Search Results - "TURNER, Jeremy J. O"

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  1. 1

    Nonsteroidal anti-inflammatory drugs increase TNF production in rheumatoid synovial membrane cultures and whole blood by Page, Theresa H, Turner, Jeremy J O, Brown, Anthony C, Timms, Emma M, Inglis, Julia J, Brennan, Fionula M, Foxwell, Brian M J, Ray, Keith P, Feldmann, Marc

    Published in The Journal of immunology (1950) (15-09-2010)
    “…Nonsteroidal anti-inflammatory drugs (NSAIDs) inhibit cyclooxygenase activity and hence PG production. However, the ability of NSAIDs to ameliorate pain and…”
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    Journal Article
  2. 2

    Constitutive P2Y2 receptor activity regulates basal lipolysis in human adipocytes by Ali, Seema B., Turner, Jeremy J. O., Fountain, Samuel J.

    Published in Journal of cell science (15-11-2018)
    “…ABSTRACT White adipocytes are key regulators of metabolic homeostasis, which release stored energy as free fatty acids via lipolysis. Adipocytes possess both…”
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    Journal Article
  3. 3

    Assessing Glycemic Control in Maintenance Hemodialysis Patients With Type 2 Diabetes by Kazempour-Ardebili, Sara, Lecamwasam, Varunika L, Dassanyake, Thushara, Frankel, Andrew H, Tam, Frederick W.K, Dornhorst, Anne, Frost, Gary, Turner, Jeremy J.O

    Published in Diabetes care (01-07-2009)
    “…OBJECTIVE: Optimizing glycemic control in diabetic patients undergoing maintenance hemodialysis requires accurate assessment. We hypothesize that 1) 48-h…”
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  4. 4

    Diagnostic challenges due to phenocopies: lessons from Multiple Endocrine Neoplasia type1 (MEN1) by Turner, Jeremy J.O, Christie, Paul T, Pearce, Simon H.S, Turnpenny, Peter D, Thakker, Rajesh V

    Published in Human mutation (2010)
    “…Phenocopies may confound the clinical diagnoses of hereditary disorders. We report phenocopies in Multiple Endocrine Neoplasia type 1 (MEN1), an autosomal…”
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  5. 5

    Hypercalcaemia – presentation and management by Turner, Jeremy J O

    Published in Clinical medicine (London, England) (01-06-2017)
    “…Hypercalcaemia is a common disorder normally caused by primary hyperparathyroidism (PHPT) or malignancy. A proportion of cases present as an emergency, which…”
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    UK national chronic hypoparathyroidism audit by Kiam, Jian Shen, Sharma, Vivek, Glenister, Liz, Fraser, William D., Turner, Jeremy J. O.

    Published in Clinical endocrinology (Oxford) (01-11-2022)
    “…Objectives Individuals with chronic hypoparathyroidism may experience suboptimal medical care with high frequency of unplanned hospitalisation and iatrogenic…”
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    Genetic Mapping Studies of Familial Juvenile Hyperuricemic Nephropathy on Chromosome 16p11-p13 by Stacey, Joanna M., Turner, Jeremy J. O., Harding, Brian, Nesbit, M. Andrew, Kotanko, Peter, Lhotta, Karl, Puig, Juan G., Torres, Rosa J., Thakker, Rajesh V.

    “…Familial juvenile hyperuricemic nephropathy (FJHN), which is inherited as an autosomal dominant disorder, is characterized by hyperuricemia, a low fractional…”
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    Hypercalcaemia and primary hyperparathyroidism by Kiam, Jian Shen, Turner, Jeremy J O

    Published in Medicine (Abingdon. 1995, UK ed.) (01-09-2021)
    “…Hypercalcaemia is most commonly caused by primary hyperparathyroidism (PHPT) or malignancy. PHPT is common, affects women more than men, and is usually caused…”
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  12. 12

    Hypercalcaemia and primary hyperparathyroidism by Turner, Jeremy J O

    Published in Medicine (Abingdon. 1995, UK ed.) (01-09-2017)
    “…Hypercalcaemia is most commonly caused by primary hyperparathyroidism (PHPT) or malignancy. PHPT is common, affects women more than men, and is usually caused…”
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    Journal Article
  13. 13

    Constitutive P2Y 2 receptor activity regulates basal lipolysis in human adipocytes by Ali, Seema B, Turner, Jeremy J O, Fountain, Samuel J

    Published in Journal of cell science (19-11-2018)
    “…White adipocytes are key regulators of metabolic homeostasis, which release stored energy as free fatty acids via lipolysis. Adipocytes possess both basal and…”
    Get full text
    Journal Article
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