Search Results - "Tahmasbi, Kamran"
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Role of Th22 cells in Helicobacter pylori-related gastritis and peptic ulcer diseases
Published in Molecular biology reports (01-12-2019)“…Helicobacter pylori ( H. pylori ) has been shown to be one of the leading causes of peptic ulcer diseases (PUDs) and gastritis. T helper-22 (Th22) cells and…”
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Up-regulated Th17 cell function is associated with increased peptic ulcer disease in Helicobacter pylori-infection
Published in Infection, genetics and evolution (01-06-2018)“…During Helicobacter pylori (H. pylori) infection CD4+ T cells in the gastric lamina propria are hyporesponsive and polarized by Th1/Th17 cell responses…”
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Correction to: Role of Th22 cells in Helicobacter pylori‑related gastritis and peptic ulcer diseases
Published in Molecular biology reports (01-09-2022)Get full text
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Downregulated regulatory T cell function is associated with increased peptic ulcer in Helicobacter pylori-infection
Published in Microbial pathogenesis (01-09-2017)“…Helicobacter pylori (H. pylori) chronically colonizes gastric/duodenal mucosa and induces gastroduodenal disease such as gastritis and peptic ulcer and induces…”
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The regulatory role of Nrf2 in antioxidants phase2 enzymes and IL-17A expression in patients with ulcerative colitis
Published in Pathology, research and practice (01-08-2018)“…[Display omitted] The performance of Nrf2 is designed in colonic tissue of patients with UC. Environmental effects trigger innate immune system to produce ROS…”
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T-bet+ Cells Polarization in Patients Infected with Helicobacter pylori Increase the Risk of Peptic Ulcer Development
Published in Archives of medical research (01-04-2019)“…Peptic ulcer disease (PUD) is a common disease worldwide moreover known as stomach ulcer or peptic ulcer. Increased the number of T CD4+ helper cells in…”
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Intensified Th9 Response is Associated with the Immunopathogenesis of Active Ulcerative Colitis
Published in Immunological investigations (03-10-2018)“…Background: Ulcerative colitis (UC) is a chronic inflammatory disorder of the large intestine histologically characterized by indistinct sustained inflammatory…”
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