Search Results - "Sulkowska, Mariola"

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  1. 1

    Increased Expression of Leptin and the Leptin Receptor as a Marker of Breast Cancer Progression: Possible Role of Obesity-Related Stimuli by GAROFALO, Cecilia, KODA, Mariusz, CASCIO, Sandra, SULKOWSKA, Mariola, KANCZUGA-KODA, Luiza, GOLASZEWSKA, Jolanta, RUSSO, Antonio, SULKOWSKI, Stanislaw, SURMACZ, Eva

    Published in Clinical cancer research (01-03-2006)
    “…Purpose: Recent in vitro studies suggested that the autocrine leptin loop might contribute to breast cancer development by enhancing cell growth and survival…”
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  2. 2

    Proteolytic-antiproteolytic balance and its regulation in carcinogenesis by Skrzydlewska, Elzbieta, Sulkowska, Mariola, Koda, Mariusz, Sulkowski, Stanislaw

    Published in World journal of gastroenterology : WJG (07-03-2005)
    “…Cancer development is essentially a tissue remodeling process in which normal tissue is substituted with cancer tissue. A crucial role in this process is…”
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  3. 3

    Lipid peroxidation and antioxidant status in colorectal cancer by Skrzydlewska, Elzbieta, Sulkowski, Stanislaw, Koda, Mariusz, Zalewski, Bogdan, Kanczuga-Koda, Luiza, Sulkowska, Mariola

    Published in World journal of gastroenterology : WJG (21-01-2005)
    “…AIM: Reactive oxygen species (ROS) can induce cardnogenesis via DNA injury. Both enzymatic and non-enzymatic parameters participate in cell protection against…”
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  4. 4

    Overexpression of the obesity hormone leptin in human colorectal cancer by Koda, Mariusz, Sulkowska, Mariola, Kanczuga-Koda, Luiza, Surmacz, Eva, Sulkowski, Stanislaw

    Published in Journal of clinical pathology (01-08-2007)
    “…Background: Leptin is an adipocyte-derived neurohormone, high levels of which are found in obese individuals. Leptin controls energy expenditure, acting in the…”
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  5. 5

    Relationships between hypoxia markers and the leptin system, estrogen receptors in human primary and metastatic breast cancer: effects of preoperative chemotherapy by Koda, Mariusz, Kanczuga-Koda, Luiza, Sulkowska, Mariola, Surmacz, Eva, Sulkowski, Stanislaw

    Published in BMC cancer (22-06-2010)
    “…Tumor hypoxia is marked by enhanced expression of hypoxia-inducible factor-alpha (HIF-1alpha) and glucose transporter-1 (Glut-1). Hypoxic conditions have also…”
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  6. 6

    Gradual Loss of Functional Gap Junction within Progression of Colorectal Cancer — A Shift from Membranous CX32 and CX43 Expression to Cytoplasmic Pattern During Colorectal Carcinogenesis by Kanczuga-Koda, Luiza, Koda, Mariusz, Sulkowski, Stanislaw, Wincewicz, Andrzej, Zalewski, Bogdan, Sulkowska, Mariola

    Published in In vivo (Athens) (01-01-2010)
    “…The aim of this study was the assessment of expression and location of CX32 and CX43 in colorectal adenomas and carcinomas as well as analysis of expression of…”
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  7. 7

    Cumulative expression of HIF-1-alpha, Bax, Bcl-xL and P53 in human colorectal cancer by Wincewicz, Andrzei, Sulkowska, Mariola, Koda, Mariusz, Sulkowski, Stanislaw

    Published in Pathology (01-06-2007)
    “…Hypoxia-inducible factor (HIF-1) which contains oxygen regulated HIF-1 α subunit maintains cyto-protective defence against hypoxic injury by induction of…”
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  8. 8

    Connexin 26 correlates with Bcl-xL and Bax proteins expression in colorectal cancer by Kanczuga-Koda, Luiza, Sulkowski, Stanislaw, Koda, Mariusz, Skrzydlewska, Elzbieta, Sulkowska, Mariola

    Published in World journal of gastroenterology : WJG (14-03-2005)
    “…AIM: To evaluate of Cx26 in correlation with Bcl-xL and Bax proteins in colorectal cancer.METHODS: Immunohistochemical staining using specific antibodies was…”
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  9. 9

    Evaluation of serum cathepsin B and D in relation to clinicopathological staging of colorectal cancer by Skrzydlewska, Elzbieta, Sulkowska, Mariola, Wincewicz, Andrzej, Koda, Mariusz, Sulkowski, Stanislaw

    Published in World journal of gastroenterology : WJG (21-07-2005)
    “…AIM: Proteolytic degradation of the extracellular matrix facilitates cancer invasion and promotes metastasis. The study aims at evaluation of preoperative and…”
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  10. 10

    Alterations in connexin26 expression during colorectal carcinogenesis by Kanczuga-Koda, Luiza, Sulkowski, Stanislaw, Koda, Mariusz, Sulkowska, Mariola

