Search Results - "Vilas‐Boas, Eloisa A."

Mitochondrial sodium/calcium exchanger (NCLX) regulates basal and starvation‐induced autophagy through calcium signaling by Ramos, Vitor M., Serna, Julian D. C., Vilas‐Boas, Eloisa A., Cabral‐Costa, João Victor, Cunha, Fernanda M., Kataura, Tetsushi, Korolchuk, Viktor I., Kowaltowski, Alicia J.

“…Mitochondria shape intracellular Ca2+ signaling through the concerted activity of Ca2+ uptake via mitochondrial calcium uniporters and efflux by Na+/Ca2+…”
Get full text

A practical and robust method to evaluate metabolic fluxes in primary pancreatic islets by Rocha, Debora S., Manucci, Antonio C., Bruni-Cardoso, Alexandre, Kowaltowski, Alicia J., Vilas-Boas, Eloisa A.

“…Evaluation of mitochondrial oxygen consumption and ATP production is important to investigate pancreatic islet pathophysiology. Most studies use cell lines due…”
Get full text

Early Cytokine-Induced Transient NOX2 Activity Is ER Stress-Dependent and Impacts β-Cell Function and Survival by Vilas-Boas, Eloisa A., Carlein, Christopher, Nalbach, Lisa, Almeida, Davidson C., Ampofo, Emmanuel, Carpinelli, Angelo R., Roma, Leticia P., Ortis, Fernanda

“…In type 1 diabetes (T1D) development, proinflammatory cytokines (PIC) released by immune cells lead to increased reactive oxygen species (ROS) production in…”
Get full text

Mitochondrial redox state, bioenergetics, and calcium transport in caloric restriction: A metabolic nexus by Vilas-Boas, Eloisa A., Kowaltowski, Alicia J.

“…Mitochondria congregate central reactions in energy metabolism, many of which involve electron transfer. As such, they are expected to both respond to changes…”
Get full text

Goldilocks calcium concentrations and the regulation of oxidative phosphorylation: Too much, too little, or just right by Vilas-Boas, Eloisa A., Cabral-Costa, João Victor, Ramos, Vitor M., Caldeira da Silva, Camille C., Kowaltowski, Alicia J.

“…Calcium (Ca2+) is a key regulator in diverse intracellular signaling pathways and has long been implicated in metabolic control and mitochondrial function…”
Get full text