Search Results - "Szodorai, Anita"

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  1. 1

    APP Anterograde Transport Requires Rab3A GTPase Activity for Assembly of the Transport Vesicle by Szodorai, Anita, Kuan, Yung-Hui, Hunzelmann, Silke, Engel, Ulrike, Sakane, Ayuko, Sasaki, Takuya, Takai, Yoshimi, Kirsch, Joachim, Muller, Ulrike, Beyreuther, Konrad, Brady, Scott, Morfini, Gerardo, Kins, Stefan

    Published in The Journal of neuroscience (18-11-2009)
    “…The amyloid precursor protein (APP) is anterogradely transported by conventional kinesin in a distinct transport vesicle, but both the biochemical composition…”
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  2. 2

    Axonal Transport, Amyloid Precursor Protein, Kinesin-1, and the Processing Apparatus: Revisited by Lazarov, Orly, Morfini, Gerardo A, Lee, Edward B, Farah, Mohamed H, Szodorai, Anita, DeBoer, Scott R, Koliatsos, Vassilis E, Kins, Stefan, Lee, Virginia M.-Y, Wong, Philip C, Price, Donald L, Brady, Scott T, Sisodia, Sangram S

    Published in The Journal of neuroscience (02-03-2005)
    “…The sequential enzymatic actions of beta-APP cleaving enzyme 1 (BACE1), presenilins (PS), and other proteins of the gamma-secretase complex liberate…”
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  3. 3

    Subcellular trafficking of the amyloid precursor protein gene family and its pathogenic role in Alzheimer's disease by Kins, Stefan, Lauther, Nadine, Szodorai, Anita, Beyreuther, Konrad

    Published in Neuro-degenerative diseases (01-01-2006)
    “…Changes in the intracellular transport of amyloid precursor protein (APP) affect the extent to which APP is exposed to alpha- or beta-secretase in a common…”
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  4. 4

    Conventional Kinesin Holoenzymes Are Composed of Heavy and Light Chain Homodimers by DeBoer, Scott R, You, YiMei, Szodorai, Anita, Kaminska, Agnieszka, Pigino, Gustavo, Nwabuisi, Evelyn, Wang, Bin, Estrada-Hernandez, Tatiana, Kins, Stefan, Brady, Scott T, Morfini, Gerardo

    Published in Biochemistry (Easton) (15-04-2008)
    “…Conventional kinesin is a major microtubule-based motor protein responsible for anterograde transport of various membrane-bounded organelles (MBO) along axons…”
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    Reciprocal modulation of Aβ42 aggregation by copper and homocysteine by Keskitalo, Salla, Farkas, Melinda, Hanenberg, Michael, Szodorai, Anita, Kulic, Luka, Semmler, Alexander, Weller, Michael, Nitsch, Roger M, Linnebank, Michael

    Published in Frontiers in aging neuroscience (2014)
    “…Hyperhomocysteinemia is a risk factor for Alzheimer's disease (AD). Both homocysteine (Hcy) and amyloid β (Aβ), which accumulates in the brain of AD patients,…”
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