Search Results - "Shih, Y. L"

RUNX1 mutations are frequent in chronic myelomonocytic leukemia and mutations at the C-terminal region might predict acute myeloid leukemia transformation by Kuo, M-C, Liang, D-C, Huang, C-F, Shih, Y-S, Wu, J-H, Lin, T-L, Shih, L-Y

“…Runt-related transcription factor 1 (RUNX1) is essential for normal hematopoiesis. RUNX1 mutations have rarely been reported in chronic myelomonocytic leukemia…”
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Comprehensive mutational analysis of primary and relapse acute promyelocytic leukemia by Madan, V, Shyamsunder, P, Han, L, Mayakonda, A, Nagata, Y, Sundaresan, J, Kanojia, D, Yoshida, K, Ganesan, S, Hattori, N, Fulton, N, Tan, K-T, Alpermann, T, Kuo, M-C, Rostami, S, Matthews, J, Sanada, M, Liu, L-Z, Shiraishi, Y, Miyano, S, Chendamarai, E, Hou, H-A, Malnassy, G, Ma, T, Garg, M, Ding, L-W, Sun, Q-Y, Chien, W, Ikezoe, T, Lill, M, Biondi, A, Larson, R A, Powell, B L, Lübbert, M, Chng, W J, Tien, H-F, Heuser, M, Ganser, A, Koren-Michowitz, M, Kornblau, S M, Kantarjian, H M, Nowak, D, Hofmann, W-K, Yang, H, Stock, W, Ghavamzadeh, A, Alimoghaddam, K, Haferlach, T, Ogawa, S, Shih, L-Y, Mathews, V, Koeffler, H P

“…Acute promyelocytic leukemia (APL) is a subtype of myeloid leukemia characterized by differentiation block at the promyelocyte stage. Besides the presence of…”
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Cadmium-induced autophagy and apoptosis are mediated by a calcium signaling pathway by Wang, S. H, Shih, Y. L, Ko, W. C, Wei, Y. H, Shih, C. M

“…The cytotoxicity of cadmium (Cd) induced autophagy and apoptosis in MES-13 cells was determined by flow cytometry. Autophagy was also assessed by formation of…”
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Somatic PHF6 mutations in 1760 cases with various myeloid neoplasms by Mori, T, Nagata, Y, Makishima, H, Sanada, M, Shiozawa, Y, Kon, A, Yoshizato, T, Sato-Otsubo, A, Kataoka, K, Shiraishi, Y, Chiba, K, Tanaka, H, Ishiyama, K, Miyawaki, S, Mori, H, Nakamaki, T, Kihara, R, Kiyoi, H, Koeffler, H P, Shih, L-Y, Miyano, S, Naoe, T, Haferlach, C, Kern, W, Haferlach, T, Ogawa, S, Yoshida, K

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NUDT15 gene polymorphism related to mercaptopurine intolerance in Taiwan Chinese children with acute lymphoblastic leukemia by Liang, D-C, Yang, C-P, Liu, H-C, Jaing, T-H, Chen, S-H, Hung, I-J, Yeh, T-C, Lin, T-H, Lai, C-L, Lai, C-Y, Shih, L-Y

“…A recent study identified a variant of the NUDT15 gene (rs116855232 C>T) associated with intolerance to thiopurine in Korean patients with Crohn’s disease…”
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EED mutants impair polycomb repressive complex 2 in myelodysplastic syndrome and related neoplasms by Ueda, T, Sanada, M, Matsui, H, Yamasaki, N, Honda, Z-i, Shih, L-Y, Mori, H, Inaba, T, Ogawa, S, Honda, H

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Antiplatelet autoantibodies elicited by dengue virus non‐structural protein 1 cause thrombocytopenia and mortality in mice by SUN, D.‐S., KING, C.‐C., HUANG, H.‐S., SHIH, Y.‐L., LEE, C.‐C., TSAI, W.‐J., YU, C.‐C., CHANG, H.‐H.

“…Background: The mechanisms responsible for thrombocytopenia associated with dengue fever (DF) and dengue hemorrhage fever (DHF) remain unclear. Objective: In…”
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Diagnosis and relapse: cytogenetically normal acute myelogenous leukemia without FLT3-ITD or MLL-PTD by Chien, W, Sun, Q-Y, Ding, L-W, Mayakonda, A, Takao, S, Liu, L, Lim, S L, Tan, K T, Garg, M, De Sousa Maria Varela, A, Xiao, J, Jacob, N, Behrens, K, Stocking, C, Lill, M, Madan, V, Hattori, N, Gery, S, Ogawa, S, Wakita, S, Ikezoe, T, Shih, L-Y, Alpermann, T, Haferlach, T, Yang, H, Koeffler, H P

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Characterization of fusion partner genes in 114 patients with de novo acute myeloid leukemia and MLL rearrangement by SHIH, L.-Y, LIANG, D.-C, LAI, C.-L, FU, J.-F, WU, J.-H, WANG, P.-N, LIN, T.-L, DUNN, P, KUO, M.-C, TANG, T.-C, LIN, T.-H

“…The fusion transcripts of MLL rearrangement [MLL(+)] in acute myeloid leukemia (AML) and their clinicohematologic correlation have not be well characterized in…”
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Acquisition of FLT3 or N-ras mutations is frequently associated with progression of myelodysplastic syndrome to acute myeloid leukemia by SHIN, L-Y, HUANG, C-F, WANG, P-N, WU, J-H, LIN, T-L, DUNN, P, KUO, M-C