    Published in Oncology (01-01-2005)
    “…Gap junctional intercellular communication (GJIC) is a mechanism for direct cell-to-cell signalling and is mediated by gap junctions, which consist of…”
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  11. 11

    Relationships of P53 and Bak with EPO and EPOR in Human Colorectal Cancer by BALTAZIAK, Marek, KODA, Mariusz, WINCEWICZ, Andrzej, SULKOWSKA, Mariola, KANCZUGA-KODA, Luiza, SULKOWSKI, Stanislaw

    Published in Anticancer research (01-10-2009)
    “…Background: EPO (erythropoietin) counteracts p53-dependent apoptosis. EPO, which acts via its receptor EPOR, protects cells and inhibits apoptosis in normal…”
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  12. 12

    STAT3, HIF-1alpha, EPO and EPOR - signaling proteins in human primary ductal breast cancers by Wincewicz, Andrzej, Sulkowska, Mariola, Koda, Mariusz, Leśniewicz, Tomasz, Kanczuga-Koda, Luiza, Sulkowski, Stanisław

    “…STAT3 upregulates expression of HIF-1 induced EPO. Receptor EPOR was reported to activate STAT3. Our study was aimed at demonstration of tissue…”
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  13. 13

    Expression of Leptin, Leptin Receptor, and Hypoxia-Inducible Factor 1α in Human Endometrial Cancer by KODA, MARIUSZ, SULKOWSKA, MARIOLA, WINCEWICZ, ANDRZEJ, KANCZUGA-KODA, LUIZA, MUSIATOWICZ, BOGUSLAW, SZYMANSKA, MAGDALENA, SULKOWSKI, STANISLAW

    Published in Annals of the New York Academy of Sciences (01-01-2007)
    “…:  Recent studies suggested that Ob (Ob) and its receptor (ObR) could be involved in the pathogenesis of various human malignancies, among others in…”
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  14. 14

    Transforming Growth Factor‐β1 and Regulators of Apoptosis: P53, Bax, and Bcl‐xL in Colorectal Cancers by Sulkowski, Stanislaw, Wincewicz, Andrzej, Sulkowska, Mariola, Koda, Mariusz

    Published in Annals of the New York Academy of Sciences (01-08-2009)
    “…Growth inhibitory function of transforming growth factor‐β1 (TGF‐β1) is abolished in colorectal cancer cells as a consequence of mutations of various…”
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  15. 15

    Transforming Growth Factor- beta 1 and Regulators of Apoptosis by Sulkowski, Stanislaw, Wincewicz, Andrzej, Sulkowska, Mariola, Koda, Mariusz

    Published in Annals of the New York Academy of Sciences (01-08-2009)
    “…Growth inhibitory function of transforming growth factor- beta 1 (TGF- beta 1) is abolished in colorectal cancer cells as a consequence of mutations of various…”
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  16. 16

    Transforming Growth Factor-β1 and Regulators of Apoptosis by Sulkowski, Stanislaw, Wincewicz, Andrzej, Sulkowska, Mariola, Koda, Mariusz

    Published in Annals of the New York Academy of Sciences (01-08-2009)
    “…Growth inhibitory function of transforming growth factor‐β1 (TGF‐β1) is abolished in colorectal cancer cells as a consequence of mutations of various…”
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  17. 17

    Transforming Growth Factor-b1 and Regulators of Apoptosis by Sulkowski, Stanislaw, Wincewicz, Andrzej, Sulkowska, Mariola, Koda, Mariusz

    Published in Annals of the New York Academy of Sciences (01-08-2009)
    “…Growth inhibitory function of transforming growth factor-b1 (TGF-b1) is abolished in colorectal cancer cells as a consequence of mutations of various…”
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  18. 18
  19. 19

    Relations of TGF-β1 with HIF-1α, GLUT-1 and longer survival of colorectal cancer patients by Sulkowska, Mariola, Wincewicz, Andrzej, Sulkowski, Stanislaw, Koda, Mariusz, Kanczuga-Koda, Luiza

    Published in Pathology (01-04-2009)
    “…During colorectal carcinogenesis, transforming growth factor β 1 (TGF-β1) undergoes a functional change from suppression of cancer cell proliferation to…”
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  20. 20

    Significant Coexpression of GLUT-1, Bcl-xL, and Bax in Colorectal Cancer by WINCEWICZ, ANDRZEJ, SULKOWSKA, MARIOLA, KODA, MARIUSZ, KANCZUGA-KODA, LUIZA, WITKOWSKA, EWA, SULKOWSKI, STANISLAW

    Published in Annals of the New York Academy of Sciences (01-01-2007)
    “…:  Hypoxic cancer cells overexpress Glucose transporter 1 (GLUT‐1) to accelerate glucose intake mainly for low effective, anaerobic respiration, so that they…”
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