Search Results - "Turpeinen, H"

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  1. 1

    The Plasma Level of Proprotein Convertase FURIN in Patients with Suspected Infection in the Emergency Room: A Prospective Cohort Study by Ranta, N., Turpeinen, H., Oksanen, A., Hämäläinen, S., Huttunen, R., Uusitalo‐Seppälä, R., Rintala, E., Aittoniemi, J., Pesu, M.

    Published in Scandinavian journal of immunology (01-12-2015)
    “…There is an increasing need for novel biomarkers that enable better diagnostic and prognostic stratification of patients with suspected infection. A proprotein…”
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    Journal Article
  2. 2

    Toll-Like Receptor Gene Polymorphisms Confer Susceptibility to Graft-Versus-Host Disease in Allogenic Hematopoietic Stem Cell Transplantation by Sivula, J., Cordova, Z. M., Tuimala, J., Jaatinen, T., Partanen, J., Volin, L., Turpeinen, H.

    Published in Scandinavian journal of immunology (01-09-2012)
    “…Graft‐versus‐host disease (GvHD) is a major complication in hematopoietic stem cell transplantation (HSCT). The immune response against gut microbes is thought…”
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    Journal Article
  3. 3

    Minor histocompatibility antigens as determinants for graft-versus-host disease after allogeneic haematopoietic stem cell transplantation by Turpeinen, H., Ojala, P. J., Ojala, K., Miettinen, M., Volin, L., Partanen, J.

    Published in International journal of immunogenetics (01-12-2013)
    “…Summary Minor histocompatibility antigens (minor H antigens) are genetically polymorphic peptides that have been shown to elicit immune response when…”
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    Journal Article
  4. 4

    Determinants of increased exhaled nitric oxide in patients with suspected asthma by Malmberg, L. P., Turpeinen, H., Rytilä, P., Sarna, S., Haahtela, T.

    Published in Allergy (Copenhagen) (01-04-2005)
    “…Exhaled nitric oxide (FENO) has been proposed as a marker of asthmatic inflammation, but it is unclear whether FENO in clinical use selects patients primarily…”
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    Journal Article
  5. 5

    Relation among mannose-binding lectin 2 genotype, β-cell autoantibodies, and risk for type 1 diabetes in Finnish children by Aittoniemi, J, Turpeinen, H, Tiittanen, M, Knip, M, Simell, O, Ilonen, J, Vaarala, O

    Published in Human immunology (01-02-2008)
    “…Summary Mannose-binding lectin (MBL) is a key mediator of innate immunity, the insufficiency of which is caused by point mutations in the MBL2 gene. MBL…”
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    Journal Article
  6. 6

    Genetic similarity of chromosome 6 between patients receiving hematopoietic stem cell transplantation and HLA matched sibling donors by Turpeinen, Hannu, Volin, Liisa, Nikkinen, Lauri, Ojala, Pauli, Palotie, Aarno, Saarela, Janna, Partanen, Jukka

    Published in Haematologica (Roma) (01-04-2009)
    “…1 Research and Development, Finnish Red Cross Blood Service, Helsinki, Finland 2 Department of Medicine, Helsinki University Central Hospital, Helsinki,…”
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  7. 7

    Killer-cell immunoglobulin-like receptor ligand compatibility in the outcome of Finnish unrelated donor hematopoietic stem cell transplantation by Sivula, J, Volin, L, Porkka, K, Vettenranta, K, Itälä, M, Partanen, J, Turpeinen, H

    Published in Transplant immunology (01-07-2007)
    “…Abstract Incompatibility in killer-cell immunoglobulin-like receptor (KIR) ligand between recipient and donor of hematopoietic stem cell transplantation has…”
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    Journal Article
  8. 8

    Heme oxygenase 1 gene polymorphisms and outcome of renal transplantation by Turpeinen, H., Kyllönen, L. E., Parkkinen, J., Laine, J., Salmela, K. T., Partanen, J.

