute ST-Segment Elevation Myocardial Infarction and Their Dynamics after Reperfusion Treatment
Published in Mediators of Inflammation (01-01-2015)“…Objective. Recent studies suggest that IL-38 is associated with autoimmune diseases. Furthermore, IL-38 is expressed in human atheromatous plaque. However, the…”
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Type 2 Diabetes, Circulating Metabolites, and Calcific Aortic Valve Stenosis: A Mendelian Randomization Study
Published in Metabolites (13-07-2024)“…Epidemiological studies have shown an association between type 2 diabetes (T2D) and calcific aortic valve stenosis (CAVS), but the potential causal…”
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Chemerin15-Ameliorated Cardiac Ischemia-Reperfusion Injury Is Associated with the Induction of Alternatively Activated Macrophages
Published in Mediators of inflammation (01-01-2015)“…Chemerin15 (C15), an endogenous anti-inflammatory component, inhibits the activity of neutrophils and macrophages through G protein-coupled receptor ChemR23;…”
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