Search Results - "Xinh, Phan Thi"

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    Down-regulated expression of NPM1 in IMS-M2 cell line by(-)-epigallocatechin-3-gallate by Chi, Hoang Thanh, Ly, Bui Thi Kim, Vu, Hoang Anh, Sato, Yuko, Dung, Phu Chi, Xinh, Phan Thi

    “…Objective:To investigate the inhibited effect of epigallocatechin-3-gallate(EGCG)on the expression of NPM1 in IMS-M2 cells harboring the NPMl…”
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    Spectrum mutations of PRF1, UNC13D, STX11, and STXBP2 genes in Vietnamese patients with hemophagocytic lymphohistiocytosis by Xinh, Phan Thi, Chuong, Ho Quoc, Diem, Truong Pham Hong, Nguyen, Tuan Minh, Van, Nguyen Dinh, Mai Anh, Nguyen Hoang, Nghia, Huynh, Vu, Hoang Anh

    “…Introduction The prevalence of gene mutations in hemophagocytic lymphohistiocytosis (HLH) varied between studies. Thus far, data on the genetic background of…”
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    Spectrum of mutations in the RB1 gene in Vietnamese patients with retinoblastoma by Kiet, Nguyen Cong, Khuong, Le Thai, Minh, Do Duc, Quan, Nguyen Huynh Minh, Xinh, Phan Thi, Trang, Nguyen Ngoc Chau, Luan, Nguyen Thanh, Khai, Nguyen Minh, Vu, Hoang Anh

    Published in Molecular vision (2019)
    “…Retinoblastoma (RB) is a rare childhood malignant disorder caused by the biallelic inactivation of the gene. Early diagnosis and identification of carriers of…”
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    Real-time Quantitative Polymerase Chain Reaction Assay for Detecting 1p and 19q Codeletion in Glioma by Vo, Niem Van Thanh, Truong, Diem Dinh-Kieu, Xinh, Phan Thi, Vu, Hoang Anh

    “…Abstract Background: Glioma is a type of tumor that occurs in the brain and spinal cord. Gliomas begin in the gluey supportive cells (glial cells) that…”
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    Synergistic effect of all-trans retinoic acid in combination with protein kinase C 412 in FMS-like tyrosine kinase 3-mutated acute myeloid leukemia cells by CHI, HOANG THANH, LY, BUI THI KIM, VU, HOANG ANH, SATO, YUKO, DUNG, PHU CHI, XINH, PHAN THI

    Published in Molecular medicine reports (01-05-2015)
    “…Acute myeloid leukemia (AML) is a heterogeneous disease. Numerous molecular abnormalities have been identified in AML and, amongst these, FMS-like tyrosine…”
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    Spectrum of EGFR gene mutations in Vietnamese patients with non-small cell lung cancer by Vu, Hoang Anh, Xinh, Phan Thi, Ha, Hua Thi Ngoc, Hanh, Ngo Thi Tuyet, Bach, Nguyen Duc, Thao, Doan Thi Phuong, Dat, Ngo Quoc, Trung, Nguyen Sao

    Published in Asia-Pacific journal of clinical oncology (01-03-2016)
    “…Aim Epidermal growth factor receptor (EGFR) mutational status is a crucial biomarker for prediction of response to tyrosine kinase inhibitors in patients with…”
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    A recurrent duodenal gastrointestinal stromal tumor with a frameshift mutation resulting in a stop codon in KIT exon 13 by Vu, Hoang Anh, Xinh, Phan Thi, Kikushima, Makoto, Zhu, Yi, Tokuhara, Makoto, Tani, Masayoshi, Shimizu, Toshio, Saito, Kiyoshi, Tokunaga, Katsushi, Sato, Yuko

