Search Results - "Völkl, Thomas M.K."

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  1. 1

    Klinefelter syndrome and mediastinal germ cell tumors by Völkl, Thomas M.K., Langer, Thorsten, Aigner, Thomas, Greess, Holger, Beck, Jörn D., Rauch, Anita M., Dörr, Helmuth G.

    “…Precocious puberty is not a typical manifestation of patients with Klinefelter syndrome (KS). However, there is an increased incidence of mediastinal germ cell…”
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  2. 2

    IGF-I–IGFBP-3–acid-labile subunit (ALS) complex in children and adolescents with classical congenital adrenal hyperplasia due to 21-hydroxylase deficiency (CAH) by Völkl, Thomas M.K, Rauh, Manfred, Schöfl, Christof, Dörr, Helmuth G

    Published in Growth hormone & IGF research (01-08-2011)
    “…Abstract It has been shown that changes in IGF-I and IGFBP levels in children with classical congenital adrenal hyperplasia due to 21-hydroxylase deficiency…”
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  3. 3

    Obesity Among Children and Adolescents With Classic Congenital Adrenal Hyperplasia Due to 21-Hydroxylase Deficiency by Volkl, Thomas M. K, Simm, Diemud, Beier, Christoph, Dorr, Helmuth G

    Published in Pediatrics (Evanston) (01-01-2006)
    “…Congenital adrenal hyperplasia (CAH) due to 21-hydroxylase deficiency is the most common inherited disorder of adrenal steroid biosynthesis. Patients with the…”
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  4. 4

    Birth Size in Neonates with Congenital Adrenal Hyperplasia due to 21-hydroxylase Deficiency by Dörr, Helmuth G., Penger, Theresa, Albrecht, Andrea, Marx, Michaela, Völkl, Thomas M. K.

    “…Classic congenital adrenal hyperplasia (CAH) secondary to 21-hydroxylase deficiency is characterized by increased prenatal adrenal androgen secretion. There…”
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    Altered 24-Hour Blood Pressure Profiles in Children and Adolescents with Classical Congenital Adrenal Hyperplasia due to 21-Hydroxylase Deficiency by Völkl, Thomas M. K., Simm, Diemud, Dötsch, Jörg, Rascher, Wolfgang, Dörr, Helmuth G.

    “…Objective: Children and adolescents with classical congenital adrenal hyperplasia have been shown to be at risk for obesity associated with higher insulin and…”
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  8. 8

    Adrenarche and puberty in children with classic congenital adrenal hyperplasia due to 21-hydroxylase deficiency by Völkl, Thomas M K, Öhl, Lisa, Rauh, Manfred, Schöfl, Christof, Dörr, Helmuth G

    Published in Hormone research in paediatrics (01-01-2011)
    “…There have been only a few studies on adrenarche in girls with classic congenital adrenal hyperplasia (CAH) showing that dehydroepiandrosterone sulfate (DHEAS)…”
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  9. 9

    Does an altered leptin axis play a role in obesity among children and adolescents with classical congenital adrenal hyperplasia due to 21-hydroxylase deficiency? by Völkl, Thomas M K, Simm, Diemud, Körner, Antje, Rascher, Wolfgang, Kiess, Wieland, Kratzsch, Jürgen, Dörr, Helmuth G

    Published in European journal of endocrinology (01-02-2009)
    “…ObjectiveCongenital adrenal hyperplasia (CAH) patients are at a higher risk to develop obesity. The role of leptin in CAH is still controversial. Our study…”
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  10. 10

    McCune-Albright syndrome: clinical picture and natural history in children and adolescents by Völkl, Thomas M K, Dörr, Helmuth G

    “…The classical triad of McCune-Albright syndrome (MAS) consists of polyostotic fibrous dysplasia (FD), skin hyperpigmentation (café-au-lait spots), and…”
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  11. 11

    Adiponectin levels are high in children with classic congenital adrenal hyperplasia (CAH) due to 21-hydroxylase deficiency by Völkl, Thomas MK, Simm, Diemud, Körner, Antje, Kiess, Wieland, Kratzsch, Jürgen, Dörr, Helmuth G

    Published in Acta Paediatrica (01-05-2009)
    “…Objective: It has been shown that adiponectin serves as an insulin‐sensitizing adipokine. Serum concentrations of adiponectin are low in children with obesity,…”
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  12. 12

    Cardiovascular anomalies in children and young adults with Ullrich‐Turner syndrome—the erlangen experience by Völkl, Thomas M. K., Degenhardt, Karin, Koch, Andreas, Simm, Diemud, Dörr, Helmuth G., Singer, Helmut

    Published in Clinical cardiology (Mahwah, N.J.) (01-02-2005)
    “…Background: Females with Ullrich‐Turner syndrome (UTS) have typical clinical features such as short stature, ovarian failure, visible dysmorphic stigmata, and…”
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  13. 13

    Cell Death Induced by Uridine 5'-triphosphate (UTP) in Contrast to Adenosine 5'-triphosphate (ATP) in Human Epidermoid Carcinoma Cells (A-431) by Völkl, Thomas, Ogilvie, Alexandra, Neuhuber, Winfried, Ogilvie, Adaling

    Published in Cellular physiology and biochemistry (01-01-2008)
    “…Objective: Extracellular ATP has been reported as an important signaling molecule mediating quite divergent specific biological effects. Recent clinical trials…”
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  14. 14

    Catch-down growth during infancy of children born small (SGA) or appropriate (AGA) for gestational age with short-statured parents by Völkl, Thomas M.K., Haas, Bettina, Beier, Christoph, Simm, Diemud, Dörr, Helmuth G.

    Published in The Journal of pediatrics (01-06-2006)
    “…We analyzed postnatal growth in children with familial short stature (FSS) with regard to small (SGA) or appropriate (AGA) for gestational age status at birth…”
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  15. 15

    Spontaneous growth hormone secretion and IGF1:IGFBP3 molar ratios in children born small for gestational age (SGA) by Völkl, Thomas M.K., Schwöbel, Katja, Simm, Diemud, Beier, Christoph, Rohrer, Tilman R., Dörr, Helmuth G.

    Published in Growth hormone & IGF research (01-12-2004)
    “…To analyze spontaneous nocturnal GH profiles, IGF1 and IGFBP3 serum levels, as well as IGF1:IGFBP3 molar ratios in SGA children without postnatal catch-up…”
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