Search Results - "Shlomovitz, Inbar"

Flipping the dogma - phosphatidylserine in non-apoptotic cell death by Shlomovitz, Inbar, Speir, Mary, Gerlic, Motti

“…The exposure of phosphatidylserine (PS) on the outer plasma membrane has long been considered a unique feature of apoptotic cells. Together with other "eat me"…”
Get full text

Macrophages, rather than DCs, are responsible for inflammasome activity in the GM-CSF BMDC model by Erlich, Ziv, Shlomovitz, Inbar, Edry-Botzer, Liat, Cohen, Hadar, Frank, Daniel, Wang, Hanqing, Lew, Andrew M., Lawlor, Kate E., Zhan, Yifan, Vince, James E., Gerlic, Motti

“…Inflammasomes are one of the most important mechanisms for innate immune defense against microbial infection but are also known to drive various inflammatory…”
Get full text

Necroptosis directly induces the release of full‐length biologically active IL‐33 in vitro and in an inflammatory disease model by Shlomovitz, Inbar, Erlich, Ziv, Speir, Mary, Zargarian, Sefi, Baram, Noam, Engler, Maya, Edry‐Botzer, Liat, Munitz, Ariel, Croker, Ben A., Gerlic, Motti

“…Interleukin‐33 (IL‐33) is a pro‐inflammatory cytokine that plays a significant role in inflammatory diseases by activating immune cells to induce type 2 immune…”
Get full text

Phosphatidylserine externalization, "necroptotic bodies" release, and phagocytosis during necroptosis by Zargarian, Sefi, Shlomovitz, Inbar, Erlich, Ziv, Hourizadeh, Aria, Ofir-Birin, Yifat, Croker, Ben A, Regev-Rudzki, Neta, Edry-Botzer, Liat, Gerlic, Motti

“…Necroptosis is a regulated, nonapoptotic form of cell death initiated by receptor-interacting protein kinase-3 (RIPK3) and mixed lineage kinase domain-like…”
Get full text

Proteomic analysis of necroptotic extracellular vesicles by Shlomovitz, Inbar, Erlich, Ziv, Arad, Gali, Edry-Botzer, Liat, Zargarian, Sefi, Cohen, Hadar, Manko, Tal, Ofir-Birin, Yifat, Cooks, Tomer, Regev-Rudzki, Neta, Gerlic, Motti

“…Necroptosis is a regulated and inflammatory form of cell death. We, and others, have previously reported that necroptotic cells release extracellular vesicles…”
Get full text

Mechanisms of RIPK3‐induced inflammation by Shlomovitz, Inbar, Zargrian, Sefi, Gerlic, Motti

“…Receptor‐interacting protein kinase 3 (RIP3/RIPK3) is a multifunctional regulator of cell death and inflammation. It controls signalling downstream of the…”
Get full text

Ptpn6 inhibits caspase-8- and Ripk3/Mlkl-dependent inflammation by Speir, Mary, Nowell, Cameron J., Chen, Alyce A., O’Donnell, Joanne A., Shamie, Isaac S., Lakin, Paul R., D’Cruz, Akshay A., Braun, Roman O., Babon, Jeff J., Lewis, Rowena S., Bliss-Moreau, Meghan, Shlomovitz, Inbar, Wang, Shu, Cengia, Louise H., Stoica, Anca I., Hakem, Razq, Kelliher, Michelle A., O’Reilly, Lorraine A., Patsiouras, Heather, Lawlor, Kate E., Weller, Edie, Lewis, Nathan E., Roberts, Andrew W., Gerlic, Motti, Croker, Ben A.

“…Ptpn6 is a cytoplasmic phosphatase that functions to prevent autoimmune and interleukin-1 (IL-1) receptor-dependent, caspase-1-independent inflammatory…”
Get full text

A genetic system for biasing the sex ratio in mice by Yosef, Ido, Edry‐Botzer, Liat, Globus, Rea, Shlomovitz, Inbar, Munitz, Ariel, Gerlic, Motti, Qimron, Udi

“…Biasing the sex ratio of populations of different organisms, including plants, insects, crustacean, and fish, has been demonstrated by genetic and non‐genetic…”
Get full text

Necroptotic extracellular vesicles – present and future by Raden, Yael, Shlomovitz, Inbar, Gerlic, Motti

“…Necroptosis is a receptor-interacting protein kinase 3 (RIPK3)-/mixed lineage kinase domain-like(MLKL)-dependent and caspase-independent form of cell death…”
Get full text

Distinguishing Necroptosis from Apoptosis by Shlomovitz, Inbar, Zargarian, Sefi, Erlich, Ziv, Edry-Botzer, Liat, Gerlic, Motti

“…Apoptosis was the first programmed cell death to be defined-highly regulated and immunologically silent, as apoptotic bodies are being removed without…”
Get more information