Search Results - "WOLF, Matthew J"

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    16S rRNA Gene PCR/Sequencing of Heart Valves for Diagnosis of Infective Endocarditis in Routine Clinical Practice by Hong, Hyo-Lim, Flurin, Laure, Greenwood-Quaintance, Kerryl E, Wolf, Matthew J, Pritt, Bobbi S, Norgan, Andrew P, Patel, Robin

    Published in Journal of clinical microbiology (23-08-2023)
    “…Sequencing is increasingly used for infective endocarditis (IE) diagnosis. Here, the performance of 16S rRNA gene PCR/sequencing of heart valves utilized in…”
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    Journal Article
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    Cell cycle specific, differentially tagged ribosomal proteins to measure phase specific transcriptomes from asynchronously cycling cells by Cochran, Jesse D., Leathers, Tess A., Maldosevic, Emir, Siejda, Klara W., Vitello, Julian, Lee, Haesol, Bradley, Leigh A., Young, Alex, Jomaa, Ahmad, Wolf, Matthew J.

    Published in Scientific reports (18-01-2024)
    “…Asynchronously cycling cells pose a challenge to the accurate characterization of phase-specific gene expression. Current strategies, including RNAseq, survey…”
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    Self-Consistent-Charge Density-Functional Tight-Binding (SCC-DFTB) Parameters for Ceria in 0D to 3D by Kullgren, Jolla, Wolf, Matthew J, Hermansson, Kersti, Köhler, Christof, Aradi, Bálint, Frauenheim, Thomas, Broqvist, Peter

    Published in Journal of physical chemistry. C (02-03-2017)
    “…Reducible oxides such as CeO2 are challenging to describe with standard density-functional theory (DFT) due to the mixed valence states of the cations; they…”
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    Charge transport modelling of perovskite solar cells accounting for non-Boltzmann statistics in organic and highly-doped transport layers by Clarke, Will, Wolf, Matthew J, Walker, Alison, Richardson, Giles

    Published in JPhys Energy (01-04-2023)
    “…We present a drift–diffusion model of a perovskite solar cell (PSC) in which carrier transport in the charge transport layers (TLs) is not based on the…”
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    The Microenvironment of the Pathogenesis of Cardiac Hypertrophy by Bazgir, Farhad, Nau, Julia, Nakhaei-Rad, Saeideh, Amin, Ehsan, Wolf, Matthew J, Saucerman, Jeffry J, Lorenz, Kristina, Ahmadian, Mohammad Reza

    Published in Cells (Basel, Switzerland) (01-07-2023)
    “…Pathological cardiac hypertrophy is a key risk factor for the development of heart failure and predisposes individuals to cardiac arrhythmia and sudden death…”
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    Heart Failure in the Era of Precision Medicine: A Scientific Statement From the American Heart Association by Cresci, Sharon, Pereira, Naveen L, Ahmad, Ferhaan, Byku, Mirnela, de las Fuentes, Lisa, Lanfear, David E, Reilly, Carolyn M, Owens, Anjali T, Wolf, Matthew J

    “…One of 5 people will develop heart failure over his or her lifetime. Early diagnosis and better understanding of the pathophysiology of this disease are…”
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    Cardiac hypertrophy induced by active Raf depends on Yorkie-mediated transcription by Yu, Lin, Daniels, Joseph P, Wu, Huihui, Wolf, Matthew J

    Published in Science signaling (03-02-2015)
    “…Organ hypertrophy can result from enlargement of individual cells or from cell proliferation or both. Activating mutations in the serine-threonine kinase Raf…”
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    Disruption of sarcoendoplasmic reticulum calcium ATPase function in Drosophila leads to cardiac dysfunction by Abraham, Dennis M, Wolf, Matthew J

    Published in PloS one (03-10-2013)
    “…Abnormal sarcoendoplasmic reticulum Calcium ATPase (SERCA) function has been associated with poor cardiac function in humans. While modifiers of SERCA function…”
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    Drosophila as a model for the identification of genes causing adult human heart disease by Wolf, M.J, Amrein, H, Izatt, J.A, Choma, M.A, Reedy, M.C, Rockman, H.A

    “…Drosophila melanogaster genetics provides the advantage of molecularly defined P-element insertions and deletions that span the entire genome. Although…”
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    In Vivo Methods to Monitor Cardiomyocyte Proliferation by Young, Alexander, Bradley, Leigh A, Wolf, Matthew J

    “…Adult mammalian cardiomyocytes demonstrate scarce cycling and even lower proliferation rates in response to injury. Signals that enhance cardiomyocyte…”
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    Suppression of store-operated calcium entry causes dilated cardiomyopathy of the Drosophila heart by Petersen, Courtney E, Wolf, Matthew J, Smyth, Jeremy T

    Published in Biology open (11-03-2020)
    “…Store-operated Ca entry (SOCE) is an essential Ca signaling mechanism present in most animal cells. SOCE refers to Ca influx that is activated by depletion of…”
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    Raf-mediated cardiac hypertrophy in adult Drosophila by Yu, Lin, Daniels, Joseph, Glaser, Alex E, Wolf, Matthew J

    Published in Disease models & mechanisms (01-07-2013)
    “…In response to stress and extracellular signals, the heart undergoes a process called cardiac hypertrophy during which cardiomyocytes increase in size. If…”
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    STM Images of Anionic Defects at CeO2(111)—A Theoretical Perspective by Wolf, Matthew J., Castleton, Christopher W. M., Hermansson, Kersti, Kullgren, Jolla

    Published in Frontiers in chemistry (06-06-2019)
    “…We present a theoretically oriented analysis of the appearance and properties of plausible candidates for the anionic defects observed in scanning tunneling…”
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    Multiscale Modeling of Agglomerated Ceria Nanoparticles: Interface Stability and Oxygen Vacancy Formation by Kim, Byung-Hyun, Kullgren, Jolla, Wolf, Matthew J, Hermansson, Kersti, Broqvist, Peter

    Published in Frontiers in chemistry (22-05-2019)
    “…The interface formation and its effect on redox processes in agglomerated ceria nanoparticles (NPs) have been investigated using a multiscale simulation…”
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    Affecting Rhomboid-3 function causes a dilated heart in adult Drosophila by Yu, Lin, Lee, Teresa, Lin, Na, Wolf, Matthew J

    Published in PLoS genetics (27-05-2010)
    “…Drosophila is a well recognized model of several human diseases, and recent investigations have demonstrated that Drosophila can be used as a model of human…”
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    “HETE”ing up mitochondria in human heart failure by Wolf, Matthew J.

    Published in The Journal of biological chemistry (05-01-2018)
    “…A decade of research has established the phospholipase iPLA2γ as being involved in cardiomyocyte dysfunction and necrosis leading to heart failure, but the…”
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