Search Results - "Shadrina, M.I."

Expression analysis of genes involved in mitochondrial biogenesis in mice with MPTP-induced model of Parkinson's disease by Rudenok, M.M., Alieva, A.Kh, Starovatykh, J.S., Nesterov, M.S., Stanishevskaya, V.A., Kolacheva, A.A., Ugryumov, M.V., Slominsky, P.A., Shadrina, M.I.

“…The mitochondrion is an extremely important organelle that performs various functions in the cell: e.g. energy production, regulation of respiration processes…”
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Whole-Transcriptome Analysis of Mouse Models with MPTP-Induced Early Stages of Parkinson’s Disease Reveals Stage-Specific Response of Transcriptome and a Possible Role of Myelin-Linked Genes in Neurodegeneration by Alieva, A.Kh, Zyrin, V.S., Rudenok, M.M., Kolacheva, A.A., Shulskaya, M.V., Ugryumov, M.V., Slominsky, P.A., Shadrina, M.I.

“…Parkinson’s disease (PD) is characterized by degeneration of dopaminergic neurons. A whole-transcriptome analysis of the substantia nigra and striatum of an…”
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MicroRNAs: Possible role in pathogenesis of Parkinson’s disease by Filatova, E. V., Alieva, A. Kh, Shadrina, M. I., Slominsky, P. A.

“…Parkinson’s disease is one of the most common human neurodegenerative disorders caused by the loss of dopaminergic neurons from the substantia nigra pars…”
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Molecular Mechanisms of Pathogenesis of Parkinson's Disease by Shadrina, M.I., Slominsky, P.A., Limborska, S.A.

“…Parkinson's disease is a complex disease characterized by a progressive degeneration of nigrostriatal dopaminergic neurons. The development of this condition…”
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Possible involvement of neuronal nicotinic acetylcholine receptors in compensatory brain mechanisms at early stages of Parkinson's disease by Kryukova, E V, Shelukhina, I V, Kolacheva, A A, Alieva, A Kh, Shadrina, M I, Slominsky, P A, Kasheverov, I E, Utkin, Y N, Ugrumov, M V, Tsetlin, V I

“…A role of nicotinic acetylcholine receptors (nAChR) in the development of Parkinson's disease (PD) has been investigated using two mouse models corresponding…”
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Neurotrophin gene expression in rat brain under the action of Semax, an analogue of ACTH4–10 by Agapova, T.Y., Agniullin, Y.V., Shadrina, M.I., Shram, S.I., Slominsky, P.A., Lymborska, S.A., Myasoedov, N.F.

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Neurotrophin gene expression in rat brain under the action of Semax, an analogue of ACTH 4–10 by Agapova, T.Y., Agniullin, Y.V., Shadrina, M.I., Shram, S.I., Slominsky, P.A., Lymborska, S.A., Myasoedov, N.F.

“…The heptapeptide Semax, an analogue of the N-terminal adrenocorticotropic hormone fragment (4–10) (ACTH 4–10), has been shown to exert a number of…”
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