Search Results - "Sun, Meijuan J S"

POLE Mutation Spectra Are Shaped by the Mutant Allele Identity, Its Abundance, and Mismatch Repair Status by Hodel, Karl P., Sun, Meijuan J.S., Ungerleider, Nathan, Park, Vivian S., Williams, Leonard G., Bauer, David L., Immethun, Victoria E., Wang, Jieqiong, Suo, Zucai, Lu, Hua, McLachlan, James B., Pursell, Zachary F.

“…Human tumors with exonuclease domain mutations in the gene encoding DNA polymerase ε (POLE) have incredibly high mutation burdens. These errors arise in four…”
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Loss of feedback regulation between FAM3B and androgen receptor driving prostate cancer progression by Ma, Tianfang, Jin, Lianjin, Bai, Shanshan, Liu, Zhan, Wang, Shuo, Shen, Beibei, Cho, Yeyoung, Cao, Subing, Sun, Meijuan J S, Fazli, Ladan, Zhang, David, Wedderburn, Chiyaro, Zhang, Derek Y, Mugon, Gavisha, Ungerleider, Nathan, Baddoo, Melody, Zhang, Kun, Schiavone, Lovisa Holmberg, Burkhardt, Brant R, Fan, Jia, You, Zongbing, Flemington, Erik K, Dong, Xuesen, Dong, Yan

“…Although the fusion of the transmembrane serine protease 2 gene (TMPRSS2) with the erythroblast transformation-specific-related gene (ERG), or TMPRSS2-ERG,…”
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Breast cancer cells survive chemotherapy by activating targetable immune-modulatory programs characterized by PD-L1 or CD80 by Shahbandi, Ashkan, Chiu, Fang-Yen, Ungerleider, Nathan A, Kvadas, Raegan, Mheidly, Zeinab, Sun, Meijuan J S, Tian, Di, Waizman, Daniel A, Anderson, Ashlyn Y, Machado, Heather L, Pursell, Zachary F, Rao, Sonia G, Jackson, James G

“…Breast cancer cells must avoid intrinsic and extrinsic cell death to relapse following chemotherapy. Entering senescence enables survival from mitotic…”
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Mouse model and human patient data reveal critical roles for Pten and p53 in suppressing POLE mutant tumor development by Park, Vivian S, Sun, Meijuan J S, Frey, Wesley D, Williams, Leonard G, Hodel, Karl P, Strauss, Juliet D, Wellens, Sydney J, Jackson, James G, Pursell, Zachary F

“…Mutations in the exonuclease domain of are associated with tumors harboring very high mutation burdens. The mechanisms linking this significant mutation…”
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Breast cancer cells survive chemotherapy by activating targetable immune modulatory programs characterized by either PD-L1 or CD80 by Shahbandi, Ashkan, Chiu, Fang-Yen, Ungerleider, Nathan A., Kvadas, Raegan, Mheidly, Zeinab, Sun, Meijuan J S, Tian, Di, Waizman, Daniel A., Anderson, Ashlyn Y., Machado, Heather L., Pursell, Zachary F., Rao, Sonia G., Jackson, James G.

“…Breast cancer cells must avoid intrinsic and extrinsic cell death to relapse following chemotherapy. Entering senescence enables survival from mitotic…”
Get full text