Search Results - "Stillwagon, Samantha J"
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Gene Expression Analyses during Spontaneous Reversal of Cardiomyopathy in Mice with Repressed Nuclear CUG-BP, Elav-Like Family (CELF) Activity in Heart Muscle
Published in PloS one (20-04-2015)“…CUG-BP, Elav-like family (CELF) proteins regulate cell type- and developmental stage-specific alternative splicing in the heart. Repression of CELF-mediated…”
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Gene expression analyses implicate an alternative splicing program in regulating contractile gene expression and serum response factor activity in mice
Published in PloS one (20-02-2013)“…Members of the CUG-BP, Elav-like family (CELF) regulate alternative splicing in the heart. In MHC-CELFΔ transgenic mice, CELF splicing activity is inhibited…”
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Muscleblind-like 1 is required for normal heart valve development in vivo
Published in BMC developmental biology (15-10-2015)“…Development of the valves and septa of the heart depends on the formation and remodeling of the endocardial cushions in the atrioventricular canal and outflow…”
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4
Loss of muscleblind-like 1 promotes invasive mesenchyme formation in endocardial cushions by stimulating autocrine TGFβ3
Published in BMC developmental biology (06-08-2012)“…Valvulogenesis and septation in the developing heart depend on the formation and remodeling of endocardial cushions in the atrioventricular canal (AVC) and…”
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5
Loss of muscleblind-like 1 promotes invasive mesenchyme formation in endocardial cushions by stimulating autocrine TGF[beta]3
Published in BMC developmental biology (06-08-2012)“…Valvulogenesis and septation in the developing heart depend on the formation and remodeling of endocardial cushions in the atrioventricular canal (AVC) and…”
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Journal Article -
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Gene Expression Analyses during Spontaneous Reversal of Cardiomyopathy in Mice with Repressed Nuclear CUG-BP, Elav-Like Family (CELF) Activity in Heart Muscle: e0124462
Published in PloS one (01-04-2015)“…CUG-BP, Elav-like family (CELF) proteins regulate cell type- and developmental stage-specific alternative splicing in the heart. Repression of CELF-mediated…”
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Journal Article