“…The role of internal tandem duplication of fms-like tyrosine kinase 3 (FLT3/ITD), mutations at tyrosine kinase domain (FLT3/TKD) and N-ras mutations in the…”
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Dlk1 in normal and abnormal hematopoiesis by SAKAJIRI, S, O'KELLY, J, TEISNER, B, KAWAMATA, N, KOEFFLER, H. P, YIN, D, MILLER, C. W, HOFMANN, W. K, OSHIMI, K, SHIH, L.-Y, KIM, K.-H, SUL, H. S, JENSEN, C. H

“…Dlk1 (Pref-1) is a transmembrane and secreted protein, which is a member of the epidermal growth factor-like family, homologous to Notch/Delta/Serrate. We have…”
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Effectiveness of national cervical cancer screening programme in Taiwan: 12-year experiences by Chen, Y-Y, You, S-L, Chen, C-A, Shih, L-Y, Koong, S-L, Chao, K-Y, Hsiao, M-L, Hsieh, C-Y, Chen, C-J

“…Background: We examined cervical cancer incidence before and after nationwide cervical cancer screening was initiated in Taiwan in mid-1995. Results: The…”
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Cooperating mutations of receptor tyrosine kinases and Ras genes in childhood core-binding factor acute myeloid leukemia and a comparative analysis on paired diagnosis and relapse samples by Shih, L-Y, Liang, D-C, Huang, C-F, Chang, Y-T, Lai, C-L, Lin, T-H, Yang, C-P, Hung, I-J, Liu, H-C, Jaing, T-H, Wang, L-Y, Yeh, T-C

“…c-KIT mutations have been described in core-binding factor (CBF) acute myeloid leukemia (AML) at diagnosis. The role of c-KIT mutations in the relapse of…”
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A nonsense mutation of IDH1 in myelodysplastic syndromes and related disorders by Yoshida, K, Sanada, M, Kato, M, Kawahata, R, Matsubara, A, Takita, J, Shih, L-Y, Mori, H, Koeffler, H P, Ogawa, S

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P744: UNBALANCED TRANSLOCATION DER(1;7)(Q10;P10) AS A DISTINCT SUBTYPE IN MYELODYSPLASTIC SYNDROMES by Okuda, R., Ochi, Y., Chonabayashi, K., Hiramoto, N., Sanada, M., Handa, H., Kasahara, S., Sato, S., Kanemura, N., Kitano, T., Watanabe, M., Kern, W., Creignou, M., Shiraishi, Y., Watanabe, M., Usuki, K., Imashuku, S., Hellstrom‐Lindberg, E., Haferlach, T., Chiba, S., Sezaki, N., Shih, L.‐Y., Miyazaki, Y., Yoshida, Y., Ishikawa, T., Ohyashiki, K., Atsuta, Y., Shiozawa, Y., Miyano, S., Makishima, H., Nannya, Y., Ogawa, S.

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P466: AMPLIFIED EPOR/JAK2 GENES DEFINE A UNIQUE SUBTYPE OF ACUTE ERYTHROID LEUKEMIA by Takeda, J., Yoshida, K., Nakagawa, M., Nannya, Y., Yoda, A., Saiki, R., Ochi, Y., Zhao, L., Okuda, R., Qi, X., Mori, T., Kon, A., Chiba, K., Tanaka, H., Shiraishi, Y., Kuo, M.‐C., Kerr, C., Nagata, Y., Morishita, D., Hiramoto, N., Hangaishi, A., Nakazawa, H., Ishiyama, K., Miyano, S., Chiba, S., Miyazaki, Y., Kitano, T., Usuki, K., Sezaki, N., Tsurumi, H., Miyawaki, S., Maciejewski, J., Ishikawa, T., Ohyashiki, K., Ganser, A., Heuser, M., Thol, F., Shih, L.‐Y., Takaori−Kondo, A., Makishima, H., Ogawa, S.

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Rubella seroepidemiology and estimations of the catch‐up immunisation rate and persistence of antibody titers in pregnant women in Taiwan by Lin, C‐C, Yang, C‐Y, Shih, Y‐L, Hsu, H‐W, Yang, T‐H, Cheng, Y‐W, Chang, C‐F, Hsieh, L‐C, Chen, B‐H, Lee, C‐H, Huang, Y‐L

“…Please cite this paper as: Lin C, Yang C, Shih Y, Hsu H, Yang T, Cheng Y, Chang C, Hsieh L, Chen B, Lee C, Huang Y. Rubella seroepidemiology and estimations of…”
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CEBPα mutations in childhood acute myeloid leukemia by LIANG, D.-C, SHIH, L.-Y, HUANG, C.-F, HUNG, I.-J, YANG, C.-P, LIU, H.-C, JAING, T.-H, WANG, L.-Y, CHANG, W.-H

“…CEBPα mutations have been described in adult acute myeloid leukemia (AML) and conferred a favorable prognosis. However, CEBPα mutation has not been reported in…”
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FLT3-TKD mutation in childhood acute myeloid leukemia by LIANG, D.-C, SHIH, L.-Y, HUNG, I.-J, YANG, C.-P, CHEN, S.-H, JAING, T.-H, LIU, H.-C, WANG, L.-Y, CHANG, W.-H

“…Mutations of receptor tyrosine kinases are implicated in the constitutive activation and development of human hematologic malignancies. An internal tandem…”
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Aml patients with CEBPα mutations mostly retain identical mutant patterns but frequently change in allelic distribution at relapse : a comparative analysis on paired diagnosis and relapse samples by SHIH, L.-Y, LIANG, D.-C, HUANG, C.-F, WU, J.-H, LIN, T.-L, WANG, P.-N, DUNN, P, KUO, M.-C, TANG, T.-C

“…The roles of CEBPα mutations and its cooperating mutations in the relapse of acute myeloid leukemia (AML) are not clear. CEBPα mutations were analyzed on 149…”
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