Search Results - "Stivers, Katlin B."

Familial hypercholesterolemia class II low-density lipoprotein receptor response to statin treatment by Omer, Linda, Hindi, Lubna, Militello, Giuseppe, Stivers, Katlin B, Tien, Kenneth C, Boyd, Nolan L

“…Low-density lipoprotein (LDL) receptor (LDLR) mutations are the primary cause of familial hypercholesterolemia (FH). Class II LDLR mutations result in a…”
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Regulating the proinflammatory response to composite biomaterials by targeting immunometabolism by Maduka, Chima V., Makela, Ashley V., Tundo, Anthony, Ural, Evran, Stivers, Katlin B., Kuhnert, Maxwell M., Alhaj, Mohammed, Hoque Apu, Ehsanul, Ashammakhi, Nureddin, Hankenson, Kurt D., Narayan, Ramani, Elisseeff, Jennifer H., Contag, Christopher H.

“…Composite biomaterials comprising polylactide (PLA) and hydroxyapatite (HA) are applied in bone, cartilage and dental regenerative medicine, where HA confers…”
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Adipose‐resident myeloid‐derived suppressor cells modulate immune cell homeostasis in healthy mice by Stivers, Katlin B, Chilton, Paula M, Beare, Jason E, Dale, Jacob R, Alard, Pascale, LeBlanc, Amanda J, Hoying, James B

“…The metabolically dynamic nature of healthy adipose places this tissue under regular inflammatory stress. A network of adipose‐resident anti‐inflammatory…”
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Transfer learning in a biomaterial fibrosis model identifies in vivo senescence heterogeneity and contributions to vascularization and matrix production across species and diverse pathologies by Cherry, Christopher, Andorko, James I., Krishnan, Kavita, Mejías, Joscelyn C., Nguyen, Helen Hieu, Stivers, Katlin B., Gray-Gaillard, Elise F., Ruta, Anna, Han, Jin, Hamada, Naomi, Hamada, Masakazu, Sturmlechner, Ines, Trewartha, Shawn, Michel, John H., Davenport Huyer, Locke, Wolf, Matthew T., Tam, Ada J., Peña, Alexis N., Keerthivasan, Shilpa, Le Saux, Claude Jordan, Fertig, Elana J., Baker, Darren J., Housseau, Franck, van Deursen, Jan M., Pardoll, Drew M., Elisseeff, Jennifer H.

“…Cellular senescence is a state of permanent growth arrest that plays an important role in wound healing, tissue fibrosis, and tumor suppression. Despite…”
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Adipose-derived cellular therapies in solid organ and vascularized-composite allotransplantation by Stivers, Katlin B., Beare, Jason E., Chilton, Paula M., Williams, Stuart K., Kaufman, Christina L., Hoying, James B.

“…Controlling acute allograft rejection following vascularized composite allotransplantation requires strict adherence to courses of systemic immunosuppression…”
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Immunometabolic cues recompose and reprogram the microenvironment around implanted biomaterials by Maduka, Chima V., Schmitter-Sánchez, Axel D., Makela, Ashley V., Ural, Evran, Stivers, Katlin B., Pope, Hunter, Kuhnert, Maxwell M., Habeeb, Oluwatosin M., Tundo, Anthony, Alhaj, Mohammed, Kiselev, Artem, Chen, Shoue, Donneys, Alexis, Winton, Wade P., Stauff, Jenelle, Scott, Peter J. H., Olive, Andrew J., Hankenson, Kurt D., Narayan, Ramani, Park, Sangbum, Elisseeff, Jennifer H., Contag, Christopher H.

“…Circulating monocytes infiltrate and coordinate immune responses in tissues surrounding implanted biomaterials and in other inflamed tissues. Here we show that…”
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Role of Mitochondrial Complex I in the Proinflammatory Response to Polylactide Implants by Maduka, Chima V., Makela, Ashley V., Tundo, Anthony, Ural, Evran, Stivers, Katlin B., Alhaj, Mohammed, Narayan, Ramani, Goodman, Stuart B., Ashammakhi, Nureddin, Elisseeff, Jennifer H., Hankenson, Kurt D., Contag, Christopher H.

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Familial hypercholesterolemia class II low density lipoprotein-receptor response to statin treatment by Omer, Linda, Hindi, Lubna, Militello, Giuseppe, Stivers, Katlin B, Tien, Kenneth C, Boyd, Nolan L

“…LDL receptor (LDLR) mutations are the primary cause of familial hypercholesterolemia (FH). Class II LDLR mutations result in a misfolded LDLR retained in the…”
Get full text