Search Results - "Szegedi, V."

Pentapeptides derived from Aβ1–42 protect neurons from the modulatory effect of Aβ fibrils—an in vitro and in vivo electrophysiological study by Szegedi, V., Fülöp, L., Farkas, T., Rózsa, E., Robotka, H., Kis, Z., Penke, Z., Horváth, S., Molnár, Z., Datki, Z., Soós, K., Toldi, J., Budai, D., Zarándi, M., Penke, B.

“…Short fragments and fragment analogues of beta-amyloid 1–42 peptide (Aβ1–42) display a protective effect against Aβ-mediated neurotoxicity. After consideration…”
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Divergent effects of A beta 1-42 on ionotropic glutamate receptor-mediated responses in CA1 neurons in vivo by Szegedi, V, Juhasz, G, Budai, D, Penke, B

“…Aggregated A beta 1-42 is hypothesized to be the central cause of Alzheimer's disease. However, early changes in synaptic activity may be detected in the…”
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Tianeptine potentiates AMPA receptors by activating CaMKII and PKA via the p38, p42/44 MAPK and JNK pathways by Szegedi, Viktor, Juhász, Gábor, Zhang, Xiaoqun, Barkóczi, Balázs, Qi, Hongshi, Madeira, Alexandra, Kapus, Gábor, Svenningsson, Per, Spedding, Michael, Penke, Botond

“…► The antidepressant Tianeptine enhances AMPA but not NMDA receptor function in the CA1. ► Tianeptine induces phosphorylation of GluA1 but not NA1 in the…”
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Integrin activation modulates NMDA and AMPA receptor function of CA1 cells in a dose-related fashion in vivo by Juhász, Gábor, Vass, Gabriella, Bozsó, Zsolt, Budai, Dénes, Penke, Botond, Szegedi, Viktor

“…Abstract The large family of heterodimeric, transmembrane cell adhesion receptors, integrins mediate numerous functions in the immature and adult CNS…”
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Differences between normal and alpha-synuclein overexpressing SH-SY5Y neuroblastoma cells after Aβ(1-42) and NAC treatment by Hunya, Ákos, Földi, István, Szegedi, Viktor, Soós, Katalin, Zarándi, Márta, Szabó, Antal, Zádori, Dénes, Penke, Botond, Datki, Zsolt L

“…Abstract Alpha-synuclein (αSN) plays a major role in numerous neurodegenerative disorders, such as Alzheimer's disease and Parkinson's disease. Intracellular…”
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Enhancement of NMDA responses by [beta]-amyloid peptides in the hippocampus in vivo by Molnar, Z, Soos, K, Lengyel, I, Penke, B, Szegedi, V, Budai, D

“…The effects of Alzheimer's disease-related beta-amyloid (A[beta]) peptides on the N-methyl-D-aspartate (NMDA)-evoked cell firing rate were studied in…”
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Endomorphin-2, an endogenous tetrapeptide, protects against Ass1-42 in vitro and in vivo by Szegedi, V, Juhasz, G, Rozsa, E, Juhasz-Vedres, G, Datki, Z, Fueloep, L, Bozso, Z, Lakatos, A, Laczko, I, Farkas, T, Kis, Z, Toth, G, Soos, K, Zarandi, M

“…The underlying cause of Alzheimer's disease (AD) is thought to be the ss-amyloid aggregates formed mainly by Ass1-42 peptide. Protective pentapeptides [e.g.,…”
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Pentapeptides derived from Abeta 1-42 protect neurons from the modulatory effect of Abeta fibrils--an in vitro and in vivo electrophysiological study by Szegedi, V, Fülöp, L, Farkas, T, Rózsa, E, Robotka, H, Kis, Z, Penke, Z, Horváth, S, Molnár, Z, Datki, Z, Soós, K, Toldi, J, Budai, D, Zarándi, M, Penke, B

“…Short fragments and fragment analogues of beta-amyloid 1-42 peptide (Abeta1-42) display a protective effect against Abeta-mediated neurotoxicity. After…”
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Pentapeptides derived from A beta 1-42 protect neurons from the modulatory effect of A beta fibrils-an in vitro and in vivo electrophysiological study by Szegedi, V, Fulop, L, Farkas, T, Rozsa, E, Robotka, H, Kis, Z, Penke, Z, Horvath, S, Molnar, Z, Datki, Z, Soos, K, Toldi, J, Budai, D, Zarandi, M, Penke, B

“…Short fragments and fragment analogues of beta-amyloid 1-42 peptide (A beta 1-42) display a protective effect against A beta -mediated neurotoxicity. After…”
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