Search Results - "Sztretye, Monika"

Refine Results
  1. 1

    Astaxanthin: A Potential Mitochondrial-Targeted Antioxidant Treatment in Diseases and with Aging by Keller-Pintér, Anikó, Dux, László, Csernoch, László, Czirják, Tamás, Gönczi, Mónika, Dienes, Beatrix, Sztretye, Mónika, Szentesi, Péter

    “…Oxidative stress is characterized by an imbalance between prooxidant and antioxidant species, leading to macromolecular damage and disruption of redox…”
    Get full text
    Journal Article
  2. 2

    Unravelling the Effects of Syndecan-4 Knockdown on Skeletal Muscle Functions by Sztretye, Mónika, Singlár, Zoltán, Ganbat, Nyamkhuu, Al-Gaadi, Dána, Szabó, Kitti, Köhler, Zoltán Márton, Dux, László, Keller-Pintér, Anikó, Csernoch, László, Szentesi, Péter

    “…The remodelling of the extracellular matrix plays an important role in skeletal muscle development and regeneration. Syndecan-4 is a cell surface proteoglycan…”
    Get full text
    Journal Article
  3. 3
  4. 4
  5. 5
  6. 6

    Restricting calcium currents is required for correct fiber type specification in skeletal muscle by Sultana, Nasreen, Dienes, Beatrix, Benedetti, Ariane, Tuluc, Petronel, Szentesi, Peter, Sztretye, Monika, Rainer, Johannes, Hess, Michael W, Schwarzer, Christoph, Obermair, Gerald J, Csernoch, Laszlo, Flucher, Bernhard E

    Published in Development (Cambridge) (01-05-2016)
    “…Skeletal muscle excitation-contraction (EC) coupling is independent of calcium influx. In fact, alternative splicing of the voltage-gated calcium channel…”
    Get full text
    Journal Article
  7. 7

    SOCE Is Important for Maintaining Sarcoplasmic Calcium Content and Release in Skeletal Muscle Fibers by Sztretye, Mónika, Geyer, Nikolett, Vincze, János, Al-Gaadi, Dána, Oláh, Tamás, Szentesi, Péter, Kis, Gréta, Antal, Miklós, Balatoni, Ildikó, Csernoch, László, Dienes, Beatrix

    Published in Biophysical journal (05-12-2017)
    “…Store-operated Ca2+ entry (SOCE) is a Ca2+-entry process activated by the depletion of intracellular stores and has an important role in many cell types. In…”
    Get full text
    Journal Article
  8. 8

    The Role of Orai1 in Regulating Sarcoplasmic Calcium Release, Mitochondrial Morphology and Function in Myostatin Deficient Skeletal Muscle by Sztretye, Mónika, Singlár, Zoltán, Balogh, Norbert, Kis, Gréta, Szentesi, Péter, Angyal, Ágnes, Balatoni, Ildikó, Csernoch, László, Dienes, Beatrix

    Published in Frontiers in physiology (21-12-2020)
    “…In mice a naturally occurring 12-bp deletion in the myostatin gene is considered responsible for the compact phenotype (Mstn , Cmpt) labeled by a tremendous…”
    Get full text
    Journal Article
  9. 9

    Measurement of RyR permeability reveals a role of calsequestrin in termination of SR Ca2+ release in skeletal muscle by Sztretye, Monika, Yi, Jianxun, Figueroa, Lourdes, Zhou, Jingsong, Royer, Leandro, Allen, Paul, Brum, Gustavo, Ríos, Eduardo

    Published in The Journal of general physiology (01-08-2011)
    “…The mechanisms that terminate Ca2+ release from the sarcoplasmic reticulum are not fully understood. D4cpv-Casq1 (Sztretye et al. 2011. J. Gen. Physiol…”
    Get full text
    Journal Article
  10. 10

    Hypermuscular mice with mutation in the myostatin gene display altered calcium signalling by Bodnár, Dóra, Geyer, Nikolett, Ruzsnavszky, Olga, Oláh, Tamás, Hegyi, Bence, Sztretye, Mónika, Fodor, János, Dienes, Beatrix, Balogh, Ágnes, Papp, Zoltán, Szabó, László, Müller, Géza, Csernoch, László, Szentesi, Péter

    Published in The Journal of physiology (15-03-2014)
    “…Key points Hypermuscularity associated with naturally occurring mutations in the myostatin gene as found in Compact mice results in increased muscle mass but…”
    Get full text
    Journal Article
  11. 11

    D4cpv-calsequestrin: a sensitive ratiometric biosensor accurately targeted to the calcium store of skeletal muscle by Sztretye, Monika, Yi, Jianxun, Figueroa, Lourdes, Zhou, Jingsong, Royer, Leandro, Ríos, Eduardo

