Search Results - "Wasserlauf, Bernard J."

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  1. 1

    A unique pathway of cardiac myocyte death caused by hypoxia-acidosis by Graham, Regina M, Frazier, Donna P, Thompson, John W, Haliko, Shannon, Li, Huifang, Wasserlauf, Bernard J, Spiga, Maria-Grazia, Bishopric, Nanette H, Webster, Keith A

    Published in Journal of experimental biology (01-08-2004)
    “…Chronic hypoxia in the presence of high glucose leads to progressive acidosis of cardiac myocytes in culture. The condition parallels myocardial ischemia in…”
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    Journal Article
  2. 2

    Reperfusion-Activated Akt Kinase Prevents Apoptosis in Transgenic Mouse Hearts Overexpressing Insulin-Like Growth Factor-1 by Yamashita, Kazuhito, Kajstura, Jan, Discher, Daryl J, Wasserlauf, Bernard J, Bishopric, Nanette H, Anversa, Piero, Webster, Keith A

    Published in Circulation research (30-03-2001)
    “…ABSTRACT—Hearts of wild-type and insulin-like growth factor-1 overexpressing (Igf-1) transgenic mice were subjected to Langendorff perfusions and progressive…”
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  3. 3

    Cytoskeletal actin microfilaments and the transient outward potassium current in hypertrophied rat ventriculocytes by Yang, Xiangjun, Pedro, J. I., Pham, Thai V., Wasserlauf, Bernard J., Marcel, J. D. Smets, Myerburg, Robert J., Gelband, Henry, Hoffman, Brian F., Bassett, Arthur L.

    Published in The Journal of physiology (01-06-2002)
    “…The durations of transmembrane action potentials recorded from single myocytes isolated from the endocardial surface of hypertrophied left ventricles of rats…”
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  4. 4

    Reperfusion-activated Akt kinase prevents apoptosis in transgenic mice hearts overexpressing insulin-like growth factor-1 by Yamashita, Kazuhito, Kajstura, Jan, Discher, Daryl J, Wasserlauf, Bernard J

    Published in Circulation research (30-03-2001)
    “…Abstract -Hearts of wild-type and insulin-like growth factor-1 overexpressing (Igf-1(+/-)) transgenic mice were subjected to Langendorff perfusions and…”
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    Journal Article
  5. 5

    Characteristics of I(K) and its response to quinidine in experimental healed myocardial infarction by Yuan, F, Pinto, J M, Li, Q, Wasserlauf, B J, Yang, X, Bassett, A L, Myerburg, R J

    “…Mechanisms and drug treatment of serious ventricular arrhythmias in patients with healed myocardial infarction (HMI) are incompletely understood, in part…”
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  6. 6

    Characteristics of I K and its Response to Quinidine in Experimental Healed Myocardial Infarction by YUAN, FENG, PINTO, JUDITH M.B., LI, Ql, WASSERLAUF, BERNARD J., YANG, XIANGJUN, BASSETT, ARTHUR L., MYERBURG, ROBERT J.

    “…I * , and Quinidine in Healed Infarction. Introduction: Mechanisms and drug treatment of serious ventricular arrhythmias in patients with healed myocardial…”
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  7. 7

    Characteristics of IK and its Response to Quinidine in Experimental Healed Myocardial Infarction by YUAN, FENG, PINTO, JUDITH M.B., LI, Ql, WASSERLAUF, BERNARD J., YANG, XIANGJUN, BASSETT, ARTHUR L., MYERBURG, ROBERT J.

    “…I*, and Quinidine in Healed Infarction. Introduction: Mechanisms and drug treatment of serious ventricular arrhythmias in patients with healed myocardial…”
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    Journal Article
  8. 8

    Regional Gradation of L-Type Calcium Currents in the Feline Heart with a Healed Myocardial Infarct by PINTO, JUDITH M.B., YUAN, FENG, WASSERLAUE, BERNARD J., BASSETT, ARTHUR L., MYERBURG, ROBERT J.

    “…ICa in Healed Myocardial Infarction. Introduction: Abnormal action potentials in myocytes adjacent to > 2‐month‐uld feline LV myocardial infarcts (MI) may…”
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  9. 9

    Hypertrophy Decreases Cardiac KATPChannel Responsiveness to Exogenous and Locally Generated (Glycolytic) ATP by Yuan, Feng, Brandt, Neil R., Pinto, Judith M.B., Wasserlauf, Bernard J., Myerburg, Robert J., Bassett, Arthur L.

    “…This study tests the hypothesis that glycolytic regulation of KATPchannel activity is altered in myocardial hypertrophy. Left ventricular (LV) subendocardial…”
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