    Published in International Journal of Immunogenetics (01-08-2007)
    “…Summary Heme oxygenase isoenzyme HO‐1 has been linked to several cytoprotective functions with a potentially beneficial role in transplantation. In the present…”
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    Journal Article
  9. 9

    Evidence for linkage to and association with type 1 diabetes at the 3q21 region in the Finnish population by Laine, A-P, Turpeinen, H, Veijola, R, Hermann, R, Simell, O, Knip, M, Ilonen, J

    Published in Genes and immunity (01-01-2006)
    “…IDDM9-region on chromosome 3q has shown suggestive evidence for linkage to type 1 diabetes in some but not all genome scans. We analyzed 22 microsatellite…”
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    Journal Article
  10. 10
  11. 11

    Effect of ice sheet growth and melting on the slip evolution of thrust faults by Turpeinen, Heidi, Hampel, Andrea, Karow, Tobias, Maniatis, Georgios

    Published in Earth and planetary science letters (15-05-2008)
    “…Field investigations suggest that postglacial unloading and rebound led to the formation or re-activation of reverse faults even in continental shields like…”
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    Journal Article
  12. 12

    HLA DR-DQ-encoded genetic determinants of childhood-onset type 1 diabetes in Finland: An analysis of 622 nuclear families by Hermann, R., Turpeinen, H., Laine, A.P., Veijola, R., Knip, M., Simell, O., Sipilä, I., Åkerblom, H.K., Ilonen, J.

    Published in Tissue antigens (01-08-2003)
    “…:  The diabetes predisposing effect of HLA genes is defined by a complex interaction of various haplotypes. We analyzed the disease association of HLA…”
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  13. 13

    Domestic and foreign donor candidates result in differential probability of matching minor histocompatibility antigens - relevance of selection for hematopoietic stem cell transplantation by Turpeinen, H., Volin, L., Partanen, J.

    Published in Tissue antigens (01-03-2009)
    “…Mismatches between patient and donor at minor histocompatibility antigens (minor H antigens) account for most of the genetic component of histocompatibility…”
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    Journal Article
  14. 14

    Association of Genetic Variation in Inducible Costimulator Gene With Outcome of Kidney Transplantation by HAIMILA, Katri, TURPEINEN, Hannu, ALAKULPPI, Noora S, KYLLÖNEN, Lauri E, SALMELA, Kaija T, PARTANEN, Jukka

    Published in Transplantation (15-02-2009)
    “…The closely-linked genes of CD28, cytotoxic T-lymphocyte associated antigen 4 (CTLA4), inducible costimulator (ICOS), and programmed cell death 1 on chromosome…”
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    Journal Article
  15. 15

    Genetic similarity of chromosome 6 between patients receiving haematopoietic stem cell transplantation and HLA-matched sibling donors by Turpeinen, H, Volin, L, Nikkinen, L, Ojala, P, Palotie, A, Saarela, J, Partanen, J

    Published in Bone marrow transplantation (Basingstoke) (01-03-2009)
    “…Background: Matching for HLA genes located in chromosome 6 is required in hematopoietic stem cell transplantation (HSCT) to reduce the incidence of…”
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    Journal Article
  16. 16

    Vitamin D receptor polymorphisms: no association with type 1 diabetes in the Finnish population by TURPEINEN, Hannu, HERMANN, Robert, VAARA, Satu, LAINE, Antti-Pekka, SIMELL, Olli, KNIP, Mikael, VEIJOLA, Riitta, ILONEN, Jorma

    Published in European journal of endocrinology (01-12-2003)
    “…The effect of polymorphisms of the vitamin D receptor (VDR) gene on susceptibility to type 1 diabetes has recently been investigated extensively. Several…”
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    Journal Article
  17. 17

    A linkage analysis of the CTLA4 gene region in Finnish patients with type 1 diabetes by Turpeinen, H., Laine, A. P., Hermann, R., Simell, O., Veijola, R., Knip, M., Ilonen, J.

    Published in European journal of immunogenetics (01-08-2003)
    “…Summary The cytotoxic T‐lymphocyte antigen 4 (CTLA4) region on 2q33 has been shown to be linked to, and associated with, type 1 diabetes (T1D) and suggested to…”
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  18. 18

    Interspliced transcription chimeras: Neglected pathological mechanism infiltrating gene accession queries? by Tolvanen, Martti, Ojala, Pauli J., Törönen, Petri, Anderson, Heidi, Partanen, Jukka, Turpeinen, Hannu

    Published in Journal of biomedical informatics (01-04-2009)
    “…Over half of the DNA of mammalian genomes is transcribed, and one of the emerging enigmas in the field of RNA research is intergenic splicing or transcription…”
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  19. 19

    A homogeneous high-throughput genotyping method based on competitive hybridization by Kiviniemi, Minna, Nurmi, Jussi, Turpeinen, Hannu, Lövgren, Timo, Ilonen, Jorma

    Published in Clinical Biochemistry (01-11-2003)
    “…A reliable high-throughput assay system is necessary for the analysis of the ever-increasing numbers of single-nucleotide polymorphisms (SNP) relevant to…”
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