Search Results - "Solem, Laura"
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A Comparative Study of Phase I and II Hepatic Microsomal Biotransformation of Phenol in Three Species of Salmonidae: Hydroquinone, Catechol, and Phenylglucuronide Formation
Published in Fishes (01-07-2024)“…The biotransformation of phenol at 11 °C was studied using pre-spawn adult rainbow ( ) (RBT), brook ( ) (BKT), and lake trout ( ) (LKT) hepatic microsomal…”
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2
Sex-Linked Changes in Biotransformation of Phenol in Brook Trout (Salvelinus fontinalis) over an Annual Reproductive Cycle
Published in Fishes (01-08-2024)“…The microsomal metabolism of phenol (11 °C) over an annual reproductive cycle from June to December was studied using fall spawning adult brook trout…”
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3
An in vivo microdialysis method for the qualitative analysis of hepatic phase I metabolites of phenol in rainbow trout ( Oncorhynchus mykiss)
Published in Aquatic toxicology (26-02-2003)“…Development of reliable and accurate methodologies for determination of xenobiotic hepatic biotransformation rate and capacity parameters is important to the…”
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4
PFAAs in wastewater treatment plants and sludge
Published in Reproductive toxicology (Elmsford, N.Y.) (01-07-2012)Get full text
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5
Exposure and effects of perfluoroalkyl compounds on tree swallows nesting at Lake Johanna in east central Minnesota, USA
Published in Reproductive toxicology (Elmsford, N.Y.) (01-07-2012)“…Tree swallow (Tachycineta bicolor) samples were collected at a reference lake and a nearby lake (Lake Johanna) in east central Minnesota, USA contaminated with…”
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Exposure and effects of perfluoroalkyl compounds on tree swallows nesting at Lake Johanna in east central Minnesota, USA: PFAA Days III A Second Special Issue on: Recent Advances in Perfluoroalkyl Acid Research
Published in Reproductive toxicology (Elmsford, N.Y.) (2012)Get full text
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Identification of a major source of perfluorooctane sulfonate (PFOS) at a wastewater treatment plant in Brainerd, Minnesota
Published in Reproductive toxicology (Elmsford, N.Y.) (01-06-2009)Get full text
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8
Disruption of mitochondrial calcium homeostasis following chronic doxorubicin administration
Published in Toxicology and applied pharmacology (01-12-1994)“…Doxorubicin (Adriamycin) is an anthracycline antibiotic with broad antineoplastic activity. However, the clinical success is limited by the incidence of…”
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9
Enhanced Induction of the Mitochondrial Permeability Transition Following Acute Menadione Administration
Published in Archives of biochemistry and biophysics (20-02-1995)“…Induction of the mitochondrial permeability transition in vitro is well-characterized and widely implicated in the mechanism of oxidant-induced cell death…”
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10
Mitochondrial-mediated cardiotoxicity of doxorubicin
Published 01-01-1995“…The clinical utility of the antineoplastic drug doxorubicin is limited by the cumulative and irreversible onset of cardiomyopathy associated with chronic drug…”
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Dissertation -
11
Enhancement of antigen-induced bronchoconstriction after intravascular complement activation with cobra venom factor. Reversal by granulocyte depletion
Published in The Journal of immunology (1950) (15-04-1993)“…Our previous studies have demonstrated that activation of the C system with cobra venom factor (CVF) in a passively sensitized guinea pig results in an…”
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12
Channel-specific induction of the cyclosporine A-sensitive mitochondrial permeability transition by menadione
Published in Journal of toxicology and environmental health (01-08-1995)“…It is well established that menadione, 2-methyl-1,4-naphthoquinone, impairs the ability of rat liver mitochondria to accumulate and retain calcium. However, it…”
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13
Activation of macrophages from aging mice by detoxified lipid A
Published in Journal of leukocyte biology (01-04-1991)“…A detoxified derivative of endotoxic lipopolysaccharides (LPS), monophosphoryl lipid A (MPL), which is capable of inducing nonspecific resistance against…”
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14
The adjuvant properties of a nontoxic monophosphoryl lipid A in hyporesponsive and aging mice
Published in Journal of biological response modifiers (01-04-1987)“…The immunomodulatory action of a nontoxic monophosphoryl lipid A (MPL) and a toxic diphosphoryl lipid A (DPL) fraction derived from endotoxins of the…”
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