Search Results - "Weir, DM"
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Cytokine responses and sudden infant death syndrome: genetic, developmental, and environmental risk factors
Published in Journal of leukocyte biology (01-12-2005)“…Despite the success of the campaigns to reduce the risk of sudden infant death syndrome (SIDS), it still remains the major cause of postneonatal mortality. The…”
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2
Increased Inflammatory Responses of Persons of Blood Group O to Helicobacter pylori
Published in The Journal of infectious diseases (01-04-2000)“…Persons of blood group O are at increased risk of peptic ulcers. Enhanced binding of Helicobacter pylori to epithelial cells of persons of blood group O has…”
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3
The role of bacterial toxins in Sudden Infant Death Syndrome (SIDS)
Published in International journal of medical microbiology (01-02-2002)“…There is increasing evidence for the involvement of bacterial toxins in some cases of sudden infant death syndrome (SIDS), particularly the pyrogenic toxins of…”
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Conference Proceeding Journal Article -
4
Neisseria meningitidis, Neisseria lactamica and Moraxella catarrhalis share cross-reactive carbohydrate antigens
Published in Vaccine (17-02-2004)“…Carriage of commensal bacteria species is associated with the development of natural immunity to meningococcal disease, with lipo-oligosaccharides (LOS) of…”
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Risk factors for cot death increase danger from infection
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Blood Group and Susceptibility to Disease Caused by Escherichia coli O157
Published in The Journal of infectious diseases (01-02-2002)“…Patients (n = 186) infected during the Escherichia coli O157 outbreak in Scotland in 1996 were assessed for blood group markers (ABO, Lewis, and P) associated…”
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7
Isolation of a cell surface component of Helicobacter pylori that binds H type 2, Lewis(a), and Lewis(b) antigens
Published in Gastroenterology (New York, N.Y. 1943) (01-04-1997)“…BACKGROUND & AIMS: Individuals of blood group O and nonsecretors of ABO blood group antigens are more susceptible to peptic ulcers. The aim of this study was…”
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Proinflammatory Responses to Lipo-oligosaccharide of Neisseria meningitidis Immunotype Strains in Relation to Virulence and Disease
Published in The Journal of infectious diseases (15-05-2002)“…Inflammatory responses to lipo-oligosaccharide (LOS) contribute to the severity of meningococcal disease. Strains that express the L(3,7,9) LOS immunotypes are…”
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9
Comparison of Moraxella catarrhalis isolates from children and adults for growth on modified New York City medium and potential virulence factors
Published in Journal of medical microbiology (01-10-2003)“…1 Department of Medical Microbiology, University of Edinburgh, Edinburgh, Scotland, UK 2 Institute for Scientific Evaluation of Naturopathy, University of…”
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10
Cytokine release of whole blood from adult female donors challenged with mistletoe lectin-1 standardised mistletoe extract and E. coli endotoxin or phytohaemagglutinin (PHA)
Published in Anticancer research (01-03-2003)“…Cytokine (TNF, alpha, interleukin-6) release of whole blood from healthy donors was challenged with mistletoe extract standardised for mistletoe lectin-1 (sML)…”
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Interleukin-10 and sudden infant death syndrome
Published in FEMS immunology and medical microbiology (01-09-2004)“…Uncontrolled pro-inflammatory responses to infections or bacterial toxins have been suggested to play a role in triggering the physiological events leading to…”
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Bactericidal activity of a monocytic cell line (THP-1) against common respiratory tract bacterial pathogens is depressed after infection with respiratory syncytial virus
Published in Journal of medical microbiology (01-03-2000)“…Department of Medical Microbiology and *Medical Statistics Unit, University of Edinburgh, Teviot Place, Edinburgh EH8 9AG Corresponding author: Dr M. W. Raza…”
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Ethnicity, infection and sudden infant death syndrome
Published in FEMS Immunology and Medical Microbiology (01-09-2004)“…Epidemiological studies found the incidence of SIDS among Indigenous groups such as Aboriginal Australians, New Zealand Maoris and Native Americans were…”
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Book Review Journal Article -
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Why is smoking a risk factor for Sudden Infant Death Syndrome?
Published in Child : care, health & development (01-09-2002)“…Smoking is a major risk factor for both Sudden Infant Death Syndrome (SIDS) and respiratory tract infections. Such infections, both viral and bacterial, also…”
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Interleukin 1-β responses to bacterial toxins and sudden infant death syndrome
Published in FEMS immunology and medical microbiology (01-09-2004)“…We tested the hypothesis that significantly higher IL-1β responses to toxic shock syndrome toxin (TSST) noted for parents of sudden infant death syndrome…”
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The role of infection in sudden infant death syndrome
Published in FEMS immunology and medical microbiology (01-08-1999)“…Abstract Studies on the potential role of infectious agents in sudden infant death syndrome (SIDS) have been published over the years in a variety of journals…”
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The effect of cigarette smoke on adherence of respiratory pathogens to buccal epithelial cells
Published in FEMS immunology and medical microbiology (1999)“…Smoking is associated with an increased risk of respiratory tract infection in adults. In children, exposure to cigarette smoke is a risk factor for…”
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18
Evidence for a genetic component in Sudden Infant Death Syndrome
Published in Child : care, health & development (01-09-2002)“…There is increasing evidence that inflammatory responses have been elicited in some Sudden Infant Death Syndrome (SIDS) infants and that these responses are…”
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Infection, inflammation and sleep: more pieces to the puzzle of sudden infant death syndrome (SIPS)
Published in APMIS : acta pathologica, microbiologica et immunologica Scandinavica (01-05-1999)“…Risk factors for sudden infant death syndrome (SIDS) parallel those for respiratory tract infections; however, infectious agents suggested to be involved in…”
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20
Helicobacter pylori in the mouth--negative isolation from dental plaque and saliva
Published in European journal of gastroenterology & hepatology (01-01-1996)“…To determine the colonization of Helicobacter pylori in dental plaque and saliva of individuals with H. pylori-associated gastritis. One hundred and twenty…”
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