Search Results - "Uçucu, Süheyl"

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  1. 1

    Artificial intelligence-driven diagnosis of β-thalassemia minor & iron deficiency anemia using machine learning models by Uçucu, Süheyl, Azik, Fatih

    Published in Journal of medical biochemistry (01-01-2024)
    “…Iron deficiency anemia (IDA) and b-thalassemia minor (BTM) are the two most common causes of microcytic anemia, and although these conditions do not share many…”
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    Journal Article
  2. 2

    Machine learning models can predict the presence of variants in hemoglobin: artificial neural network-based recognition of human hemoglobin variants by HPLC by Uçucu, Süheyl, Karabıyık, Talha, Azik, Fatih Mehmet

    Published in Türk biyokimya dergisi (15-03-2023)
    “…This article presents the use of machine learning techniques such as artificial neural networks, K-nearest neighbors (KNN), naive Bayes, and decision trees in…”
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    Journal Article
  3. 3

    Difficulties in the diagnosis of HbS/beta thalassemia: Really a mild disease? by Uçucu, Süheyl, Karabıyık, Talha, Azik, Fatih

    Published in Journal of medical biochemistry (02-02-2022)
    “…HbS/b cases having clinical, hematologic and electrophoretic similarities cannot be sufficiently distinguished from sickle cell anemia cases and are…”
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    Journal Article
  4. 4

    Comparison of the diagnostic accuracy of CKD-EPI cystatin-C, CKD-EPI creatinine and 24-hour creatinine clearance for estimating GFR: a preliminary study by Uçucu,Süheyl, Ayan,Durmuş, Serin,Erdinç

    Published in Cukurova Medical Journal (01-01-2021)
    “…Purpose: Serum cystatin C level is a specific marker to estimate the glomerular filtration rate (GFR). Due to this specificity, we assume that GFR estimations…”
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    Journal Article
  5. 5

    IVS-II-16 (G>C) (HBB: c.315+16G>C) or IVS-II-666 (C>T) (HBB: c.316-185C>T) Mutations Trigger an Hb S (HBB: c.20A>T)/β+-Thalassemia Phenotype in an Hb S Trait Patient by Uçucu, Süheyl, Karabıyık, Talha, Azik, Fatih M.

    Published in Hemoglobin (04-07-2021)
    “…Sickle cell trait is a medical condition caused by the presence of both mutant Hb S (HBB: c.20A>T) and normal Hb A alleles. Although sickle cell trait is…”
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    Journal Article