Search Results - "Summersgill, James T."

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    Phenotypic differences in virulence and immune response in closely related clinical isolates of influenza A 2009 H1N1 pandemic viruses in mice by Camp, Jeremy V, Chu, Yong-Kyu, Chung, Dong-Hoon, McAllister, Ryan C, Adcock, Robert S, Gerlach, Rachael L, Wiemken, Timothy L, Peyrani, Paula, Ramirez, Julio A, Summersgill, James T, Jonsson, Colleen B

    Published in PloS one (18-02-2013)
    “…To capture the possible genotypic and phenotypic differences of the 2009 influenza A virus H1N1 pandemic (H1N1pdm) strains circulating in adult hospitalized…”
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    Journal Article
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    Protein Expression Profiles of Chlamydia pneumoniae in Models of Persistence versus Those of Heat Shock Stress Response by Mukhopadhyay, Sanghamitra, Miller, Richard D, Sullivan, Erin D, Theodoropoulos, Christina, Mathews, Sarah A, Timms, Peter, Summersgill, James T

    Published in Infection and Immunity (01-07-2006)
    “…Chlamydia pneumoniae is an obligate intracellular pathogen that causes both acute and chronic human disease. Several in vitro models of chlamydial persistence…”
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    Detailed Protocol for Purification of Chlamydia pneumoniae Elementary Bodies by MUKHOPADHYAY, Sanghamitra, CLARK, Alejandra P, SULLIVAN, Erin D, MILLER, Richard D, SUMMERSGILL, James T

    Published in Journal of Clinical Microbiology (01-07-2004)
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    Identification of Chlamydia pneumoniae Proteins in the Transition from Reticulate to Elementary Body Formation by Mukhopadhyay, Sanghamitra, Good, David, Miller, Richard D., Graham, James E., Mathews, Sarah A., Timms, Peter, Summersgill, James T.

    Published in Molecular & cellular proteomics (01-12-2006)
    “…Chlamydia pneumoniae is an important human respiratory pathogen that is responsible for an estimated 10% of community-acquired pneumonia and 5% of bronchitis…”
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    Proteomic Analysis of Differentially Expressed Chlamydia pneumoniae Genes during Persistent Infection of HEp-2 Cells by MOLESTINA, Robert E, KLEIN, Jon B, MILLER, Richard D, PICRCE, William H, RAMIREZ, Julio A, SUMMERSGILL, James T

    Published in Infection and Immunity (01-06-2002)
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    Mixed cryoglobulinemia and secondary membranoproliferative glomerulonephritis associated with ehrlichiosis by Caster, Dawn J., Summersgill, James T., Paueksakon, Paisit, Massung, Robert F., Shieh, Wun-Ju, McLeish, Kenneth R.

    Published in CEN case reports (01-11-2014)
    “…Ehrlichiosis is a tick-borne disease with diverse clinical presentations, ranging in severity from a flu-like illness with fever and myalgias to a serious…”
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    Detection of Ehrlichia spp. and Anaplasma phagocytophilum in whole blood specimens using a duplex real-time PCR assay on the ARIES instrument by Wolf, Leslie A., Marimuthu, Subathra, Summersgill, James T.

    Published in Ticks and tick-borne diseases (01-05-2020)
    “…Cases of tick-borne diseases are increasing in the United States, and new tick-borne pathogen species causing human illness are being discovered. The specific…”
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    Increases in c-Jun N-Terminal Kinase/Stress-Activated Protein Kinase and p38 Activity in Monocyte-Derived Macrophages following the Uptake of Legionella pneumophila by WELSH, Chad T, SUMMERSGILL, James T, MILLER, Richard D

    Published in Infection and Immunity (01-03-2004)
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    Characterization of Chlamydia pneumoniae Persistence in HEp-2 Cells Treated with Gamma Interferon by PANTOJA, Laura G, MILLER, Richard D, RAMIREZ, Julio A, MOLESTINA, Robert E, SUMMERSGILL, James T

    Published in Infection and Immunity (01-12-2001)
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    Role of Atypical Pathogens in the Etiology of Community-Acquired Pneumonia by Arnold, Forest W, Summersgill, James T, Ramirez, Julio A

    “…Atypical pneumonia has been described for over 100 years, but some of the pathogens attributed to it have been identified only in the past decades. The most…”
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    Tissue MicroArray (TMA) analysis of normal and persistent Chlamydophila pneumoniae infection by Borel, Nicole, Mukhopadhyay, Sanghamitra, Kaiser, Carmen, Sullivan, Erin D, Miller, Richard D, Timms, Peter, Summersgill, James T, Ramirez, Julio A, Pospischil, Andreas

    Published in BMC infectious diseases (19-10-2006)
    “…Chlamydophila pneumoniae infection has been implicated as a potential risk factor for atherosclerosis, however the mechanism leading to persistent infection…”
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    Interactions of Chlamydia pneumoniae with Human Endothelial Cells by Summersgill, James T., Molestina, Robert E., Miller, Richard D., Ramirez, Julio A.

    Published in The Journal of infectious diseases (01-06-2000)
    “…In order to fulfill the “biological plausibility” criterion of a role for infection with Chlamydia pneumoniae in the pathogenesis of human atherosclerosis,…”
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    Journal Article Conference Proceeding
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    Effect of Macrolide Antibiotics on Human Endothelial Cells Activated by Chlamydia pneumoniae Infection and Tumor Necrosis Factor-α by Uriarte, Silvia M., Molestina, Robert E., Miller, Richard D., Bernabo, Jorge, Farinati, Alicia, Eiguchi, Kumiko, Ramirez, Julio A., Summersgill, James T.

    Published in The Journal of infectious diseases (01-06-2002)
    “…This study investigated the potential anti-inflammatory activity of 3 macrolide antibiotics, clarithromycin, roxithromycin, and azithromycin, in an in vitro…”
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    Evidence for persistent Chlamydia pneumoniae infection of human coronary atheromas by Borel, Nicole, Summersgill, James T, Mukhopadhyay, Sanghamitra, Miller, Richard D, Ramirez, Julio A, Pospischil, Andreas

    Published in Atherosclerosis (01-07-2008)
    “…Abstract To date, structures representing developmental stages of Chlamydia pneumoniae , especially persistent forms of this intracellular bacteria, have not…”
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    Antigens of persistent Chlamydia pneumoniae within coronary atheroma from patients undergoing heart transplantation by Borel, Nicole, Pospischil, Andreas, Dowling, Robert D, Dumrese, Claudia, Gaydos, Charlotte A, Bunk, Sebastian, Hermann, Corinna, Ramirez, Julio A, Summersgill, James T

    Published in Journal of clinical pathology (01-02-2012)
    “…In order for Chlamydia pneumoniae to play a causative role in chronic human disease, it would need to persist within infected tissue for extended periods of…”
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    Journal Article