    Published in The Journal of general physiology (01-08-2011)
    “…Current fluorescent monitors of free [Ca(2+)] in the sarcoplasmic reticulum (SR) of skeletal muscle cells are of limited quantitative value. They provide…”
    Get full text
    Journal Article
  12. 12

    Paradoxical buffering of calcium by calsequestrin demonstrated for the calcium store of skeletal muscle by Royer, Leandro, Sztretye, Monika, Manno, Carlo, Pouvreau, Sandrine, Zhou, Jingsong, Knollmann, Bjorn C, Protasi, Feliciano, Allen, Paul D, Ríos, Eduardo

    Published in The Journal of general physiology (01-09-2010)
    “…Contractile activation in striated muscles requires a Ca(2+) reservoir of large capacity inside the sarcoplasmic reticulum (SR), presumably the protein…”
    Get full text
    Journal Article
  13. 13

    Charged Surface Area of Maurocalcine Determines Its Interaction with the Skeletal Ryanodine Receptor by Lukács, Balázs, Sztretye, Mónika, Almássy, János, Sárközi, Sándor, Dienes, Beatrix, Mabrouk, Kamel, Simut, Cecilia, Szabó, László, Szentesi, Péter, De Waard, Michel, Ronjat, Michel, Jóna, István, Csernoch, László

    Published in Biophysical journal (01-10-2008)
    “…The 33 amino acid scorpion toxin maurocalcine (MCa) has been shown to modify the gating of the skeletal-type ryanodine receptor (RyR1). Here we explored the…”
    Get full text
    Journal Article
  14. 14
  15. 15

    Dysfunctional endocannabinoid CB1 receptor expression and signaling contribute to skeletal muscle cell toxicity induced by simvastatin by Kalkan, Hilal, Panza, Elisabetta, Pagano, Ester, Ercolano, Giuseppe, Moriello, Claudia, Piscitelli, Fabiana, Sztretye, Mónika, Capasso, Raffaele, Di Marzo, Vincenzo, Iannotti, Fabio Arturo

    Published in Cell death & disease (23-08-2023)
    “…Statins are the most prescribed lipid-lowering agents worldwide. Their use is generally safe, although muscular toxicity occurs in about 1 in 10.000 patients…”
    Get full text
    Journal Article
  16. 16

    Altered sarcoplasmic reticulum calcium transport in the presence of the heavy metal chelator TPEN by Sztretye, Mónika, Almássy, János, Deli, Tamás, Szentesi, Péter, Jung, Carole, Dienes, Beatrix, Simut, Cecilia A, Niggli, Ernst, Jona, István, Csernoch, László

    Published in Cell calcium (Edinburgh) (01-11-2009)
    “…Abstract TPEN (N,N,N′,N′-tetrakis(2-pyridylmethyl)-ethylenediamine) is a membrane-permeable heavy-metal ion chelator with a dissociation constant for Ca2+…”
    Get full text
    Journal Article
  17. 17

    Effect of TPEN on the calcium release of cultured C2C12 mouse myotubes by Sztretye, Mónika, Deli, Tamás, Szentesi, Péter, Szigeti, Gyula, Csernoch, László

    “…N,N,N',N'-tetrakis(2-pyridylmethyl)-ethilenediamine (TPEN) is a membrane permeable heavy metal chelator that has been used to study intracellular calcium…”
    Get full text
    Journal Article
  18. 18

    Alterations in the calcium homeostasis of skeletal muscle from postmyocardial infarcted rats by Szigeti, Gyula Péter, Almássy, János, Sztretye, Mónika, Dienes, Beatrix, Szabó, László, Szentesi, Péter, Vassort, Guy, Sárközi, Sándor, Csernoch, László, Jóna, István

    Published in Pflügers Archiv (01-12-2007)
    “…In chronic heart failure, skeletal muscles develop a weakness that is not associated to an impaired circulatory function but rather to alterations in the…”
    Get full text
    Journal Article
  19. 19

    Calcium Homeostasis Is Modified in Skeletal Muscle Fibers of Small Ankyrin1 Knockout Mice by Pierantozzi, Enrico, Szentesi, Péter, Al-Gaadi, Dána, Oláh, Tamás, Dienes, Beatrix, Sztretye, Mónika, Rossi, Daniela, Sorrentino, Vincenzo, Csernoch, László

    “…Small Ankyrins (sAnk1) are muscle-specific isoforms generated by the gene that participate in the organization of the sarcoplasmic reticulum (SR) of striated…”
    Get full text
    Journal Article
  20. 20

    Assessing the Potential of Nutraceuticals as Geroprotectors on Muscle Performance and Cognition in Aging Mice by Singlár, Zoltán, Szentesi, Péter, Fodor, János, Angyal, Ágnes, Csernoch, László, Sztretye, Mónika

    Published in Antioxidants (04-09-2021)
    “…Aging and frailty are associated with a decline in muscle force generation, which is a direct consequence of reduced muscle quantity and quality. Among the…”
    Get full text
    Journal Article