Search Results - "Teske, Rebecca"

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  1. 1

    Pro-proliferative and inflammatory signaling converge on FoxO1 transcription factor in pulmonary hypertension by Savai, Rajkumar, Al-Tamari, Hamza M, Sedding, Daniel, Kojonazarov, Baktybek, Muecke, Christian, Teske, Rebecca, Capecchi, Mario R, Weissmann, Norbert, Grimminger, Friedrich, Seeger, Werner, Schermuly, Ralph Theo, Pullamsetti, Soni Savai

    Published in Nature medicine (01-11-2014)
    “…Activation of the transcription factor FoxO1 ameliorates vascular remodeling in pulmonary hypertension, pointing to a potential new therapeutic strategy for…”
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    Journal Article
  2. 2

    The Cardiovascular Effects of Treatment with Hydroxychloroquine and Azithromycin by Simmering, Jacob E., Polgreen, Linnea A., Polgreen, Philip M., Teske, Rebecca E., Comellas, Alejandro P., Carter, Barry L.

    Published in Pharmacotherapy (01-09-2020)
    “…Hydroxychloroquine combined with azithromycin has been investigated for activity against coronavirus disease 2019 (COVID‐19), but concerns about adverse…”
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  3. 3
  4. 4

    Inhibition of STAT3 signaling prevents vascular smooth muscle cell proliferation and neointima formation by Daniel, Jan-Marcus, Dutzmann, Jochen, Bielenberg, Wiebke, Widmer-Teske, Rebecca, Gündüz, Dursun, Hamm, Christian W., Sedding, Daniel G.

    Published in Basic research in cardiology (01-05-2012)
    “…Dedifferentiation, migration, and proliferation of resident vascular smooth muscle cells (SMCs) are key components of neointima formation after vascular…”
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  5. 5

    Role of the phosphatase PTEN in early vascular remodeling by Sedding, Daniel G, Widmer-Teske, Rebecca, Mueller, Andreas, Stieger, Philipp, Daniel, Jan-Marcus, Gündüz, Dursun, Pullamsetti, Soni, Nef, Holger, Moellmann, Helge, Troidl, Christian, Hamm, Christian, Braun-Dullaeus, Rüdiger

    Published in PloS one (22-03-2013)
    “…The phosphatase PTEN represents an important physiological inhibitor of phosphatidylinositol-3 kinase (PI3-K)/protein kinase B (Akt) signalling, however, the…”
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    Journal Article
  6. 6

    Effect of Endocervical Specimen Adequacy on Ligase Chain Reaction Detection of Chlamydia trachomatis by LOEFFELHOLZ, Mike J, JIRSA, Sandra J, TESKE, Rebecca K, WOODS, Juliet N

    Published in Journal of Clinical Microbiology (01-11-2001)
    “…Article Usage Stats Services JCM Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley…”
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    Cleaved high-molecular-weight kininogen inhibits neointima formation following vascular injury by Daniel, Jan-Marcus, Reich, Fabian, Dutzmann, Jochen, Weisheit, Simona, Teske, Rebecca, Gündüz, Dursun, Bauersachs, Johann, Preissner, Klaus T, Sedding, Daniel G

    Published in Thrombosis and haemostasis (31-08-2015)
    “…Cleaved high-molecular-weight kininogen (HKa) or its peptide domain 5 (D5) alone exert anti-adhesive properties in vitro related to impeding integrin-mediated…”
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    Journal Article
  10. 10

    Abstract 18368: Role of miR-146a in Angiogenesis and Myocardial Regeneration by Widmer-Teske, Rebecca, Kueppers, Kai, Bielenberg, Wiebke, Daniel, Jan-Marcus, Troidl, Christian, Nef, Holger, Hamm, Christian, Sedding, Daniel

    Published in Circulation (New York, N.Y.) (20-11-2012)
    “…Abstract only Background Myocardial infarction leads to cardiac remodeling and development of heart failure. Myocardial remodeling and regeneration critically…”
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  11. 11

    In vitro spheroid model of placental vasculogenesis: does it work? by Baal, Nelli, Widmer-Teske, Rebecca, McKinnon, Timothy, Preissner, Klaus T, Zygmunt, Marek T

    Published in Laboratory investigation (01-02-2009)
    “…Placental vascular development begins very early in pregnancy and is characterized by construction of a primitive vascular network in a low-oxygen environment…”
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  12. 12

    Abstract 19359: Inhibition of miR-146a Attenuates Neointima Formation by Sedding, Daniel, Korte, Laura, Teske, Rebecca, Donde, Katrin, Bauersachs, Johann

    Published in Circulation (New York, N.Y.) (11-11-2016)
    “…IntroductionDrug-eluting stents are used to prevent in-stent-restenosis. However, anti-proliferative agents delivered lack cell specificity and also impair…”
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