Search Results - "Vinogradova, T M"
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Modulation of the transient outward current in adult rat ventricular myocytes by polyunsaturated fatty acids
Published in The American journal of physiology (01-02-1998)“…With the whole cell patch-clamp technique, we studied the effects of the n-3 and n-6 polyunsaturated fatty acids (PUFAs), linoleic (C18:2n-6), eicosapentaenoic…”
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Cyclic Variation of Intracellular Calcium: A Critical Factor for Cardiac Pacemaker Cell Dominance
Published in Circulation research (21-02-2003)“…ABSTRACT—While a diversity of cell types and distribution within the sinoatrial node and cell-cell interactions add complexity to a complete elucidation of the…”
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Diastolic Calcium Release Controls the Beating Rate of Rabbit Sinoatrial Node Cells: Numerical Modeling of the Coupling Process
Published in Biophysical journal (01-04-2004)“…Recent studies employing Ca 2+ indicators and confocal microscopy demonstrate substantial local Ca 2+ release beneath the cell plasma membrane (subspace) of…”
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Local Cholinergic Suppression of Pacemaker Activity in the Rabbit Sinoatrial Node
Published in Journal of cardiovascular pharmacology (01-09-1998)“…The effects of transmural vagal stimulation and acetylcholine (ACh) superfusion on primary and latent pacemaker cells of the rabbit sinoatrial node were…”
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Novel Perspectives on the Beating Rate of the Heart
Published in Circulation research (23-08-2002)Get full text
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Sinoatrial node pacemaker activity requires Ca(2+)/calmodulin-dependent protein kinase II activation
Published in Circulation research (27-10-2000)“…Cardiac beating arises from the spontaneous rhythmic excitation of sinoatrial (SA) node cells. Here we report that SA node pacemaker activity is critically…”
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Dual Activation of Phosphodiesterases 3 and 4 Regulates Basal Spontaneous Beating Rate of Cardiac Pacemaker Cells: Role of Compartmentalization?
Published in Frontiers in physiology (09-10-2018)“…Spontaneous firing of sinoatrial (SA) node cells (SANCs) is regulated by cyclic adenosine monophosphate (cAMP)-mediated, protein kinase A (PKA)-dependent…”
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Sinoatrial nodal cell ryanodine receptor and Na+-Ca2+ exchanger: Molecular partners in pacemaker regulation
Published in Circulation research (22-06-2001)Get full text
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Оцінка впливу різних терапевтичних підходів на характер змін уробіому в дітей із рекурентним перебігом інфекції сечової системи
Published in Ukraïnsʹkij žurnal Perinatologìâ ì pedìatrìâ (Online) (01-12-2019)“…Мета — вивчити функціональну динаміку уробіому в дітей із рекурентною інфекцією сечової системи (ІСС) на тлі традиційної й альтернативної терапії у групах…”
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Basal Spontaneous Firing of Rabbit Sinoatrial Node Cells Is Regulated by Dual Activation of PDEs (Phosphodiesterases) 3 and 4
Published in Circulation. Arrhythmia and electrophysiology (01-06-2018)“…BACKGROUND:Spontaneous firing of sinoatrial node cells (SANCs) is regulated by cAMP-mediated, PKA (protein kinase A)-dependent (cAMP/PKA) local subsarcolemmal…”
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High Basal Protein Kinase A–Dependent Phosphorylation Drives Rhythmic Internal Ca2+ Store Oscillations and Spontaneous Beating of Cardiac Pacemaker Cells
Published in Circulation research (03-03-2006)“…Local, rhythmic, subsarcolemmal Ca releases (LCRs) from the sarcoplasmic reticulum (SR) during diastolic depolarization in sinoatrial nodal cells (SANC) occur…”
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Rhythmic Ryanodine Receptor Ca2+ Releases During Diastolic Depolarization of Sinoatrial Pacemaker Cells Do Not Require Membrane Depolarization
Published in Circulation research (02-04-2004)“…ABSTRACT—Localized, subsarcolemmal Ca release (LCR) via ryanodine receptors (RyRs) during diastolic depolarization of sinoatrial nodal cells augments the…”
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β-Adrenergic Stimulation Modulates Ryanodine Receptor Ca2+ Release During Diastolic Depolarization to Accelerate Pacemaker Activity in Rabbit Sinoatrial Nodal Cells
Published in Circulation research (11-01-2002)“…It has long been recognized that activation of sympathetic β-adrenoceptors (β-ARs) increases the spontaneous beating rate of sinoatrial nodal cells (SANCs);…”
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Reorganization of mitotic apparatus in the etoposide-treated CHO-K1 cells precedes apoptotic death
Published in Cell and tissue biology (01-06-2008)“…In a culture of CHO-K1 cells, etoposide (1 h, 25 μM) has been shown to produce interphase arrest, after which the cells resume mitotic division and, after some…”
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The organization of the mitotic apparatus poles in etoposide-treated CHO-K1 cells
Published in T͡S︡itologii͡a (2008)“…In this study, we have examined the organization of the mitotic spindle poles in CHO-K1 cells dividing after treatment with the etoposide (1 h, 25 microM). We…”
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The reorganization of the mitotic apparatus in etoposide-treated CHO-K1 cells precedes accumulation of the apoptotic cells
Published in T͡S︡itologii͡a (2008)“…Etoposide (1 h, 25 microM) causes interphase arrest in CHO-K1 after which cells resume mitotic division and die due to apoptosis after a certain time period…”
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Organization of mitotic apparatus poles in etoposide-treated CHO-K1 cells
Published in Cell and tissue biology (01-06-2008)“…Analyzed in this study is the organization of mitotic spindle poles in CHO-K1 cells dividing after treatment with etoposide (1 h, 25 μM). At various periods…”
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Changes in rhythm of the isolated and intact frog heart with time
Published in Bulletin of experimental biology and medicine (01-10-1990)Get full text
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Sinoatrial Node Pacemaker Activity Requires Ca2+/Calmodulin-Dependent Protein Kinase II Activation
Published in Circulation research (27-10-2000)“…ABSTRACT—Cardiac beating arises from the spontaneous rhythmic excitation of sinoatrial (SA) node cells. Here we report that SA node pacemaker activity is…”
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