Search Results - "Tejada, Maria A."

Heteromeric Slick/Slack K+ channels show graded sensitivity to cell volume changes by Tejada, Maria A, Hashem, Nadia, Calloe, Kirstine, Klaerke, Dan A

“…Slick and Slack high-conductance K+ channels are found in the CNS, kidneys, pancreas, among other organs, where they play an important role in cell…”
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Cell volume changes regulate slick (Slo2.1), but not slack (Slo2.2) K+ channels by Tejada, Maria A, Stople, Kathleen, Hammami Bomholtz, Sofia, Meinild, Anne-Kristine, Poulsen, Asser Nyander, Klaerke, Dan A

“…Slick (Slo2.1) and Slack (Slo2.2) channels belong to the family of high-conductance K+ channels and have been found widely distributed in the CNS. Both…”
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Molecular cloning and functional expression of the K+ channel KV7.1 and the regulatory subunit KCNE1 from equine myocardium by Pedersen, Philip J., Thomsen, Kirsten B., Flak, Jon B., Tejada, Maria A., Hauser, Frank, Trachsel, Dagmar, Buhl, Rikke, Kalbfleisch, Theodore, DePriest, Michael Scott, MacLeod, James N., Calloe, Kirstine, Klaerke, Dan A.

“…The voltage-gated K+-channel KV7.1 and the subunit KCNE1, encoded by the KCNQ1 and KCNE1 genes, respectively, are responsible for termination of the cardiac…”
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Corrigendum to “Molecular cloning and functional expression of the K+ channel KV7.1 and the regulatory subunit KCNE1 from equine myocardium” [Res. Vet. Sci. 113 (2017) 79–86] by Pedersen, Philip J., Thomsen, Kirsten B., Flak, Jon B., Tejada, Maria A., Hauser, Frank, Trachsel, Dagmar, Buhl, Rikke, Kalbfleisch, Theodore, DePriest, Michael Scott, MacLeod, James N., Calloe, Kirstine, Klaerke, Dan A.

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Molecular cloning and functional expression of the K + channel K V 7.1 and the regulatory subunit KCNE1 from equine myocardium by Pedersen, Philip J, Thomsen, Kirsten B, Flak, Jon B, Tejada, Maria A, Hauser, Frank, Trachsel, Dagmar, Buhl, Rikke, Kalbfleisch, Theodore, DePriest, Michael Scott, MacLeod, James N, Calloe, Kirstine, Klaerke, Dan A

“…The voltage-gated K -channel K 7.1 and the subunit KCNE1, encoded by the KCNQ1 and KCNE1 genes, respectively, are responsible for termination of the cardiac…”
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Heteromeric Slick/Slack K.sup.+ channels show graded sensitivity to cell volume changes by Tejada, Maria A, Hashem, Nadia, Calloe, Kirstine, Klaerke, Dan A

“…Slick and Slack high-conductance K.sup.+ channels are found in the CNS, kidneys, pancreas, among other organs, where they play an important role in cell…”
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Single-Channel Recordings of K+ Channels Expressed in Saccharomyces Cerevisiae by Tejada, Maria A., Molbaek, Karen, Cazimi, Halim, Ellekvist, Peter, Calloe, Kirstine, Nielsen, Claus H., Pedersen, Per Amstrup, Klaerke, Dan A.

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Characterization of a NaV1.5 Gain-of-Function Mutation (G213D) causing Multifocal Atrial and Ventricular Premature Ectopies and an Increased Risk of Dilated Cardiomyopathy by Calloe, Kirstine, Broendberg, Anders K., Christensen, Alex H., Pedersen, Lisbeth N., Olesen, Morten S., Tejada, Maria A., Friis, Soren, Thomsen, Morten B., Bundgaard, Henning, Jensen, Henrik K.

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Characterization of a Na V 1.5 Gain-of-Function Mutation (G213D) causing Multifocal Atrial and Ventricular Premature Ectopies and an Increased Risk of Dilated Cardiomyopathy by Calloe, Kirstine, Broendberg, Anders K., Christensen, Alex H., Pedersen, Lisbeth N., Olesen, Morten S., Tejada, Maria A., Friis, Soren, Thomsen, Morten B., Bundgaard, Henning, Jensen, Henrik K.

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The Mutation P.T613a in the Pore Helix of the Kv11.1 Potassium Channel is Associated with Long QT Syndrome by POULSEN, KRISTIAN L., HOTAIT, MOSTAFA, CALLOE, KIRSTINE, KLAERKE, DAN A., REBEIZ, ABDALLAH, NEMER, GEORGES, TEJADA, MARIA A., REFAAT, MARWAN M.

“…Background Loss‐of‐function mutations in the voltage gated potassium channel Kv11.1 have been associated with the Long QT Syndrome (LQTS) type 2. We identified…”
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Cell Volume Changes Regulate Slick K.sup.+ Channels by Tejada, Maria A, Stople, Kathleen, Hammami Bomholtz, Sofia, Meinild, Anne-Kristine, Poulsen, Asser Nyander, Klaerke, Dan A

“…Slick (Slo2.1) and Slack (Slo2.2) channels belong to the family of high-conductance K.sup.+ channels and have been found widely distributed in the CNS. Both…”
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Cell Volume Changes Regulate Slick by Tejada, Maria A, Stople, Kathleen, Hammami Bomholtz, Sofia, Meinild, Anne-Kristine, Poulsen, Asser Nyander, Klaerke, Dan A

“…Slick (Slo2.1) and Slack (Slo2.2) channels belong to the family of high-conductance K.sup.+ channels and have been found widely distributed in the CNS. Both…”
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The Mutation P.T613a in the Pore Helix of the K v 11.1 Potassium Channel is Associated with Long QT Syndrome: K V 11.1_T613A IN LQTS2 by POULSEN, KRISTIAN L., HOTAIT, MOSTAFA, CALLOE, KIRSTINE, KLAERKE, DAN A., REBEIZ, ABDALLAH, NEMER, GEORGES, TEJADA, MARIA A., REFAAT, MARWAN M.

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Corrigendum to "Molecular cloning and functional expression of the K + channel K V 7.1 and the regulatory subunit KCNE1 from equine myocardium" [Res. Vet. Sci. 113 (2017) 79-86] by Pedersen, Philip J, Thomsen, Kirsten B, Flak, Jon B, Tejada, Maria A, Hauser, Frank, Trachsel, Dagmar, Buhl, Rikke, Kalbfleisch, Theodore, DePriest, Michael Scott, MacLeod, James N, Calloe, Kirstine, Klaerke, Dan A

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The Mutation P.T613a in the Pore Helix of the Kv 11.1 Potassium Channel is Associated with Long QT Syndrome by Poulsen, Kristian L, Hotait, Mostafa, Calloe, Kirstine, Klaerke, Dan A, Rebeiz, Abdallah, Nemer, Georges, Tejada, Maria A, Refaat, Marwan M

“…Loss-of-function mutations in the voltage gated potassium channel Kv 11.1 have been associated with the Long QT Syndrome (LQTS) type 2. We identified the…”
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Cell Volume Changes Regulate Slick (Slo2.1), but Not Slack (Slo2.2) K+ Channels: e110833 by Tejada, Maria A, Stople, Kathleen, Bomholtz, Sofia Hammami, Meinild, Anne-Kristine, Poulsen, Asser Nyander, Klaerke, Dan A

“…Slick (Slo2.1) and Slack (Slo2.2) channels belong to the family of high-conductance K+ channels and have been found widely distributed in the CNS. Both…”
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Las enfermedades cardäacas y su importancia social by MARIA TEJADA, A

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