Search Results - "Watzinger, Simon"

The Role of Tenascin C in Cardiac Reverse Remodeling Following Banding-Debanding of the Ascending Aorta by Perera-Gonzalez, Mireia, Kiss, Attila, Kaiser, Philipp, Holzweber, Michael, Nagel, Felix, Watzinger, Simon, Acar, Eylem, Szabo, Petra Lujza, Gonçalves, Inês Fonseca, Weber, Lukas, Pilz, Patrick Michael, Budinsky, Lubos, Helbich, Thomas, Podesser, Bruno Karl

“…Tenascin-C (TN-C) plays a maladaptive role in left ventricular (LV) hypertrophy following pressure overload. However, the role of TN-C in LV regression…”
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Cardiovascular phenotype of the Dmdmdx rat – a suitable animal model for Duchenne muscular dystrophy by Szabó, Petra Lujza, Ebner, Janine, Koenig, Xaver, Hamza, Ouafa, Watzinger, Simon, Trojanek, Sandra, Abraham, Dietmar, Todt, Hannes, Kubista, Helmut, Schicker, Klaus, Remy, Séverine, Anegon, Ignacio, Kiss, Attila, Podesser, Bruno K., Hilber, Karlheinz

“…ABSTRACT Besides skeletal muscle abnormalities, Duchenne muscular dystrophy (DMD) patients present with dilated cardiomyopathy development, which considerably…”
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MicroRNA-30d-5p—A Potential New Therapeutic Target for Prevention of Ischemic Cardiomyopathy after Myocardial Infarction by Boxhammer, Elke, Paar, Vera, Wernly, Bernhard, Kiss, Attila, Mirna, Moritz, Aigner, Achim, Acar, Eylem, Watzinger, Simon, Podesser, Bruno K, Zauner, Roland, Wally, Verena, Ablinger, Michael, Hackl, Matthias, Hoppe, Uta C, Lichtenauer, Michael

“…(1) Background and Objective: MicroRNAs (miRs) are biomarkers for assessing the extent of cardiac remodeling after myocardial infarction (MI) and important…”
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A new player in the game: treatment with antagomiR-21a-5p significantly attenuates histological and echocardiographic effects of experimental autoimmune myocarditis by Mirna, Moritz, Paar, Vera, Topf, Albert, Kraus, Theo, Sotlar, Karl, Aigner, Achim, Ewe, Alexander, Watzinger, Simon, Podesser, Bruno K, Hackl, Matthias, Pistulli, Rudin, Hoppe, Uta C, Kiss, Attila, Lichtenauer, Michael

“…Abstract Aims  Myocarditis is associated with formidable symptoms and increased risk of adverse outcomes. Current approaches mostly rely on symptomatic…”
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Abstract 10973: Tenascin-C Deficiency Rescues the Effect of Diabetes on Cardiac and Vascular Dysfunction in Mice by K, Attila, Arnold, Zsuzsanna, Aykac, Ibrahim, Acar, Eylem, Szabó, Petra L, Fee, Alexander, Szekeres, Maria, Watzinger, Simon, Nádasy, György, Hamdani, Nazha, Podesser, Bruno K

“…IntroductionTenascin-C (TN-C) upregulation is linked to worse clinical outcome in diabetic patients. However, the role of TN-C in the development of diabetic…”
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Remote Ischemic Perconditioning Ameliorates Myocardial Ischemia and Reperfusion-Induced Coronary Endothelial Dysfunction and Aortic Stiffness in Rats by Szabó, Petra Lujza, Dostal, Christopher, Pilz, Patrick Michael, Hamza, Ouafa, Acar, Eylem, Watzinger, Simon, Mathew, Shalett, Kager, Gerd, Hallström, Seth, Podesser, Bruno K, Kiss, Attila

“…Vascular stiffness and endothelial dysfunction are accelerated by acute myocardial infarction (AMI) and subsequently increase the risk for recurrent coronary…”
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Neuregulin-1β Improves Uremic Cardiomyopathy and Renal Dysfunction in Rats by Sárközy, Márta, Watzinger, Simon, Kovács, Zsuzsanna Z.A., Acar, Eylem, Márványkövi, Fanni, Szűcs, Gergő, Lauber, Gülsüm Yilmaz, Galla, Zsolt, Siska, Andrea, Földesi, Imre, Fintha, Attila, Kriston, András, Kovács, Ferenc, Horváth, Péter, Kővári, Bence, Cserni, Gábor, Krenács, Tibor, Szabó, Petra Lujza, Szabó, Gábor Tamás, Monostori, Péter, Zins, Karin, Abraham, Dietmar, Csont, Tamás, Pokreisz, Peter, Podesser, Bruno K., Kiss, Attila

“…[Display omitted] •Uremic cardiomyopathy is characterized by LVH, diastolic dysfunction, and cardiac fibrosis, leading to HF.•Dysregulation of NRG-1β signaling…”
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Cardiovascular phenotype of the Dmd mdx rat - a suitable animal model for Duchenne muscular dystrophy by Szabó, Petra Lujza, Ebner, Janine, Koenig, Xaver, Hamza, Ouafa, Watzinger, Simon, Trojanek, Sandra, Abraham, Dietmar, Todt, Hannes, Kubista, Helmut, Schicker, Klaus, Remy, Séverine, Anegon, Ignacio, Kiss, Attila, Podesser, Bruno K, Hilber, Karlheinz

“…Besides skeletal muscle abnormalities, Duchenne muscular dystrophy (DMD) patients present with dilated cardiomyopathy development, which considerably…”
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