Search Results - "Thuillez, Christian"

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    Arterial Stiffness Is Regulated by Nitric Oxide and Endothelium-Derived Hyperpolarizing Factor During Changes in Blood Flow in Humans by Bellien, Jeremy, Favre, Julie, Iacob, Michele, Gao, Ji, Thuillez, Christian, Richard, Vincent, Joannidès, Robinson

    Published in Hypertension (Dallas, Tex. 1979) (01-03-2010)
    “…Cytochrome-derived epoxyeicosatrienoic acids may be important endothelium-derived hyperpolarizing factors, opening calcium-activated potassium channels, but…”
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    Journal Article
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    Role of M2-like macrophage recruitment during angiogenic growth factor therapy by Barbay, Virginie, Houssari, Mahmoud, Mekki, Malik, Banquet, Sebastien, Edwards-Lévy, Florence, Henry, Jean-Paul, Dumesnil, Anais, Adriouch, Sahil, Thuillez, Christian, Richard, Vincent, Brakenhielm, Ebba

    Published in Angiogenesis (London) (01-04-2015)
    “…Therapeutic angiogenesis has yet to fulfill its promise for the clinical treatment of ischemic diseases. Given the impact of macrophages during…”
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    Toll-Like Receptors 2-Deficient Mice Are Protected Against Postischemic Coronary Endothelial Dysfunction by Favre, Julie, Musette, Philippe, Douin-Echinard, Victorine, Laude, Karine, Henry, Jean-Paul, Arnal, Jean-François, Thuillez, Christian, Richard, Vincent

    “…OBJECTIVES—Toll-like receptors (TLR) 2 are expressed in cardiac and inflammatory cells, and regulate leukocyte function. Because leukocyte adhesion is a…”
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    Contribution of endothelium-derived hyperpolarizing factors to the regulation of vascular tone in humans by Bellien, Jeremy, Thuillez, Christian, Joannides, Robinson

    Published in Fundamental & clinical pharmacology (01-08-2008)
    “…Endothelium plays a crucial role in the regulation of cardiovascular homeostasis through the release of vasoactive factors. Besides nitric oxide (NO) and…”
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    Mitochondrial impairment contributes to cocaine-induced cardiac dysfunction: Prevention by the targeted antioxidant MitoQ by Vergeade, Aurélia, Mulder, Paul, Vendeville-Dehaudt, Cathy, Estour, François, Fortin, Dominique, Ventura-Clapier, Renée, Thuillez, Christian, Monteil, Christelle

    Published in Free radical biology & medicine (01-09-2010)
    “…The goal of this study was to assess mitochondrial function and ROS production in an experimental model of cocaine-induced cardiac dysfunction. We hypothesized…”
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    Intestinal preconditioning prevents inflammatory response by modulating heme oxygenase-1 expression in endotoxic shock model by Tamion, Fabienne, Richard, Vincent, Renet, Sylvanie, Thuillez, Christian

    “…Gut mucosal injury observed during ischemia-reperfusion is believed to trigger a systemic inflammatory response leading to multiple organ failure. It should be…”
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    Heart Rate Reduction Induced by the If Current Inhibitor Ivabradine Improves Diastolic Function and Attenuates Cardiac Tissue Hypoxia by Fang, Yuehua, Debunne, Manuelle, Vercauteren, Magali, Brakenhielm, Ebba, Richard, Vincent, Lallemand, Francoise, Henry, Jean-Paul, Mulder, Paul, Thuillez, Christian

    Published in Journal of cardiovascular pharmacology (01-03-2012)
    “…AIMSEnhanced heart rate (HR) is a compensatory mechanism in chronic heart failure (CHF), preserving cardiac output, but at the cost of increased left…”
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    Clinical methods for the evaluation of endothelial function - a focus on resistance arteries by Joannides, Robinson, Bellien, Jeremy, Thuillez, Christian

    Published in Fundamental & clinical pharmacology (01-06-2006)
    “…Endothelial dysfunction is a key event in the pathophysiology of cardiovascular diseases and appears as a strong independent predictor of cardiovascular…”
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    Improvement of peripheral endothelial dysfunction by protein tyrosine phosphatase inhibitors in heart failure by VERCAUTEREN, Magali, REMY, Elise, RICHARD, Vincent, DEVAUX, Corinne, DAUTREAUX, Brigitte, HENRY, Jean-Paul, BAUER, Fabrice, MULDER, Paul, VAN HUIJSDUIJNEN, Rob Hooft, BOMBRUN, Agnès, THUILLEZ, Christian

    Published in Circulation (New York, N.Y.) (05-12-2006)
    “…Chronic heart failure (CHF) induces endothelial dysfunction characterized by a decrease in nitric oxide (NO) production in response to flow (flow-mediated…”
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