    Published in Genes chromosomes & cancer (01-02-2005)
    “…Gastrointestinal stromal tumors (GISTs) are a specific and rare subset of human gastrointestinal tract tumors. Most GISTs show gain‐of‐function mutations of…”
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    Spectrum of HBB gene mutations among 696 β–thalassemia patients and carriers in Southern Vietnam by Xinh, Phan Thị, Chuong, Ho Quoc, Ha, Nguyen Thi Thanh, Tram, Huynh Duong Bich, Van Dong, Cao, Thanh, Le Vu Ha, Hoa, Nguyen Thi Hong, Nghia, Huynh, Binh, Nguyen Tan, Dung, Phu Chi, Vu, Hoang Anh

    Published in Molecular biology reports (01-04-2022)
    “…Background Thalassemias are common inherited blood disorders that have been extensively studied in Asia. Thus far, data on mutations of the HBB gene in…”
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    Spectrum of WAS gene mutations in Vietnamese patients with Wiskott-Aldrich syndrome by Chuong, Ho Quoc, Xinh, Phan Thi, Tram, Duong Bich, Ha, Nguyen Thi Thanh, Nguyen, Tuan Minh, Anh, Phan Nguyen Lien, Van, Nguyen Dinh, Anh, Nguyen Hoang Mai, Dung, Phu Chi, Nghia, Huynh, Vu, Hoang Anh

    Published in Pediatrics international (01-01-2024)
    “…WAS gene mutational analysis is crucial to establish a definite diagnosis of Wiskott-Aldrich syndrome (WAS). Data on the genetic background of WAS in…”
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    Clinical and Hematological Relevance of JAK2V617F, CALR, and MPL Mutations in Vietnamese Patients with Essential Thrombocythemia by Vu, Hoang Anh, Thao, Tran Thi, Dong, Cao Van, Vuong, Nguyen Lam, Chuong, Ho Quoc, Van, Phan Nguyen Thanh, Nghia, Huynh, Binh, Nguyen Tan, Dung, Phu Chi, Xinh, Phan Thi

    “…Background: The picture of Vietnamese patients with essential thrombocythemia (ET) remains mostly undetermined. Our study intended to determine the frequency…”
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    ID:2035 Chromosomal abnormalities and treatment response in Multiple myeloma patients at Cho Ray hospital by Suzanne, Thanh Thanh, Le, Oanh Hoang, Phan, Xinh Thi, Tran, Tung Thanh, Nguyen, Cuong Kim, Phan, Thang Thanh, Huynh, Hoa Hong

    Published in Biomedical research and therapy (05-09-2017)
    “…Multiple myeloma (MM) is a malignant plasma cell, generating abnormal immunoglobulins in the blood and urin. From January 2012 to December 2014, we performed…”
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    The juxtamembrane domain in ETV6/FLT3 is critical for PIM-1 up-regulation and cell proliferation by Vu, Hoang Anh, Xinh, Phan Thi, Kano, Yasuhiko, Tokunaga, Katsushi, Sato, Yuko

    “…We recently reported that the ETV6/FLT3 fusion protein conferred interleukin-3-independent growth on Ba/F3 cells. The present study has been conducted to…”
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    Activation of Nuclear Factor KappaB in ETV6/FLT3-Transformed Ba/F3 Cells Depends on the FLT3 Tyrosine 591 by Vu, Hoang Anh, Xinh, Phan Thi, Kano, Yasuhiko, Ishida, Takaomi, Watanabe, Toshiki, Tokunaga, Katsushi, Sato, Yuko

    Published in Blood (16-11-2007)
    “…The FLT3 gene is one of the most frequently mutated genes in hematologic malignancies. Mutated FLT3 has been found to lead to strong constitutive activation of…”
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    MEL1 at 1p36.3 Is Fused to Several Partner Genes Including HOXA9 Located at Different Chromosomal Bands from 3q21.3 in t(1;3)(p36.3;q21.3)-Leukemia by Xinh, Phan Thi, Anh, Hoang, Tanaka, Hideo, Fujisawa, Shin, Taketazu, Fumitoshi, Rowley, Janet D., Tokunaga, Katsushi, Sato, Yuko

    Published in Blood (16-11-2007)
    “…In MDS/AML with t(1;3)(p36;q21), overexpression of MEL1 lacking PR domain (MEL1S) driven by the RPN1 promoter at 3q21.3 is reported to be closely associated…”
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