Search Results - "Smedt, H D"

Proceedings: Chemical control of the lipid phase transition in black lecithin films by Smedt, H D, Borghgraef, R

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Staphylokinase-Specific Cell-Mediated Immunity in Humans by Warmerdam, Petra A. M, Vanderlick, Kristel, Vandervoort, Petra, De Smedt, Heidi, Plaisance, Stephane, De Maeyer, Marc, Collen, Desire

“…Staphylokinase is a highly fibrin-specific clot-dissolving agent that constitutes a promising drug for clinical development. It is of bacterial origin, and the…”
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Agonist-induced down-regulation of type 1 and type 3 inositol 1,4,5-trisphosphate receptors in A7r5 and DDT1 MF-2 smooth muscle cells by Sipma, H, Deelman, L, Smedt, H D, Missiaen, L, Parys, J B, Vanlingen, S, Henning, R H, Casteels, R

“…Prolonged stimulation of rat A7r5 aortic smooth muscle cells with 3 microM vasopressin, or of hamster DDT1 MF-2 smooth muscle cells with 10 microM bradykinin…”
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Effect of adenine nucleotides on myo-inositol-1,4,5-trisphosphate-induced calcium release by Missiaen, L, Parys, J B, Smedt, H D, Sienaert, I, Sipma, H, Vanlingen, S, Maes, K, Casteels, R

“…The effects of a whole series of adenine nucleotides on Ins(1,4,5)P3-induced Ca2+ release were characterized in permeabilized A7r5 smooth-muscle cells. Several…”
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Mechanisms responsible for quantal Ca2+ release from inositol trisphosphate-sensitive calcium stores by Parys, J B, Missiaen, L, Smedt, H D, Sienaert, I, Casteels, R

“…Activation of cells by hormones, growth factors or neurotransmitters leads to an increased production of inositol trisphosphate (InsP3) and, after activation…”
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Modulation of Inositol 1,4,5-Trisphosphate Binding to the Recombinant Ligand-binding Site of the Type-1 Inositol 1,4,5-Trisphosphate Receptor by Ca super(2+) and Calmodulin by Sipma, H, De Smet, P, Sienaert, I, Vanlingen, S, Missiaen, L, Parys, J B, Smedt, H D

“…A recombinant protein (Lbs-1) containing the N-terminal 581 amino acids of the mouse type 1 inositol 1,4,5-trisphosphate receptor (IP sub(3)R-1), including the…”
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Perceived medication adverse effects and coping strategies reported by chronic heart failure patients by De Smedt, R H E, Denig, P, Haaijer-Ruskamp, F M, Jaarsma, T

“…Data on medication adverse effects (AEs) in chronic heart failure (CHF) are primarily based on results from clinical trials. Little is known about AEs…”
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Ca2+ extrusion across plasma membrane and Ca2+ uptake by intracellular stores by Missiaen, L, Wuytack, F, Raeymaekers, L, De Smedt, H, Droogmans, G, Declerck, I, Casteels, R

“…The aim of this review is to summarize the various systems that remove Ca2+ from the cytoplasm. We will initially focus on the Ca2+ pump and the Na(+)-Ca2+…”
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Regulation of splicing is responsible for the expression of the muscle-specific 2a isoform of the sarco/endoplasmic-reticulum Ca2+-ATPase by VAN DEN BOSCH, L, EGGERMONT, J, DE SMEDT, H, MERTENS, L, WUYTACK, F, CASTEELS, R

“…Tissue-specific alternative processing of sarco/endoplasmic reticulum Ca(2+)-ATPase 2 (SERCA2) transcripts generates functionally different Ca2+ pump isoforms…”
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2,5-Di-(tert-butyl)-1,4-benzohydroquinone and cyclopiazonic acid decrease the Ca2+ permeability of endoplasmic reticulum by Missiaen, L, De Smedt, H, Droogmans, G, Casteels, R

“…Specific inhibitors of the endoplasmic-reticulum Ca2+ pump will deplete intracellular stores and are therefore useful to study the role of store depletion on…”
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Poorly selective cation channels in the apical membrane of A6 cells by VAN DRIESSCHE, W, DE SMET, P, DE SMEDT, H

“…This paper describes a Ca(2+)-blockable, poorly selective cation pathway in the apical membrane of A6 epithelia. This pathway has properties that resemble the…”
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Calmodulin Stimulation of Renal (Ca super(2+) + Mg super(2+))-ATPase by Smedt, H D, Parys, J B, Borghgraef, R, Wuytack, F

“…There is evidence for at least two different transport systems for Ca super(2+) in basal--lateral plasma membranes of proximal tubular epithelial cells: a Na…”
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Kinetics of empty store-activated Ca2+ influx in HeLa cell by MISSIAEN, L, DE SMEDT, H, PARY, J. B, OIKE, M, CASTEELS, R

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Luminal Ca2+ controls the activation of the inositol 1,4,5-triphosphate receptor by cytosolic Ca2 by MISSIAEN, L, DE SMEDT, H, DROOGMANS, G, CASTEELS, R

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Stimulation of the catalytic cycle of the Ca2+ pump of porcine plasma-membranes by negatively charged phospholipids by Lehotsky, J, Raeymaekers, L, Missiaen, L, Wuytack, F, De Smedt, H, Casteels, R

“…The (Ca(2+)+Mg2+)-ATPase of the plasma membrane is activated by negatively charged phospholipids. The mechanism of this activation was investigated by studying…”
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Analysis of membrane-bound acceptors. A correction function for non-specific accumulation of poorly water-soluble hydrophobic or amphipathic ligands based on the ligand partition concept by Heirwegh, K P, De Smedt, H, Vermeir, M

“…Non-specific ligand accumulation into membrane material, which may contribute considerably to the experimental signal obtained in binding studies with labelled…”
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Vasopressin responses in electrically coupled A7r5 cells by MISSIAEN, L, OIKE, M, BOOTMAN, M. D, DE SMEDT, H, PARYS, J. B, CASTEELS, R

“…Changes in cytosolic Ca2+ concentration ([Ca2+]i) and in membrane potential were monitored in single A7r5 smooth-muscle cells during spontaneous spiking and…”
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Co-activation of inositol trisphosphate-induced Ca2+ release by cytosolic Ca2+ is loading-dependent by MISSIAEN, L, DE SMEDT, H, PARYS, J. B, CASTEELS, R

“…Luminal and cytosolic Ca2+ control the sensitivity of the intracellular Ca2+ stores to inositol 1,4,5-trisphosphate (InsP3). In this work, we have…”
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Different pattern of differentiation in two LLC-PK1 clones by Van den Bosch, L, De Smedt, H, Borghgraef, R

“…Two clones (LD3 and LC3) were isolated from the established renal cell line LLC-PK1. They differed with respect to the development of the Na+-dependent…”
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Normal Ca2+ signalling in glutathione-depleted and dithiothreitol-treated HeLa cells by MISSIAEN, L, PARYS, J. B, BOOTMAN, M. D, DE SMEDT, H, DROOGMANS, G, CASTEELS, R

“…We have investigated whether reducing agents and substances that interfere with glutathione metabolism would affect the histamine-induced rises in internal…”
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