Search Results - "Shao, Dingwu"
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Factor H Autoantibodies and Complement-Mediated Diseases
Published in Frontiers in immunology (15-12-2020)“…Factor H (FH), a member of the regulators-of-complement-activation (RCA) family of proteins, circulates in human plasma at concentrations of 180-420 mg/L where…”
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Complement Factor I Variants in Complement-Mediated Renal Diseases
Published in Frontiers in immunology (10-05-2022)“…C3 glomerulopathy (C3G) and atypical hemolytic uremic syndrome (aHUS) are two rare diseases caused by dysregulated activity of the alternative pathway of…”
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Factor B and C4b2a Autoantibodies in C3 Glomerulopathy
Published in Frontiers in immunology (04-04-2019)“…C3 Glomerulopathy (C3G) is a renal disease mediated primarily by dysregulation of the alternative pathway of complement. Complement is the cornerstone of…”
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C4 Nephritic Factors in C3 Glomerulopathy: A Case Series
Published in American journal of kidney diseases (01-12-2017)“…C3 glomerulopathy (C3G) defines a group of rare complement-mediated kidney diseases with a shared underlying pathophysiology: dysregulation of complement in…”
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Novel FHR fusion genes identified by MLPA and Western blot analysis
Published in Molecular immunology (01-10-2018)Get full text
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Defining the complement biomarker profile of C3 glomerulopathy
Published in Clinical journal of the American Society of Nephrology (07-11-2014)“…C3 glomerulopathy (C3G) applies to a group of renal diseases defined by a specific renal biopsy finding: a dominant pattern of C3 fragment deposition on…”
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Compstatin analog Cp40 inhibits complement dysregulation in vitro in C3 glomerulopathy
Published in Immunobiology (1979) (01-08-2015)“…Graphical abstract…”
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Defining Nephritic Factors as Diverse Drivers of Systemic Complement Dysregulation in C3 Glomerulopathy
Published in Kidney international reports (01-02-2024)“…C3 glomerulopathy (C3G) is an ultrarare renal disease characterized by deposition of complement component C3 in the glomerular basement membrane (GBM). Rare…”
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Rescue of Growth Retardation and Perinatal Lethality of Nedd4‐1 Null Mice by Loss of Maternally Inherited Grb10
Published in The FASEB journal (01-03-2008)“…Abstract only Nedd4‐1, a HECT type ubiquitin ligase, has been proposed to be involved in the regulation of type 1 insulin‐like growth factor receptor (IFG‐1R)…”
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